Abstracts Poster Verona2015
Abstracts Poster Verona2015
Abstracts Poster Verona2015
(1) School of Psychology - University of Birmingham - United Kingdom | (2) School of Psychology University of Birmingham - UK
Email: [email protected] | [email protected]
Presenter and Poster Info
SEPARATE AND COMBINED EFFECTS OF NALTREXONE AND EXTENDEDRELEASE ALPRAZOLAM ON THE REINFORCING, SUBJECT-RATED, AND
PHYSIOLOGICAL EFFECTS OF METHAMPHETAMINE
Pike, Erika (1) | Marks, Katherine, R (1) | Lile, Joshua, A (1) | Stoops, William, W (1) |
Glaser, Paul, E A (1) | Hays, Lon, R (1) | Rush, Craig, R (1)
Opioid antagonists (e.g., naltrexone) and positive modulators of GABA-A receptors (e.g.,
alprazolam) modestly attenuate the abuse-related effects of stimulants. Side effects preclude
the use of higher doses to achieve greater efficacy. Combining naltrexone and extended-release
alprazolam (alprazolam XR) might maximize efficacy while avoiding the untoward effects of
higher doses of the constituent compounds.
This study tested the influence of naltrexone and alprazolam XR on the reinforcing, subjectrated, and physiological effects of methamphetamine. We hypothesized that maintenance on
the combination of naltrexone and alprazolam XR would be well tolerated, decrease selfadministration, and reduce the positive subject-rated drug-effects of methamphetamine to
a greater extent than the constituent drugs alone.
Eight non-treatment-seeking, stimulant-users were maintained on placebo, naltrexone (50 mg),
alprazolam XR (1 mg), and a combination of naltrexone and alprazolam. Participants completed
three sessions under each maintenance condition during which they first sampled one of three
doses of intranasal methamphetamine (0, 10, and 30 mg) and completed physiological and
subjective effects measures. Participants then had the opportunity to respond on a progressive
ratio task to earn all, some, or none of the sampled methamphetamine dose.
Methamphetamine produced prototypical physiological and stimulant-like positive subjective
effects (e.g., Like Drug). Naltrexone maintenance decreased self-administration of 10 mg
methamphetamine compared to all other maintenance conditions. Naltrexone and alprazolam
XR alone each significantly attenuated some of the subjective effects of methamphetamine.
The combination did not attenuate methamphetamine self-administration or subjective effects.
Overall, the results support the continued evaluation of naltrexone for methamphetamine
dependence, with particular attention to the identification of other drugs that might enhance
its ability to reduce drug-taking behavior.
This research was supported by NIDA Grants R01 DA025591 (CRR), T32 DA035200 (CRR and KRM),
K02 DA031766 (JAL) and CTSA Grants UL1 TR000117 and UL1 TR000115 (KRM).
(1) University of Kentucky - USA
Email: [email protected] | [email protected] | [email protected] | [email protected] |
[email protected] | [email protected] | [email protected]
Aims: Orexigenic peptides ghrelin and orexin exhibit central neurobiological effects which are
involved in food intake, reward, learning and mood. Both peptides elicit mostly pro-anxiety
effects. Thus we wanted to test the potential anxiolytic-like effects of the ghrelin receptor
antagonist (JMW1959) and orexin receptor antagonist (SB334867) which has yet to be done and
for the first time use the aversive ultrasonic vocalization (AUV) in rats for testing these peptides
mechanisms participation.
Methods: The automatic chamber ANL-937-1 Ultrasonic Vocalization Detector (Med Associates
Inc.) was used with bat detector set to 22 kHz (corresponding to rat's aversive ultrasonic
communication). One day before the experiment the aversive behavior was established (i.e.
rats were individually exposed in the chamber for 7min to 6 electro-shocks). The aversion was
fixed on the next day (1 shock during 1min exposure), and thereafter antagonists JMW2959 (0.6,
6; 12 mg/kg) and SB334867 (1; 10; 20 mg/kg) or ghrelin (30; 300 g/kg) or saline were i.p.
applied. Thirty minutes later the rats AUV was measured again in the chamber for 10min (no
electro-shocks). The anxiolytic-like effects were considered compared to the control/saline
rats (6 - 10 rats in the groups).
