Poisonous Plants
Poisonous Plants
Poisonous Plants
oisonous Plants
of the Southeastern United States
Contributing Authors
John W. Everest, Extension Weed Scientist, Professor, Agronomy and Soils,
Auburn University
Thomas A. Powe, Jr., Professor, Large Animal Surgery and Medicine,
Auburn University
John D. Freeman, Extension Plant Taxonomist, Associate Professor (Retired),
Botany and Microbiology, Auburn University
ii
Contents
Foreword ...................................................................................................................v
iii
oleander...........................................Nerium oleander .......................................................14
perilla mint ......................................Perilla frutescens......................................................45
poison hemlock ................................Conium maculatum ...................................................27
poison ivy.........................................Toxicodendron radicans ............................................34
poison oak........................................Toxicodendron toxicarium .........................................34
poison sumac ...................................Toxicodendron vernix ................................................35
pokeweed .........................................Phytolacca americana...............................................10
rattlebox ..........................................Sesbania punicea ......................................................18
red buckeye......................................Aesculus pavia ..........................................................25
red maple.........................................Acer rubrum..............................................................46
redroot pigweed ...............................Amaranthus retroflexus ............................................29
rosebay ............................................Rhododendron catawbiense ........................................4
Scotch broom...................................Cytisus scoparius......................................................19
sesbania...........................................Sesbania exaltata......................................................17
sheep laurel .....................................Kalmia angustifolia .....................................................3
showy crotalaria ..............................Crotalaria spectabilis..................................................1
sicklepod..........................................Senna obtusifolia.......................................................16
spotted water hemlock.....................Cicuta maculata ........................................................26
St. John’s wort .................................Hypericum spp..........................................................41
sweet clover.....................................Melilotus spp. ...........................................................18
sweetshrub ......................................Calycanthus floridus .................................................42
white snakeroot ...............................Eupatorium rugosum ................................................31
yellow jessamine..............................Gelsemium sempervirens............................................9
Glossary ..................................................................................................................47
iv
Foreword
oisonous plants cause significant annual losses of money through injury to hu-
P mans and livestock. No verifiable sums are available, but figures as high as “sev-
eral million dollars” are often quoted. Undoubtedly, poisonous plants reduce pro-
ductivity, and some can even kill people and all classes of livestock.
If people could quickly recognize some of the more common poisonous plants, they
might avoid injury. In addition, livestock producers could remove poisonous plants from
fence lines and pastures where feasible.
The need for a regional publication on the identification and toxicity of common poiso-
nous plants was discussed at the annual meeting of southern Extension Directors held
July 1974. Dean William D. Bishop (Tennessee) appointed a committee to prepare such a
publication. Extension weed specialists and Extension veterinarians in the South were
surveyed to develop a master list of poisonous plants. The publication committee and au-
thors of the original publication were Dr. A. H. Kates, Extension (Virginia Polytechnic In-
stitute and State University); Dr. D. E. Davis, Botanist (Auburn University); Dr. John Mc-
Cormack, Extension Veterinarian (University of Georgia); and Dr. James F. Miller,
Chairman—Extension (University of Georgia).
In general, plant species in this publication were shown in order of their poisoning
frequency and relative importance according to the survey of southeastern states. In
cases where the toxic principle was the same or similar, plants were grouped together
within the list.
The current publication is a revision of the original publication, which was reprinted
several times over the years. Changes have been made in taxonomic nomenclature, plant
descriptions, and suggested treatments. In addition, several new poisonous plants have
been included. The contributing authors to this revised publication are Dr. John W. Ever-
est, Extension Weed Scientist, Professor, Agronomy and Soils (Auburn University); Dr.
Thomas A. Powe, Jr., Professor, Large Animal Surgery and Medicine (Auburn University);
and Dr. John D. Freeman, Extension Plant Taxonomist, Associate Professor (Retired),
Botany and Microbiology (Auburn University).
v
Showy crotalaria Crotalaria spectabilis
Toxicity
The toxic principle is the alka-
loid monocrotaline. Chickens, hors-
es, cattle, and swine are the
species usually affected, but sheep,
goats, mules, and dogs can be af-
fected to a lesser degree.
All parts of the plant are poisonous, whether green or dried in hay. The seeds are es-
pecially poisonous. Poisoning occurs when animals consume the green plant, hay contami-
nated with crotalaria, or dried seed in harvested grain.
Symptoms
Chickens can die from eating as few as 80 seeds. Fatalities may occur within a few
days or up to several weeks after ingestion. Symptoms include diarrhea, a pale comb (sig-
nifying anemia), ruffled feathers, and depression. Quail are also easily poisoned, but
turkeys are more tolerant.
Horses develop chronic unthriftiness, become uncoordinated, walk aimlessly, and may
“head press” against objects. Mucous membranes often exhibit jaundice, related to severe
liver damage.
In cattle, three syndromes are recognized: acute, chronic, and intermediate types. The
chronic type is most commonly seen with animals dying several months after consuming
1
the toxic material, usually hay contaminated with crotalaria. Symptoms include bloody di-
arrhea, icterus, rough hair coat, unthriftiness, edema, and weakness.
Swine may exhibit either an acute form, characterized by sudden gastric hemorrhage
and death, or a chronic form with anemia, ascites, loss of hair, and unthriftiness.
Treatment
There is no specific treatment.
Toxicity
The poisonous principle is the en-
zyme thiaminase, which inactivates thiamine (Vitamin B1) in the horse. In ruminants, an
aplastic-anemia factor causes depression of the bone marrow. Sheep are less susceptible
to the toxic effects than cattle and horses.
All portions of the plant are toxic whether green or dry. Poisoning by the plant is cu-
mulative, and symptoms may not appear until several weeks or months later. Clinical
cases are most often seen in the spring or late summer or fall, especially after periods of
drought when other forage is short or not available. Animals have shown toxicity from
consuming hay containing the dried plants.
Symptoms
Horses exhibit incoordination, often standing with their legs spread apart as if bracing
themselves. The affected animal arches its back and neck into a crouching stance. Occa-
sionally a fever is present up to 104°F. Before death horses may “head press” objects and
have spasms with the head and neck drawn backwards.
Cattle may exhibit two types of symptoms. The laryngeal form is seen often in younger
animals and is characterized by edema of the throat region, resulting in difficult and loud
2
breathing. The enteric form may be preceded by the laryngeal form. The enteric form is
characterized by bloody feces, blood in the urine, and excessive bleeding from fly bites.
The blood is slow to clot since platelets are deficient. Death usually occurs within a few
days after symptoms appear.
Sheep have shown blindness due to degeneration of the retinal epithelial cells after
grazing bracken fern.
Treatment
Remove animals from areas infested with bracken fern.
Give horses injections of thiamine at a dosage of 100 to 200 mg per day for 7 to 14
days.
In cattle, use whole blood transfusions, broad spectrum antibiotics, DL-batyl alcohol,
and protamine sulfate.
