Muscle phosphoglycerate mutase deficiency revisited

Arch Neurol. 2009 Mar;66(3):394-8. doi: 10.1001/archneurol.2008.584.

Abstract

Background: Phosphoglycerate mutase (PGAM) deficiency (glycogen storage disease type X) has been reported in 12 patients of whom 9 were African American.

Objective: To describe 2 patients, 1 of Pakistani and 1 of Italian ethnic origin, with typical clinical and biochemical changes of glycogen storage disease type X and novel mutations in the gene encoding the muscle subunit of PGAM (PGAM2).

Design: Clinical, pathological, biochemical, and molecular analyses.

Setting: Tertiary care university hospitals and academic institutions. Patients A 37-year-old Danish man of Pakistani origin who had exercise-related cramps and myoglobinuria and a 65-year-old Italian man who had exercise intolerance and myalgia but no pigmenturia and had undergone long-term statin therapy.

Main outcome measures: Clinical course and biochemical and molecular features.

Results: Biochemical evidence showed severe isolated PGAM deficiency, and molecular studies revealed 2 novel homozygous mutations, a nonsense mutation and a single nucleotide deletion. Pathological studies of muscle showed mild glycogen accumulation but prominent tubular aggregates in both patients.

Conclusions: We found that glycogen storage disease type X is not confined to the African American population, is often associated with sarcoplasmic reticulum (SR) proliferation, and is genetically heterogeneous.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aged
  • Creatine Kinase / blood
  • Glycogen Storage Disease / genetics*
  • Glycogen Storage Disease / metabolism*
  • Glycogen Storage Disease / physiopathology
  • Humans
  • Italy
  • Male
  • Pakistan
  • Phosphoglycerate Mutase / deficiency*
  • Phosphoglycerate Mutase / genetics

Substances

  • Creatine Kinase
  • Phosphoglycerate Mutase