Results: The ghrelin antagonist significantly decreased AUV in doses 6 mg/kg (decrease of
74.2%) and 12 mg/kg (decrease of 62.8%) versus control. The observed SB33487-induced
anxiolytic-like and ghrelin-induced anxiety-like effects were not significant.
Conclusion: We have found that ghrelin antagonist significantly decreased the rats AUV, which
supports the idea, that ghrelin antagonism might be used for prevention of distress and anxietyinduced overeating and drug consumption.
Acknowledgements: This study was supported by PRVOUK34, 260168/SVV/2015, GAUK 54313.
(1) Department of Pharmacology - Third Faculty of Medicine - Charles University in Prague - Czech
Republic
Email: [email protected] | [email protected] | [email protected] |
[email protected]
Presenter and Poster Info
PREVENTION AND REVERSAL OF ESCALATED COCAINE SELFADMINISTRATION BY CRF R1 ANTAGONIST IN SOCIALLY DEFEATED MICE
Han, Xiao (1) | DeBold , Joseph (1) | Miczek, Klaus (1)
Social defeat stress can result in escalated cocaine self-administration in rodents and also
induces long-term adaptations in the mesolimbic dopamine system. Corticotrophin releasing
factor (CRF) has been shown to be closely associated with stress-induced escalation of drug use.
CRF receptor 1 (CRF R1) antagonists can block intermittent social defeat stress-induced cocaine
binge in rats. However, the role of CRF R1 during different phases of stress-induced cocaine
self-administration still remains to be defined. The current study examines the effects of CRF
R1 antagonist on escalated intravenous cocaine self-administration as a result of exposure to
social defeat stress in mice. (1) CRF R1 antagonism (CP 376,395, 15mg/kg) given 30 min prior
to each social defeat episode prevented stress-induced cocaine self-administration in mice. (2)
Administration of CP 376,395 (5 mg/kg and 15 mg/kg) 10 days after the last episode of social
stress and dose dependently reversed the escalation of cocaine intake. (3) To further explore
the role of CRF R1 in specific brain sites, CP 376,395 (0.5 g/ 0.2 l and 1 g/ 0.2 l) was
delivered directly into the ventral tegmental area (VTA) 10 min before cocaine selfadministration session 10 days after the last stress. Intra-VTA antagonism of CRF R1 was
sufficient to reverse social defeat stress-induced escalated cocaine self-administration during
the expression phase. These findings suggest that CRF R1 exerts multiple roles during initial
reaction to social stress and in long-term neuroadaptations that are relevant to escalated
cocaine self-administration, providing a potential target for therapeutic inventions for stressinduced drug use disorders.
Acknowledgement:
This research is supported by NIDA grant # DA 03174, PI. Klaus A. Miczek. The authors declare
no conflict of interest.
(1) Tufts University - USA
Email: [email protected] | [email protected]
Presenter and Poster Info
Halfon2,
Pierre J. Magistretti1,3,4,
(1) Research Group Developmental Neuropsychopharmacology - CIMH Mannheim - Germany | (2) CIMH
| (3) Institute Physiological Chemistry - University Mainz - Germany
Email: [email protected]
Presenter and Poster Info
(1) Universitat Pompeu Fabra-Spain | (2) IMIM-Spain | (3) Universitat Autnoma de Barcelona-Spain
Email: [email protected] | [email protected] | [email protected] | [email protected] |
[email protected] | [email protected]
Presenter and Poster Info
Gracia-Rubio I | [email protected]
Monday 14th September
Wildtype (WT) and NPY knockout (NPY KO) mice housed either under standard
environment (SE) or EE were submitted to a behavioural battery consisting of the elevated plus
maze (EPM), open field test (OF), stress-induced hyperthermia (SIH) and forced swim test (FST)
after 9 weeks of differential housing. One day after the last behavioural test amygdalar and
hippocampal NPY levels were measured by PCR and ELISA.