Toxicity
The resinoid andromedotoxin and the glucoside arbutin are the toxic principles respon-
sible for symptoms. Sheep, goats, and cattle are susceptible to poisoning if they eat the
plant, especially the leaves. There are recorded cases of toxicity in humans and monkeys.
3
Most clinical cases of laurel toxicity are seen in the winter and early spring months.
When other forage is not available, livestock may consume the toxic evergreen laurels.
Symptoms
Signs of toxicity occur usually within 6 hours after the plants are eaten. Symptoms in-
clude incoordination, excessive salivation, vomiting, bloat, weakness, muscular spasms,
coma, and death. The animals are often found down, unable to stand, with their heads
weaving from side to side.
Treatment
In severe cases, do not drench animals or give medicine by mouth since they may be
unable to swallow due to weakness of the throat muscles. Administer mineral oil or saline
laxatives by stomach tube. Give intravenous electrolyte solutions.
Rosebay Rhododendron
catawbiense
Shrub or densely branched
small tree 1 to 3 m tall. Leaves alternate, leathery, evergreen, entire, lanceolate to ellip-
tic, 8 to 15 cm long, 3 to 7 cm wide. Flowers showy, pink to purple, 1.5 to 2 cm long, in
terminal clusters. Found almost exclusively in the mountains of Alabama, Georgia, Ken-
tucky, Tennessee, West Virginia; mostly on rocky slopes and on ridges sometimes called
“hog-backs.”
Toxicity
Most of the many species of Rhododendron are considered poisonous (deciduous
Rhododendron spp. are known as azaleas). The toxic principle is called andromedotoxin,
which is a white carbohydrate material. Some may also contain a glucoside of hydro-
4
quinone. While Kalmia spp. are also termed “laurel,” they cause poisoning by other toxic
principles and result in other symptomology.
Poisoning can occur at any time of the year but is more commonly seen in the early
spring or in wintertime when snow covers other vegetation. Sheep, goats, and cattle are
commonly affected by grazing all portions of the plant, but particularly the leaves. Deaths
have also been recorded in humans and in sheep.
Symptoms
Symptoms include vomiting, bloating, salivation, and abdominal pain as evidenced by
straining. Eventually the animals grow weak, stagger, and become prostrate. Occasionally,
pneumonia is present due to inhalation of rumen contents into the lungs during vomiting.
Treatment
Use sound judgment in treatment. For instance, don’t drench or otherwise orally med-
icate animals that are vomiting or showing excessive swallowing movements. Inhalation
pneumonia may result. Veterinarians may be able to pass a large bore stomach tube to re-
lieve bloat or perform gastric lavage (This is very difficult on roughage diets).
Use mineral oil, magnesium sulfate, and calcium gluconate. Administer intravenous
fluids, such as glucose and saline solution.
Fetterbush
Leucothoe axillaris
Shrub up to 1.5 m tall with green,
slightly arching branches. Leaves alter-
nate, simple, evergreen, lanceolate to elliptic, 5 to 13 cm long, 1.5 to 5 cm wide. Racemes
2 to 7 cm long, bearing white flowers on all sides of the rachis; flowers 2 to 6 mm long.
Fruit a capsule 3 to 3.5 mm long, 5 to 6 mm broad. Seed shiny, light brown, 1 to 1.4 mm
long. Found from Virginia to Florida to Mississippi in Coastal Plain and sometimes in adja-
cent Piedmont; mostly along streams and in bogs, swamp forests, and moist woods.
5
Fetterbush Leucothoe recurva
Shrub similar to L. axillaris but with deciduous leaves and growing to 4 m tall. Differs
from L. racemosa in that the anthers are four-awned while they are two-awned for L.
racemosa. The capsule of L. racemosa is rounded on the sutures, and the seeds are wing-
less and 0.3 to 1.2 mm long; the capsule is angular and seeds winged and 2 to 2.5 mm
long for L. recurva. Leucothoe recurva is found primarily in rocky woods at higher eleva-
tions in Georgia, Tennessee, Virginia, and West Virginia.
Toxicity
The resinoid andromedotoxin and the glucoside arbutin are the toxic principles re-
sponsible for symptoms. Sheep, goats, and cattle are susceptible to poisoning if they con-
sume the plant, especially the leaves. There are recorded cases of toxicity in humans and
monkeys.
Most clinical cases of toxicity are seen in the winter and early spring months. When
other forage is not available, livestock may consume the toxic plants.
Symptoms
Signs of toxicity occur usually within 6 hours after the plants are consumed. Symp-
toms include incoordination, excessive salivation, vomiting, bloat, weakness, muscular
spasms, coma, and death. The animals are often found down, unable to stand, with their
heads weaving from side to side.
Treatment
In severe cases, do not drench animals or give medicine by mouth since they may be
unable to swallow due to weakness of the throat muscles. Administer mineral oil or saline
laxatives by stomach tube. Use intravenous electrolyte solutions.
6
Toxicity
The resinoid andromedotoxin and the glucoside arbutin are the toxic principles respon-
sible for symptoms. Sheep, goats, and cattle are susceptible to poisoning if they consume
the plant, especially the leaves. There are recorded cases of toxicity in humans and mon-
keys.
Most clinical cases of toxicity are seen in the winter and early spring months. When
other forage is not available, livestock may consume the toxic plants.
Symptoms
Signs of toxicity occur usually within 6 hours after the plants are eaten. Symptoms in-
clude incoordination, excessive salivation, vomiting, bloat, weakness, muscular spasms,
coma, and death. The animals are often found down, unable to stand, with their heads
weaving from side to side.
Treatment
In severe cases, do not drench animals or give medicine by mouth since they may be
unable to swallow due to weakness of the throat muscles. Administer mineral oil or saline
laxatives by stomach tube. Use intravenous electrolyte solutions.
7
fruit black, shiny, juicy, 0.7 to 1 cm long. Distributed throughout the South; most common
in fence rows, open woods, and pastures.
Toxicity
The toxic principle is hydrocyanic acid (also called prussic acid), which is created by
enzymatic action on the glucoside amygdalin. It is present primarily in the wilted leaves of
trees that have fallen. The bark and twigs are also toxic.
Ruminants (cattle, sheep, and goats) are most often affected, but single-stomach ani-
mals, like the horse, can also be affected. Poisoning may occur in spring, summer, or fall.
Symptoms
Symptoms are difficult breathing, bloat, an anxious expression, moaning, staggering,
recumbency, and convulsions before death. Animals may show signs within 15 to 30 min-
utes after consuming plants containing cyanide and may die within 1 hour of consuming
plants. Both the mucous membranes and the blood are bright red in color.
Treatment
Give intravenous injection of sodium nitrite and sodium thiosulfate as early as possi-
ble. If necessary, repeat treatments within a few hours.
Symptoms
Cyanide poisoning is very acute, and affected animals exhibit difficult breathing, anx-
ious expression, staggering and usually become recumbent, have convulsions, and die. An-
imals may show signs within 15 to 30 minutes after consuming plants containing cyanide
and may die within 1 hour of consuming plants. The blood is usually bright red.
In nitrate poisoning, the symptoms are similar except the blood is characteristically
chocolate brown.
