Central nervous system cells

Villegas, S. N., Poletta, F. A. and Carr, N. G. Glia a reassessment based on novel data on the developing and mature central nervous system. Cell Biol. Int. 27 599-609, 2003. [Pg.591]

NO has complex roles in immunological host responses against viruses, and especially against HIV-1 infection. In HIV-1 infection, NO cannot be rigidly classified as an anti-inflammatory or proinflammatory molecule, but it can be deemed a true inflammatory mediator. Many studies support a proviral effect of NO in HIV-1 infection, mainly based on stimulation of viral replication, and on toxic effects on various cells, including central nervous system cells, via oxidative injury that may cause cellular and organ dysfunction, and immunosuppression and immunopathology, especially in the central nervous system. [Pg.23]

Fukumoto, H., Deng, A., Irizarry, M.C., Fitzgerald, M.L., Rebeck, G.W. (2002) Induction of the cholesterol transport ABCAl in central nervous system cells by liver X receptor agonists increases secreted AP levels. J. Biol. Chem., 277, 48508-48513. [Pg.353]

Johansson, C.B., Momma, S., Clarke, D.L., Risling, M., Lendahl, U., Frisen, J. (1999). Identification of a neural stem cell in the adult mammalian central nervous system. Cell,... [Pg.100]

Habgood MD, Begley DJ, Abbott NJ. Determinants of passive drug entry into the central nervous system. Cell Mol Neurobiol. 2000 20 231-253. [Pg.63]

Y. Yamada, T. Doi, T. Hamakubo and T. Kodama, Scavenger receptor family proteins roles for atherosclerosis, host defense and disorders of the central nervous system, Cell Mol. Life Sci. 54 (1998) 628-640. [Pg.314]

Chameau P, van Hooft JA (2006) Serotonin 5-HT(3) receptors in the central nervous system. Cell Tissue Res 326 573-81... [Pg.516]

M. Chillon, A. Bosch, J. Zabner, L. Law, D. Armentano, M. J. Welsh, and B. L. Davidson, Group D adenoviruses infect primary central nervous system cells more efficiently than those from group C, J. Virol. 73 2537 (1999). [Pg.280]

Henkemeyer, M. et al. (1996). Nuk controls pathfinding of commissural axons in the mammalian central nervous system. Cell 86, 35-46. [Pg.102]

Alternatively, endothelial cells and astrocytes (which compose the blood-brain banier) are infected directly (Strelow et al., 2001). A valiant of this theory is that infectious virions are transported in the cytoplasm of astrocytes and endothelial cells, and eventually transfeired to central nervous system cells by trans-cytosis or macropynocytosis) (Marechal et al., 2001). [Pg.605]

Patrick, J. Heinemarm, S. Members of a nicotinic acetyl-chohne receptor gene family are expressed in different regions of the mammalian central nervous system. Cell 47. 1987, 48, 965-973. [Pg.3127]

Richardson, W.D., Pringle, N., Mosley, M.J., Westermark, B. and Dubois-Dalcq, M. (1988) A role for platelet-derived growth factor in normal gliogenesis in the central nervous system. Cell 53 309-319. [Pg.146]

Chem, Y. (2000) Regulation of adenylyl cyclase in the central nervous system. Cell Signal 12 195-204. [Pg.64]

Linthicum DS, Munoz JJ, Blaskett A. Acute experimental autoimmune encephalomyelitis in mice. I adjuvant action of bordetella pertussis is due to vasoactive amine sensitization and increased vascular permeability of the central nervous system. Cell Immunol 1982 73(2) 299-310. [Pg.11]

Kawano, H., Kimura-Kuroda, J., Komuta, Y, Yoshioka, N., Li, H.R, Kawamura, K., Li, Y, Raisman, G., 2012. Role of the lesion scar in the response to damage and repair of the central nervous system. Cell Tissue Res. 349, 169-180. [Pg.114]

H. G. Craighead, S. W. Turner, R. C Davis, C. James, A. M. Perez, L. Kam, W. Shain, N. J. Turner and G. Banker, Chemical and topographical surface modification for control of central nervous system cell adhesion. Biomedical Microdevices, submitted for publication (1998). [Pg.45]

Studies on brain development during malnutrition have continued to demonstrate the vulnerability of the developing brain to nutritional insult. Winick (1968) has emphasized that nutritional deficiency occurring while cells of the central nervous system are actively dividing results in a permanent decrease in central nervous system cell number. Later nutritional deficiency which results in decrease in cell size appears to be recoverable. Perhaps even more important than effects of malnutrition on brain cell number is the effect on brain protein synthesis and rayelination. [Pg.314]

Join-Lambert OF, Ezine S, Le Moraiier A, et at Listeria monocytogenes-inlected bone marrow myeloid cells promote bacterial invasion of the central nervous system. Cell Microbiol. 2005 7(2) 167-180. [Pg.307]

Uchida, K. Yamada, M. Hayashi, T. Mine, Y Kawase, T. Possible harmful effects on central nervous system cells in the use of physiological saline as an irrigant during neurosurgical procedures. Surg. Neurol. 2004, 62, 96-105. [Pg.420]

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