Methods:
EE-housed WT mice made significantly more entries to the open arms of the EPM
compared to SE-housed WT mice, which indicates an anxiolytic effect of EE. Importantly, this
beneficial effect was not seen in NPY KOs. In contrast in the OF and the FST, NPY KOs showed
increased anxiety, reduced locomotor activity and depression-like behaviour independent of
housing conditions. Housing itself also affected FST behaviour as both EE-housed WTs and NPY
KOs spent more time climbing. The SIH suggested a negative effect of EE for NPY KOs but not
WT animals as only EE-housed NPY KOs had higher stress-induced rectal temperatures.
Molecular analysis of the WT brains revealed increased amygdalar and hippocampal NPY gene
expression but no change in the corresponding peptide levels suggesting higher NPY turnover in
EE-housed mice.
Results:
The current molecular and behavioural data favour the contention that NPY
contributes to the anxiolytic effects of EE. The absence of NPY abolishes this beneficial effect
and even induces negative effects in response to environmental stimulation.
Conclusion:
Research support: Supported by the Austrian Science Fund (FWF grant P 25912-B23).
(1) Medical University of Graz - Austria | (2) Garvan Institute of Medical Research - Australia
Email: [email protected] | [email protected] | [email protected] |
[email protected]
Presenter and Poster Info
PATTERN AND PATH ENCODING: NEW TOOL FOR UNWELDING VISUOSPATIAL MEMORY THROUGH THE USE OF A SENSORIZED PLATFORM
Lupo, Michela (1,2) | Tedesco, Anna Maria (1,2) | Aloise, Fabio (3) | Ferlazzo, Fabio (1) |
Cardillo, Chiara (1) | Leggio, Maria (1,2)
It has been demonstrated that the processing of visuospatial information in reaching or
navigational space is supported by independent systems.
Moreover, it has been proposed that visuospatial information may require a pattern or a path
encoding. The pattern encoding is specifically used when the task requires a simultaneous
processing of spatial positions, while the path encoding is used in sequential processing that
allows to link different spatial positions in correct succession.
The aim of the present study was to disentangle these different components of visuospatial
information processing in navigational tasks by the development of an innovative sensorized
platform, and to verify if subjects use the same strategies when similar tasks are performed in
reaching space. We assessed the performance of 70 healthy volunteers on the sensorized
platform to investigate topographical orientation (route memory) in three different tasks that
required a sequential strategy (named Route A and Route B tasks, which differ for sequential
load required for their implementation) or a simultaneous strategy (named Simultaneous
Walking Test).
Subsequently, similar tasks were performed in reaching space, (by means Corsi Block tapping
Test and a Modified Corsi Test for the sequential strategy and Simultaneous Paper Test for the
simultaneous strategy).
The six tasks were submitted to factor analysis (Principal Axis Factoring). This analysis showed
four factors that explaining the 51.3% of the total variance: Corsi Block tapping Test (.501);
Simultaneous Paper and Walking Test (-.74 and -.51, respectively); Route A (-.38); Modified
Corsi Test and the Route B (.72 and .67, respectively). This clustering demonstrates that - the
sensorized platform, set up in the present study, allows to identify the different strategies
performed to correctly solve visuo-spatial tasks in navigational space - the same strategies can
be also used in reaching space according to the task demand.
(1) Department of Psychology - Sapienza University - Rome - Italy | (2) Ataxia Laboratory - IRCCS Santa
Lucia Foundation - Rome - Italy | (3) Alfameg s.r.l. - Rome - Italy
Email: [email protected] | [email protected] | [email protected] |
[email protected] | [email protected] | [email protected]
Presenter and Poster Info
(1) Department of Cognitive Neuroscience - Donders Institute for Brain Cognition and Behaviour Nederland | (2) Department of Psychiatry - Radboud University Medical Centre - Nederland
Email: [email protected] | [email protected] | [email protected] |
[email protected] | [email protected]
Presenter and Poster Info
THE NEUROTENSIN NTS1 RECEPTOR AGONIST PD149163 REDUCES FEARPOTENTIATED STARTLE IN MALE AND FEMALE MICE
Vanden Avond, Mark, A (1) | Smith, Claire (1) | Ritchie, Taylor (1) | Prus, Adam, J (1)
Neurotensin, a neuropeptide, has been associated with having anxiolytic effects, and systemic
administration of the NTS receptor agonist PD149163 has exhibited anxiolytic effects in male
rats. The present study sought to further evaluate the potential anxiolytic effects of PD149163
by assessing this compound in both male and female C57BL/J6 mice using the fear-potentiated
startle (FPS) paradigm. Startle chambers were equipped with a shock-grid floor, fluorescent
light, and an acoustic startle speaker. Conditioning took place between the light and floor
shock, and test sessions measured startle to a 120 dB noise burst while the light was on (FPS)
or off. Startle magnitude did not differ between the male and female mice. PD149163
produced significant reduction in FPS, with greater effects occurring in the female mice. The
anxiolytic and partial 5-HT agonist buspirone produced a significant reduction in FPS, which
was found to be greater in the male mice. The reduction in FPS by PD149163 coincides with
previous studies conducted in male rats. The greater reduction in FPS found in female mice
suggests that more research is needed to examine the neurotensin system and sex differences.