Treatment
For cyanide poisoning, use sodium thiosulfate and sodium nitrite given intravenously
as an antidote.
For nitrate poisoning, use methylene blue as an antidote.
Prevention
Be careful when allowing cattle to graze johnsongrass, sorghums, etc., that have been
frosted, wilted, trampled, or drought stricken.
Ensile plants containing cyanide, or cut for hay. Drying eliminates most of the cyanide.
Analyze hay if you suspect that it may contain nitrate.
9
most abundant along fence rows and in open woods. Often confused in the vegetative
stage with Lonicera japonica, which has broader, deciduous leaves.
Toxicity
The toxic principles are the alkaloids gelsemine, gelseminine, and gelsemoidine. These
toxins are related to strychnine.
Livestock are affected, usually in the winter and spring months, from eating any part
of the plant. Humans have been poisoned from sucking nectar from the flowers or from
eating honey made from the flowers. Bees have died from consuming the nectar.
Symptoms
Animals are usually found staggering and incoordinated, with dilated eyes and convul-
sive movements. Often the animals are found down in comatose condition. Death usually
occurs soon after animals become comatose.
Treatment
There is no specific treatment.
Provide supportive therapy with intravenous fluids. Give cardiac and respiratory stim-
ulants such as caffeine and sodium benzoate.
10
Toxicity
The poisonous principles are oxalic acid and a saponin called phytolaccotoxin. In addi-
tion, alkaloids may be present. The root of the plant is the most toxic portion, but all other
parts of the plant contain smaller amounts of the toxic principles. Cattle, horses, swine,
and humans have all been poisoned after consuming this plant. Recognizable clinical
cases are rare, however. Swine are most often affected since they grub up the roots.
Poisoning occurs during spring, summer, or fall. In the springtime people commonly
cook the leaves and consume them. This “poke salad” is generally safe if the water in
which the leaves are cooked is poured off.
Symptoms
The most common symptom is a severe gastroenteritis with cramping, diarrhea, and
convulsions. Postmortem lesions include severe ulcerative gastritis, mucosal hemorrhage,
and a dark liver. In most cases the animal recovers within 24 to 48 hours.
Treatment
Give gastrointestinal protectives such as mineral oil or various clays. Administer tan-
nic acid and sedatives; a specific antidote is dilute vinegar. Provide respiratory stimulants.
Toxicity
The toxic principles of this common hog lot and barnyard plant are the alkaloids at-
ropine, hyoscyamine, and scopolamine. All parts of the plant are considered poisonous,
whether green or dry. However, the seeds are particularly poisonous. Usually, this plant is
not eaten except when other forage is unavailable. Cattle and swine are primarily affected,
11
but horses, poultry, dogs, and humans have been affected. Cows can be poisoned by con-
suming 0.5 to 1 pound of the green plant. The toxin is not destroyed by drying. Plants may
also take up nitrates.
Symptoms
Early symptoms include a weak and rapid pulse and heartbeat, widely dilated eyes,
and dryness of mouth and other mucous membranes. Animals may appear blind. Later
symptoms include slow breathing, lowered temperature, convulsions, or coma.
After eating the plants, sheep may have abnormal leg movements, disturbed vision,
and intense thirst; they may bite at imaginary objects in the air.
Pregnant sows consuming jimsonweed during their second and third months of gesta-
tion have produced deformed pigs. Some pigs may be born alive but exhibit varying de-
grees of flexed hips, stifles, and forelegs. The hocks may be overextended.
Treatment
Nonspecific. Use tannic acid, gastric lavage, and respiratory stimulants. Destroy
weeds in order to prevent problems.
Buttercup (crowfoot)
Ranunculus sardous and other species
12
Toxicity
This plant contains an irritant oil called protoanemonin. This oil is not a highly toxic
substance and is present in various species of buttercup in differing amounts. In general,
the flowering plant contains more toxin than the younger plant. The toxin is present in the
stems and leaves. The plant is very distasteful to livestock. All livestock are affected.
Symptoms
Signs include abdominal pain, severe diarrhea, convulsions, and death. Milk from af-
fected cows will be bitter and may be reddish in color. Although buttercup poisoning is un-
common, it occasionally occurs in cattle when other forages are in short supply.
Treatment
Nonspecific. Give purgatives initially, then gastrointestinal protectives later.
Toxicity
This ornamental shrub contains lantanin, a triterpenoid, and other compounds irritat-
ing to the mucosa of the gastrointestinal tract. All parts of the plant are quite toxic, and
poisoning may occur year-round but is most common in summer and fall. Many poisoning
cases occur when clippings are thrown into the pasture.
Sheep, cattle, horses, and humans are sensitive to the effects of the plant. Cattle are
most often affected. Children have been poisoned by eating the berries.
13
Symptoms
There are two forms of toxicity: acute and chronic. The acute form usually occurs
within 24 hours after the plants are eaten. Animals exhibit gastroenteritis with bloody,
watery feces. Severe weakness and paralysis of the limbs are followed by death in 3 to 4
days. The chronic form is characterized by jaundiced mucous membranes, photosensitiza-
tion, and ulcerations of the mucous membranes of the nose and oral cavity. The skin may
peel, leaving raw areas that are vulnerable to blowfly strikes and bacterial infection. Se-
vere keratitis may result in temporary or permanent blindness.
Treatment
Remove animals from direct sunlight. Use antibiotic injections and topical applications
of protective antibiotic creams. Treat with 20 percent sodium thiosulfate (1 ounce per 100
pounds); repeat treatment every other day. Use topical application of cortisone to relieve
itching.
Toxicity
The toxic principles are two glycosides, oleandroside and nerioside, which can be iso-
lated from all parts of the plant. Toxins may also be inhaled in smoke when plants are
burned. Human poisoning occasionally occurs from eating hot dogs roasted on sticks from
nearby oleander plants. This extremely toxic plant can poison livestock and humans at any
time of the year.
14
Symptoms
Severe gastroenteritis, diarrhea, abdominal pain, sweating, and weakness are the
usual symptoms. These signs appear within a few hours after eating the leaves. Cardiac
irregularities are common, often characterized by increased heart rate. However, a slow-
er heart rate is often detected in the later stages.
Treatment
Nonspecific. Treat symptoms although symptomatic treatment is usually unsuccessful.
Symptoms
Sheep and cattle exhibit hemorrhagic diarrhea, shallow and rapid respiration, and
fast irregular pulse; they become comatose before death. Affected cattle may become con-
stipated.
15
Postmortem examination reveals hemorrhages in the abomasum and intestines,
rumen stasis, and dark tarry blood. The rumen usually contains seeds of the plant.
Treatment
Remove all animals immediately from pastures containing the plant and confine them
to clean pastures or a dry lot. Provide general supportive treatment, including saline laxa-
tives, rumen stimulants, and intravenous fluid therapy. Specific antidote is dilute vinegar
for saponins in the early stages of toxicosis.
