Ischemia increases extracellular concentration of potassium, [K + ] o , reduces the cytosolic ATP concentrations, and reduces cytosolic pH.

Ischemia increases extracellular concentration of potassium, [K+]o, reduces the cytosolic ATP concentrations, and reduces cytosolic pH. The sodium-.

The model's AP has sensitivity to the ischemic parameters in agreement with experimental data, however, the sensitivity of the AP to pH was found to be ...

Ischemia increases extracellular concentration of potassium, [K+]o, reduces the cytosolic ATP concentrations, and reduces cytosolic pH. The sodiumpotassium ...

Ischemia increases extracellular concentration of potassium,. [K+jo, reduces the cytosolic. ATP concentrations, and reduces cytosolic pH. The sodium.

Na-K ATPase maintains ionic gradients in astrocytes and is suggested as an indicator of ischemic injury in glial cells.

Ischemia not only depletes bulk ATP but also induces a progressive series of cellular changes, all of which may modulate Na/K ATPase activity. These include: ...

Ischemia also depletes cellular ATP which inactivates ATPases (e.g., Na+/K+ ATPase), reduces active Ca2+ efflux, and limits the reuptake of calcium by the ...

Acute myocardial ischemia induces hyperkalemia (accumulation of extracellular potassium), a major perpetrator of lethal reentrant ventricular arrhythmias.

To study the ionic mechanisms of electrophysiologic changes in cell excitability and action potential duration during the acute phase of myocardial ischemia.