Toxicity
The toxic principles have not been clearly established. The seeds appear to exert their
toxicity upon the skeletal muscles, kidney, and liver. The leaves and stem, whether green
or dry, also contain toxin. Sicklepod is much more prevalent but somewhat less toxic than
coffee senna. Animals can be poisoned by consuming the plant in the field, in green chop,
in hay or if the seed is mixed with grain. Cattle are susceptible to the effects of these
plants, and other animals are probably susceptible as well.
16
Symptoms
Diarrhea is usually the first symptom. Later, the animals go off feed, appear lethargic,
and tremors occur in the hind legs, indicating muscle degeneration. As muscle degenera-
tion progresses, the urine becomes dark and coffee colored. The animal becomes recum-
bent and is unable to rise. Death often occurs within 12 hours after the animal goes down.
There is no fever.
Treatment
Once animals become recumbent, treatment is usually ineffective. Do not give seleni-
um and Vitamin E injections because they will potentiate the disease. Vitamin E is the
more important component in this potentiation.
Toxicity
The poisonous principle is a saponin that is toxic to livestock and humans. The seeds
are the most toxic part of the plant and are consumed in the late summer, fall, or winter
when other forage is scarce. Cattle are often affected when moved into new pastures con-
taining the plant. Cattle often develop a craving for the seeds.
Symptoms
Affected cattle are often found dead. An opened rumen may reveal the sprouted seeds,
and there will be a hemorrhagic inflammation of the abomasum and intestines. Symptoms
are variable and include hemorrhagic diarrhea, but constipation can also occur. The ani-
mals walk stiffly with an arched back, have shallow respiration and a weak rapid pulse.
They become prostrate and comatose before death.
17
Treatment
Symptomatic. In severe diarrhea, insert a stomach tube and administer intestinal pro-
tectives. If animals are constipated, give mineral oil by the same route. Administer intra-
venous fluids in dehydrated animals. Use dilute vinegar to counteract saponin in the early
stage of the toxicity.
Toxicity
The seeds contain a saponin that is quite toxic to poultry, cattle, sheep, goats, and hu-
mans. As few as nine seeds per bird can be fatal. Sheep can be killed by consuming as lit-
tle as 50 grams per 100 pounds of body weight. Poisoning usually occurs in the fall when
other forage is scarce.
Symptoms
Animals appear severely depressed and have a rapid pulse and diarrhea.
Treatment
Give saline purgatives. Specific antidote is dilute vinegar in early stages of toxicosis to
counteract saponin.
Symptoms
Symptoms are related to massive blood loss. Swellings appear under the skin due to
accumulations of blood. These swellings vary in size and may occur at any site on the
body but particularly in areas that are susceptible to bruising. Other symptoms can in-
clude pale mucous membranes, rapid and weak pulse, and weakness. Females may hem-
orrhage following calving. Occasionally animals hemorrhage internally and exhibit signs of
shock.
Treatment
Remove sweet clover hay from animals immediately. Give Vitamin K injections and
transfusions of whole blood.
Toxicity
Poisoning from this shrub is usually of a mild type. Large amounts are required to
cause symptoms in animals. Alkaloids have been identified as being the toxic principle.
Cytisin, sparteine, and isosparteine are found in the twigs, leaves, and seeds in small
19
amounts. A glycoside, scoparin, has also been isolated. Horses are most susceptible to
poisoning by Scotch broom.
Symptoms
The alkaloid portions cause depression of the nervous system, and the glycoside caus-
es a diuretic effect. Symptoms include incoordination and occasional excitement. Ingestion
of large amounts of this shrub can cause coma and death.
Treatment
Nonspecific. Treat symptoms.
Toxicity
Toxic principles include the phytotoxin robinin and the glycoside robitin. Horses, cat-
tle, sheep, poultry, and humans may be poisoned by ingesting roots, bark, sprouts, seed
pods, or trimmings. Horses are the animal most susceptible to the effects of black locust.
Symptoms
Symptoms include weakness, posterior paralysis, depression, loss of appetite, irregu-
lar pulse, difficult breathing, and diarrhea.
20
Treatment
Insert a stomach tube, and administer a laxative, such as mineral oil. Use stimulants,
such as Digitalis (1⁄ 8 grain every 15 minutes for four doses).
Toxicity
The toxic principles are tetranortriterpene neurotoxins and unidentified resins. The
fruit (berries) are the most toxic part of the tree. The leaves, bark, and flowers are only
mildly toxic and usually cause no problem. Most poisonings occur in the fall or winter
when the berries ripen.
Swine and sheep are most often affected. Toxicity may occur after consumption of
more than 0.5 percent of body weight. Poultry and cattle can be poisoned, but larger
amounts are required. Children have been poisoned by eating the berries.
Symptoms
The gastrointestinal tract is affected; therefore, common symptoms include vomiting
and diarrhea. Occasionally, the central nervous system is affected, and animals are se-
verely depressed or excited.
Treatment
Evacuate the affected animal’s gastrointestinal system. Use lentin-carbocal gastroin-
testinal protectives, respiratory stimulants, and caffeine.
21
Common cocklebur Xanthium strumarium
Toxicity
The toxic principle is the glycoside carboxyotractyloside. It is concentrated in the
seeds and seedlings (cotyledon state). Mature plants are distasteful to animals and con-
tain less of the toxin.
Swine are the animals most commonly poisoned. They root up and ingest the two-leaf
stage of the plant in the springtime. Chickens and other livestock have also been poisoned.
Symptoms
Symptoms include vomiting and gastrointestinal irritation with occasional diarrhea.
Large amounts often cause nervous symptoms, including spasmodic running movements
and convulsions.
Treatment
Treatment is of little or no value once symptoms have been observed. Try neutralizing
the toxin with vegetable oil, whole milk or cream, or activated charcoal.
22
Horsenettle Solanum carolinense
Toxicity
The toxic alkaloid solanine has
been isolated from Solanum spp. Toxici-
ty of these plants varies depending
upon maturity, environment, and por-
tion of plant ingested. The berries are
the most toxic part and are more toxic
when they have matured and turned
yellow. Immature green fruit are also
considered toxic. Leaves are also toxic, but to a less-
er degree. Humans and all classes of livestock have
been poisoned.
Symptoms
Two syndromes have been described: acute and chronic. The acutely poisoned animal
is characterized by irritation of the mouth and gastrointestinal lesions. In the chronic
form, symptoms include unthriftiness, jaundiced mucous membranes, abdominal dropsy,
and constipation.
Treatment
Nonspecific. Treat early symptoms with tannic acid, charcoal, protectants, and evacu-
ation of the stomach.
23
Black nightshade Solanum nigrum
Toxicity
The toxic alkaloid solanine has been isolated from this group of plants. Toxicity of
these plants varies, depending on maturity, environment, and portion of plant ingested.
The berries are the most toxic part and are more toxic when they are green and imma-
ture. The berries are somewhat less toxic when they mature and turn black. Leaves are
also toxic but to a lesser degree. Humans and all classes of livestock have been poisoned.
A closely related plant S. intrusum is known as “garden huckleberry” or “wonderber-
ry.” Garden huckleberry is a cultivated plant with nontoxic fruit. However, distinguishing
S. intrusum from S. nigrum is almost impossible.
Symptoms
Two syndromes have been described: acute and chronic. The acutely poisoned animal
is characterized by irritation of the mouth and gastrointestinal lesions. In the chronic
form, characteristic symptoms are unthriftiness, jaundiced mucous membranes, abdomi-
nal dropsy, and constipation.
Treatment
Nonspecific. Treat early symptoms with tannic acid, charcoal, protectants, and evacu-
ation of the stomach.
24
Red buckeye Aesculus pavia
Toxicity
The glycoside aesculin has been detected in the buckeye sprout, young leaves, and the
mature seed. Humans and all classes of livestock can be affected by ingestion of the buck-
eye. Cattle are most frequently affected, usually by consuming young shoots and leaves in
the early springtime.
Symptoms
Generally, symptoms are drunkenness, trembling, muscular weakness, and incoordi-
nation, with affected animal having extreme difficulty going downhill. Other symptoms in-
clude vomiting, irritated mucous membranes, and paralysis.
Treatment
Normally, no treatment is required. If animals are removed from infested areas, they
will usually recover.
25
Spotted water hemlock Cicuta maculata
Toxicity
A very poisonous alkaloid and a
resinoid toxin are found in all parts of
the spotted water hemlock, but pri-
marily in the roots. The pithy area be-
tween the nodes contains a greenish
yellow oil, which contains the toxins.
Livestock and humans are especially susceptible to this poison. The plant grows in
wet, damp soil, which enables animals to easily pull up the plant. Most livestock poisoning
cases occur in the springtime; children have been poisoned by making “pea-shooters”
from the hollow stem segments.
Symptoms
Animals exhibit nervous symptoms because of the toxin, which is a convulsant. Trem-
bling and jerking motions are followed by convulsions. In addition, animals froth at the
mouth, move jaws as if they are chewing, and may vomit. Eyes are widely dilated, and
temperature is elevated. Death occurs from respiratory failure. Chronic ingestion may
lead to abnormal fetal development and malformation in swine and horses.
26
Treatment
Nonspecific. Treat symptoms.
Toxicity
The poison hemlock contains coniine, an alkaloid, and other compounds that are capa-
ble of poisoning livestock, poultry, and humans. The stems, leaves, and mature fruits are
toxic. The leaves are more dangerous in the springtime, and the fruit is the most danger-
ous in the fall.
Symptoms
Symptoms are gastrointestinal irritation, nervousness, trembling, staggering, coldness
of the extremities, and slow heartbeat. Eventually coma and death occur.
Treatment
Use respiratory stimulants or intestinal protectives.
Symptoms
Animals are most often poisoned when feed grains have become contaminated with
the castor bean seeds. Depending upon the amount consumed, symptoms appear several
hours to several days after animals consume the toxin. Violent purgation in the form of
straining and bloody diarrhea is the classical sign. Other signs are dullness, abdominal
pain, weakness, trembling, and incoordination.
Treatment
Administer intestinal protectives in large amounts by stomach tube. If affected animal
is dehydrated, provide large amounts of intravenous fluids.
28
Toxicity
All parts of plant are poisonous, especially the fruit. The unripe capsules and the
plant sap contain saponin and toxalbumin (protein toxin).
Symptoms
Symptoms are diarrhea, watery green feces with blood, listlessness, weakness, and
dehydration. Symptoms are frequently delayed 2 to 4 days after consumption of the plant.
Treatment
There is no antidote. Correct dehydration with intravenous fluids and electrolytes. Use
intestinal protectives. May do rumenatomy or flush. In chronic poisoning of cattle, stimu-
late rumen movement. Try to improve appetite. Use intravenous fluids for energy.
Toxicity
This plant may accumulate ni-
trates after treatment with herbicides
such as 2,4-D and also after heavy ni-
trogen fertilization. This plant also be-
comes more palatable after treatment
with herbicides. Cattle, sheep, and
other ruminants are susceptible.
Symptoms
Symptoms suddenly appear 5 to
10 days after animals consume plants.
Animals exhibit weakness, trembling,
29
and incoordination. In 48 hours, symptoms progress rapidly from knuckling of posterior to
paralysis, sternal recumbency, and death.
Another syndrome in swine and cattle occurs in which incoordination is followed
closely by coma and death. Necropsy examination reveals degeneration of the brain and
edema in the kidney region with degeneration of the kidney tubules.
Treatment
To treat non-nitrate type toxicosis, use mineral-corticoid hormones, IV calcium glu-
conate to normalize ECG, and dextrose IV.
Toxicity
The leaves and flowers of this plant contain a cardioactive glycoside and are attrac-
tive to cattle and sheep in the early spring. The evergreen appearance is particularly at-
tractive to cattle when other forage is not available.
Symptoms
Staggering, trembling, convulsions, diarrhea, and other gastrointestinal symptoms are
common signs.
Treatment
There is no specific treatment. Use gastrointestinal protectives if diarrhea is present.
Use sedatives if convulsions occur.
30
White snakeroot Eupatorium rugosum
Toxicity
The toxic principle has been identified as an alcohol called tremetol. It is found in all
parts of the plant whether green or dry. All domestic livestock, some laboratory animals,
and humans are susceptible to the effects of this plant. Animals may be poisoned from
consuming the actual plant or from ingesting milk from cows, sheep, or mares that have
eaten the plant. Drinking milk from cows that have eaten white snakeroot has accounted
for an illness called “milk sickness” and for deaths in humans.
Symptoms
Trembling is the most common symptom; in fact, this condition has been called “trem-
bles.” Animals are stiff and sluggish, stand with feet wide apart, and may eventually be-
come recumbent. Slobbering, vomiting, constipation, and dribbling of urine also occur. The
breath has a ketone odor. Humans may exhibit delirium after drinking toxic milk.
Treatment
There is no specific treatment. Provide laxatives for some relief.
31
Autumn sneezeweed Helenium autumnale
Toxicity
The toxic principles are the glucoside sesquiterpene lactone and a phenol. Autumn
sneezeweed appears to cause more severe symptoms than does bitter sneezeweed (see
page 33). In the Rocky Mountain area, sheep have been severely poisoned by consumption
of all portions of the autumn sneezeweed plant. Cattle may also be affected but require a
much larger amount.
The plant retains its toxicity even after drying; therefore, heavily contaminated hay
can cause problems. Most cases occur when animals are on summer pasture and other
forage is not available.
Symptoms
Autumn sneezeweed is a severe irritant to the mucous membranes. Dullness, trem-
bling, and weakness are first symptoms. In many instances, vomiting is prominent. For
this reason, the illness is called “spewing sickness.” Many vomiting animals inhale part of
the regurgitated material into the trachea and develop inhalation pneumonia. These ani-
mals usually survive only to become chronically poor performers and perhaps die later
from secondary ailments.
Treatment
There is no effective treatment.
Remove from source. Feed high protein feed, mineral supplement of 75 pounds salt
and 25 pounds dicalcium phosphate; or feed sodium sulfate.
32
Bitter sneezeweed (bitterweed)
Helenium amarum
Toxicity
The toxic principles are the glucoside sesquiterpene lactone and a phenol. Bitter
sneezeweed causes less severe symptoms than does autumn sneezeweed. The plant re-
tains its toxicity even after drying; therefore, heavily contaminated hay can cause prob-
lems. Most cases occur when animals are on summer pasture and other forage is not
available.
Symptoms
Bitter sneezeweed is a severe irritant to the mucous membranes. Dullness, trembling,
and weakness are first symptoms. In many instances, vomiting is prominent. For this rea-
son, the illness is called “spewing sickness.” Many vomiting animals inhale part of the re-
gurgitated material into the trachea and develop inhalation pneumonia. These animals
usually survive only to become chronically poor performers and perhaps die later from
secondary ailments.
Bitterweed can cause similar problems under experimental conditions. Cattle con-
sume bitterweed only if other forage is unavailable.
Treatment
There is no effective treatment.
Remove from source. Feed high protein feed, mineral supplement of 75 pounds salt
and 25 pounds dicalcium phosphate; or feed sodium sulfate.
33
Poison ivy Toxicodendron radicans
P erennial, high-climbing,
woody vine. Leaves al-
ternate, deciduous, pinnate-
ly compound; leaflets three,
thin, bright green, shiny,
ovate to elliptic, 2 to 12 cm
long, 2 to 12 cm wide, en-
tire to serrate to shallowly
lobed. Flowers small, yel-
lowish green, in clusters of
two to six in lower leaf
axils. Fruit a scarcely fleshy
drupe, glabrous to short pu-
bescent, 0.4 to 0.5 cm
broad. Found throughout the southern states east of the Mississippi River; most abundant
in moist woods but also in pastures, fence rows, roadsides, and waste places.
Toxicity
The toxic principle is a phenolic compound called urushiol. It is a skin and mucous
membrane irritant and is found in all parts of the plant. Some humans are quite sensitive
to the effects of the toxin, whereas others show no ill effects from coming into contact
with the plant. The toxin has little or no effect on animals, but pets may carry the irritat-
ing substance on their hair and thereby transmit it to humans.
Symptoms
Susceptible humans exhibit intense itching with inflammation and the formation of
blisters at the areas of contact. Animals are rarely affected. Burning may be dangerous
because the irritant may be transmitted by smoke.
Treatment
Consult a physician for proper treatment.
34
Poison sumac Toxicodendron vernix
Toxicity
The toxic principle is a phenolic compound called urushiol. It is a skin and mucous
membrane irritant and is found in all parts of the plant. Some humans are quite sensitive
to the effects of the toxin while others show no ill effects from coming into contact with
the plant. The toxin has little or no effect on animals, but pets may carry the irritating
substance on their hair and thereby transmit it to humans.
Symptoms
Susceptible humans exhibit intense itching with inflammation and the formation of
blisters at the areas of contact. Animals are rarely affected. Burning may be dangerous
because the irritant may be transmitted by smoke.
Treatment
Consult a physician for proper treatment.
35
Atamasco lily Zephyranthes atamasco
Toxicity
The toxic principle is an alkaloid. The bulb of this plant is the most toxic portion. The
leaves are also toxic but to a much lesser degree. Animals are usually poisoned in the
springtime when the ground is wet and there is little forage. Cattle, horses, and poultry
have been poisoned by this plant.
Symptoms
Symptoms usually appear in 24 to 48 hours after the bulbs are eaten. Staggering,
diarrhea with blood, collapse, and death are the usual symptoms.
Treatment
Give gastrointestinal protectives.
36
Fly poison (stagger grass)
Amianthium muscaetoxicum
Toxicity
The highest concentration of
the toxin, an alkaloid, is found in
the bulb of the plant. The alkaloid,
which is cumulative, is also found
in the fruit and leaves. Animals
consume the plant only when other
forage is unavailable. Cases of toxi-
city occur in the spring, summer,
and fall. Cattle and sheep are most
commonly affected.
Symptoms
Animals exhibit vomiting, frothing at the mouth, staggering, rapid respiration, subnor-
mal temperature, and weakness. Death is caused by respiratory failure.
Treatment
Keep animals quiet. Give sedatives. Administer gastric protectives by stomach tube
unless this procedure excites the animal.
37
Milkweed Asclepias tuberosa and other species
Toxicity
Various species of milkweeds have yielded resinoids, alkaloids, and glycosides. All
parts of the plant are toxic, whether consumed green or dried in hay. Cattle, sheep, goats,
horses, and poultry are all sensitive to the effects of milkweed. Consuming the toxic plant
in the amount of 2 percent of body weight can cause symptoms.
Symptoms
Losses usually occur when animals are forced to graze the plant due to lack of other
forage. Usual signs include staggering, depression, weakness, labored respiration, and di-
lated pupils. Animals go down and exhibit tetanic spasms before going into a coma and
dying.
Treatment
Give laxatives and intravenous fluids.
38
Common yarrow Achillea millefolium
39
Toxicity
Bitter glycosides, cephalin and cephalanthin, are found primarily in the leaves of this
plant. Other parts of the plant are less toxic. Cattle are usually affected in the summer
and fall when other forage is scarce.
Symptoms
Symptoms include vomiting and muscular weakness. Occasionally convulsions and
death result.
Treatment
Administer laxatives by stomach tube.
Toxicity
Members of the mustard family contain isothiocyanates, commonly called mustard
oils. Certain members, such as the rutabaga, have a high concentration of goitrogenic sub-
stance that has produced goiter in livestock. Animals are most often poisoned from spring
to fall but can show symptoms of toxicity throughout the year if hay is contaminated with
a large amount of mustard. All parts of the plant are toxic, especially the seeds. Swine,
cattle, and horses are susceptible.
Symptoms
Symptoms are primarily those of a severe gastroenteritis due to irritation of the mu-
cous membranes. Abdominal pain, salivation, and diarrhea are commonly seen.
40
Rape, a winter annual, often causes a series of problems including pulmonary emphy-
sema, diarrhea, blindness, and hemoglobinuria. Photosensitization can occur in swine and
light-skinned animals. Abortions can occur in sows.
Treatment
There is no effective treatment.
Include hay free of mustards in the diet. If rape forage is being used, allow access to
other types of pastures.
Toxicity
A red fluorescent pigment, hypericin, is the toxic agent. It is a primary photosensitiz-
ing compound, but the liver is not affected. Cattle, sheep, and horses are affected; goats
are also affected, but to a lesser degree. Animals are poisoned by eating the fresh plant or
dried hay. The toxic principle is not destroyed by drying or excessive heat. Younger plants
are more palatable than the older ones.
Symptoms
In cattle, consumption of the plant equal to 1 percent of the body weight is toxic. Pho-
tosensitization is noticed within 2 days to 3 weeks after ingestion. Light areas of the skin
41
surface become hypersensitive to sunlight. The white skin peels off and may hang from the
body before falling away. Affected animals exhibit itching and try to scratch themselves on
objects. Blindness often results from inflammation and secondary infection of the cornea.
Animals occasionally have convulsions in the acute form of poisoning. More commonly,
however, the photosensitization results in secondary infection of the affected areas. Ani-
mals may actually die of starvation.
Treatment
Remove animals from contaminated pastures to barns, woods, pasture, or areas
where animals have access to shade. Treat affected skin areas with antibacterial prepara-
tions; administer antibiotic injections to prevent infection.
Toxicity
An alkaloid is contained in the seed of the plant. It has been reported to be toxic to
cattle. The plant is dangerous but not commonly eaten.
Symptoms
Strychnine-like action results in an increased excitability in which tetanic convulsions
are exhibited.
Treatment
Use sedatives to control the convulsions caused by the strychnine-like action of the
toxin.
42
Mexican pricklepoppy Argemone mexicana
Toxicity
The alkaloids berberine and protopine are contained in the entire plant. In addition,
the seeds contain sanquinarine and dihydrosanquinarine. If seeds are consumed in suffi-
cient quantities, livestock, humans, and chickens can be poisoned. One ounce of seed
causes symptoms in poultry, and 2 ounces usually produce death.
Symptoms
In humans and chickens, widespread edema (dropsy) is the main symptom. In chick-
ens, wattles swell, the tips of the comb darken, and egg production decreases. Other
symptoms in chickens are weakness, hemorrhagic enteritis, and death.
Treatment
Nonspecific. Give diuretics to livestock.
43
Hairy vetch Vicia villosa
Toxicity
A cyanogenetic glycoside is present in vetch seed. In Alabama, cattle have exhibited
signs of cyanide poisoning after eating from a sack of vetch seed left in a barnyard.
Symptoms
Symptoms include bellowing, sexual excitement, a wild appearance, crawling on the
ground, and death. Some species of vetch have been known to cause liver damage and
photosensitization.
Treatment
Treat cyanide poisoning with sodium thiosulfate and sodium nitrite. If cattle exhibit
photosensitization, keep them in a shady area and give antibiotics to prevent secondary
infection.
44
Perilla mint Perilla frutescens
Toxicity
This toxic plant contains “perilla ketone,”
which produces pulmonary edema and pleural
effusion in a variety of animals. Toxic cases are
seen sporadically, usually in the late summer or fall after grazing of the plant, most often
in cattle and horses. May account for birth defects in calves when hay containing perilla
mint is fed to cows early in the gestation period.
Symptoms
Affected animals exhibit respiratory distress. They have difficulty breathing especially
when exhaling. They may grunt when exhaling and may have a nasal discharge and an ele-
vated temperature; friction sounds occur through the chest wall.
Postmortem examination reveals pulmonary emphysema and edema. The perilla mint
seeds are often found in the rumen of the cow.
Treatment
Once symptoms of severe pulmonary edema and emphysema occur, treatment is usu-
ally ineffective. Try injections of antihistamines, steroids, and antibiotics. Handle cattle
quietly to prevent further respiratory complication and subsequent death.
45
Red maple Acer rubrum
Toxicity
The wilted leaves of this plant con-
tain an unknown compound, which is be-
lieved to be an oxidant. Cattle and hors-
es have been poisoned when wilted
leaves were ingested.
Symptoms
Weakness, rapid heartbeat and respiration, depression, icterus, cyanosis, and brown-
ish discoloration of blood and urine are the major symptoms.
Treatment
Treat symptoms by providing fluids and oxygen. Use methylene blue in the early
stages of the disease.
46
Glossary
abomasum – the fourth stomach of a ruminate animal.
achene – a small, dry, hard one-locular, one-seeded, indehiscent fruit.
acuminate – a type of apex with concave sides that taper to a sharp point.
acute – a type of apex or base with straight sides that meet at less than a 90° angle.
ascites – an excessive accumulation of fluid in the peritoneal (abdominal) cavity.
axil – the angle between any two structures; usually applied to the angle between the leaf
and stem.
axillary – in an axil.
bipinnate – twice pinnate.
blade – the expanded portion of a leaf.
bract – a leaflike structure subtending a flower or flower cluster.
capsule – a dry, or occasionally leathery, dehiscent fruit.
ciliate – fringed with hairs at the margin.
comatose – affected with a coma, which indicates a state of complete loss of conscious-
ness from which the animal cannot be aroused.
compound – composed of two or more similar and united parts, as in a compound leaf.
cordate – heart-shaped with the point at the apex.
crenate – shallowly round-toothed or teeth obtuse; scalloped.
cyanosis – bluish discoloration of skin and mucous membranes due to poor oxygenation of
the hemoglobin in the blood.
dehiscent – opening regularly, usually by slits like a pea or pod or cotton boll.
dentate – sharp-toothed, the sharp or coarse teeth perpendicular to the margin.
denticulate – minutely or finely dentate.
diuretic – a drug or other agent that promotes the secretion of urine.
dropsy – an abnormal accumulation of fluid in the cellular tissues or in a body cavity.
drupe – a fleshy, indehiscent fruit, containing one or more single-seeded pits or stones,
such as a cherry or plum.
edema – the accumulation of abnormally large amounts of fluid in the intercellular tissue
spaces of the body.
elliptic – being narrowed to relatively round ends and widest at or about the middle.
entire – a margin without teeth or other irregularities.
follicle – a dry, usually several- to many-seeded fruit dehiscent along one line.
funnelform – gradually widening from base to apex; funnel-shaped.
47
gastritis – inflammation of the stomach.
gastroenteritis – inflammation of the stomach and intestines.
glabrate – losing hair with aging or maturity of the plant part or structure.
glabrous – without hairs.
hemorrhage – excessive bleeding; copious escape of blood from vessels.
hemorrhagic enteritis – inflammation of the intestines characterized by the passing of
blood in the droppings.
icterus – a yellow appearance of the skin and mucous membranes. It is caused by liver
damage, impairment of bile flow, or excessive destruction of red blood cells.
indehiscent – not regularly opening.
jaundice – a common term for icterus.
lanceolate – lance-shaped, much longer than wide, widened at or above the base and ta-
pering to the apex.
linear – long and narrow with essentially parallel margins, as the blades of most grasses.
necropsy – an examination of the body after death. An autopsy or postmortem examina-
tion.
nutlets – one of the sections of a mature ovary; a small nut.
ob – a prefix signifying inversion, as obovate.
oblanceolate – much longer than wide, with widest point above the middle and the petiole
at the narrower end.
ovate – with an outline like that of a hen’s egg, the broader end below the middle.
palmate – lobed, divided or ribbed like the fingers from the palm of the hand.
panicle – a cluster of flowers (inflorescence) in which the branches of the primary axis
are racemose and the flowers pedicellate.
pappus – a ring of appendages (modified calyx), often hairs, capping the fruit of many
Compositae.
petal – one unit of the corolla.
petiole – leaf stalk between blade and stem.
photosensitization – sensitivity to light. Usually used to indicate a condition in animals
when superficial layers of non-pigmented skin exhibit dermatitis when exposed to
sunlight. The condition may develop when animals consume certain poisonous plants
or other agents that are not properly metabolized by the liver.
pilose – covered with long, soft, spreading hairs.
pinnate – with the leaflets arranged along a common axis (rachis); featherlike.
pistil – one or more fused carpels consisting of stigma, style (if present), and ovary.
pistillate – flower type with pistils and without fertile stamens; female.
puberulent – covered with fine, minute hairs.
48
pubescent – covered with hairs, often soft and downy.
raceme – a simple, elongated inflorescence with stalked flowers.
rachis – central elongate axis bearing leaflets (in a pinnate leaf) or flowers (in an inflores-
cence).
recumbent – lying down. Usually used to indicate an animal that is down and unable to
arise.
rhizome – usually elongate, horizontal, underground stem.
rhizomatous – producing or possessing rhizomes.
rhombic – diamond-shaped.
rootstock – subterranean stem; rhizomes.
rumenatomy – surgical opening of the rumen.
samara – a dry, indehiscent, winged fruit.
scape – a flowering stem that is leafless above ground level.
scapose – resembling a scape; stem essentially naked.
sepal – a single unit of the calyx.
serrate – margin with teeth like a saw, teeth pointing toward the tip of the leaf.
simple – leaf type of one continuous area, not divided into leaflets.
sinuate – the margin wavy in and out with regular strong indentations.
spike – an elongated, simple inflorescence with sessile flowers.
spikelet – a secondary spike; a part of a compound inflorescence; the “flower” of grasses.
stamen – the pollen-bearing organ of a seed plant.
staminate – flower type having stamens and no functional pistils; male.
stasis – a stoppage of the flow of blood or other body fluid in any part. In animals, this
term often indicates a paralysis or abnormal slowing down of the gastrointestinal
tract.
stellate – starlike, with radiating branches.
tetanic spasm – a sudden, persistent, violent, involuntary contraction of a muscle or a
group of muscles. The body becomes rigid, often with the head drawn backwards.
umbel – an inflorescence with pedicels or peduncles (rays), or both, arising from a com-
mon point.
wattles – small fleshy pendulous processes usually about the head or neck.
49
Contents Listed By Family
Family Common Name Genus And Species Page
Aceraceae ...............................red maple .......................Acer rubrum.............................46
Amaranthaceae .......................redroot pigweed .............Amaranthus retroflexus ............29
Amaryllidaceae .......................atamasco lily ..................Zephyranthes atamasco .............36
Anacardiaceae.........................poison ivy .......................Toxicodendron radicans..............34
Anacardiaceae.........................poison oak ......................Toxicodendron toxicarium ..........34
Anacardiaceae.........................poison sumac .................Toxicodendron vernix .................35
Apocynaceae ...........................oleander .........................Nerium oleander ........................14
Asclepiadaceae .......................milkweed ........................Asclepias tuberosa .....................38
Calycanthaceae .......................sweetshrub.....................Calycanthus floridus...................42
Compositae (Asteraceae) ........autumn sneezeweeed .....Helenium autumnale ..................32
Compositae (Asteraceae) ........bitter sneezeweed ..........Helenium amarum......................33
Compositae (Asteraceae) ........common cocklebur .........Xanthium strumarium ................22
Compositae (Asteraceae) ........common yarrow..............Achillea millefolium....................39
Compositae (Asteraceae) ........eastern baccharis...........Baccharis halimifolia .................30
Compositae (Asteraceae) ........white snakeroot..............Eupatorium rugosum..................31
Cruciferae (Brassicaceae).......mustard..........................Brassica spp...............................40
Ericaceae ................................fetterbush.......................Leucothoe axillaris.......................5
Ericaceae ................................fetterbush.......................Leucothoe racemosa ....................5
Ericaceae ................................fetterbush.......................Leucothoe recurva .......................6
Ericaceae ................................great laurel ....................Rhododendron maximum..............4
Ericaceae ................................maleberry.......................Lyonia ligustrina...........................6
Ericaceae ................................mountain laurel..............Kalmia latifolia .............................3
Ericaceae ................................rosebay...........................Rhododendron catawbiense..........4
Ericaceae ................................sheep laurel ...................Kalmia angustifolia.......................3
Euphorbiaceae ........................castor bean ....................Ricinus communis ......................27
Euphorbiaceae ........................Chinese tallowtree .........Sapium sebiferum ......................28
Gramineae (Poaceae)..............johnsongrass ..................Sorghum halepense ......................8
Hippocastanaceae...................red buckeye....................Aesculus pavia ...........................25
Hypericaceae ..........................St. John’s wort................Hypericum spp. ..........................41
Labiatae (Lamiaceae) .............perilla mint.....................Perilla frutescens .......................45
Lauraceae ...............................Mexican pricklepoppy ....Argemone mexicana ...................43
50
Leguminosae (Fabaceae) ........black locust ....................Robinia pseudoacacia.................20
Leguminosae (Fabaceae) ........bladderpod .....................Glottidium vesicarium ................15
Leguminosae (Fabaceae) ........coffee senna ...................Senna occidentalis .....................16
Leguminosae (Fabaceae) ........hairy vetch .....................Vicia villosa ................................44
Leguminosae (Fabaceae) ........rattlebox.........................Sesbania punicea .......................18
Leguminosae (Fabaceae) ........Scotch broom .................Cytisus scoparius .......................19
Leguminosae (Fabaceae) ........sesbania .........................Sesbania exaltata .......................17
Leguminosae (Fabaceae) ........showy crotalaria.............Crotalaria spectabilis ...................1
Leguminosae (Fabaceae) ........sicklepod ........................Senna obtusifolia........................16
Leguminosae (Fabaceae) ........sweet clover ...................Melilotus spp..............................18
Liliaceae..................................fly poison........................Amianthium muscaetoxicum ......37
Loganiaceae ............................yellow jessamine ............Gelsemium sempervirens.............9
Meliaceae................................chinaberry......................Melia azederach .........................21
Phytolaccaceae .......................pokeweed .......................Phytolacca americana ................10
Pteridaceae .............................bracken fern...................Pteridium aquilinum.....................2
Ranunculaceae........................buttercup........................Ranunculus sardous ...................12
Rosaceae.................................black cherry ...................Prunus serotina............................7
Rosaceae.................................choke cherry ..................Prunus virginiana.........................8
Rosaceae.................................laurel cherry ..................Prunus caroliniana.......................7
Rubiaceae................................common buttonbush .......Cephalanthus occidentalis..........39
Solanaceae..............................black nightshade ............Solanum nigrum .........................24
Solanaceae..............................horsenettle .....................Solanum carolinense ..................23
Solanaceae..............................jimsonweed ....................Datura stramonium....................11
Umbelliferae (Apiaceae)..........poison hemlock...............Conium maculatum ....................27
Umbelliferae (Apiaceae)..........spotted water hemlock ...Cicuta maculata .........................26
Verbenaceae............................lantana ...........................Lantana camara .........................13
51