Overall, these findings support targeting the neurotensin system for the development of novel
strategies for treating anxiety disorders.
(1) Northern Michigan University - USA
Email: [email protected] | [email protected] | [email protected] | [email protected]
Presenter and Poster Info
(1) Nencki Institute of Experimental Biology - Warsaw - Poland | (2) University of Physical Education Warsaw - Poland | (3) Military University of Technology - Warsaw - Poland
Email: [email protected] | [email protected] | [email protected] |
[email protected] | [email protected] | [email protected] | [email protected]
Presenter and Poster Info
KregielJakubJK | [email protected]
Sunday, 13th September
PROBABILISTIC SCHEDULES OF REWARD FOR ASSESSING THE ROLE OF 5HT2C RECEPTORS IN COGNITIVE FLEXIBILITY
Borton, Maxine L. (1) | Clifton, Peter G. (1)
Deficits in flexible cognition are associated with many neuropsychiatric conditions, yet remain
a challenge for existing treatments. Therefore animal models used to develop novel procognitive drugs must have high translational value. Probabilistic reversal learning (PRL) tasks,
which assess ability to reverse learned associations on the basis of both accurate and misleading
information, may allow more direct comparison to human paradigms than conventional tasks
that offer only accurate response feedback. In addition, they provide measures of reward and
punishment prediction accuracy and sensitivity through analysis of choice behaviour in response
to spurious feedback, which are of theoretical and clinical relevance. Although implemented
successfully in non-human primates and rats, it has been suggested that mice are challenged
by some aspects of these tasks. In order to further validate use of probabilistic tasks in mice
and also examine the role of 5-HT 2C receptors in flexible cognition, this study examined the
effect of the 5-HT 2C receptor antagonist SB242084 (0.5mg/kg) on PRL task performance in
C57BL/6J mice. Probability of inaccurate feedback was set at a threshold of p = .02 (2/10
correct responses punished, 2/10 incorrect responses rewarded) and the location of the correct
response was reversed over three serial tests. The majority of animals demonstrated successful
reversal learning, and both groups showed accurate reward prediction at both the correct and
incorrect location by late-stage reversal. Importantly the animals responded appropriately in
trials providing both accurate and misleading feedback. SB242084-treated mice showed
improved performance on the first reversal test, requiring fewer sessions and incorrect trials
to criterion compared with controls. The present study validates the use of PRL tasks in mouse
models, and identifies a more ecologically valid and sensitive tool for assessing the effects of
serotonergic manipulations on flexible responding. Research funded by a BBSRC (UK) Doctoral
Studentship.
(1) School of Psychology - University of Sussex - United Kingdom
Email: [email protected] | [email protected]
Presenter and Poster Info
DISSOCIATING
WANTING
AND
ANTICIPATED
LIKING
FROM
CONSUMMATORY LIKING IN SMOKERS WITH DIFFERENT LEVELS OF
NICOTINE DEPENDENCY
Selby, Danielle L (1) | Harrison, Amanda A (2) | Shepherd, Therese E (1) | Kolokotroni, Katerina Z (1)
The Incentive Sensitisation theory (Robinson and Berridge, 1993) suggests wanting and liking
are dissociable concepts, with wanting, but not liking typically increasing with repeated drug
use. Wanting is associated with anticipation of reward (before drug intake), whereas liking
relates to perceived pleasure derived from consummatory behaviour (during/after drug use).
However, numerous studies attempting to parse these components of reward have
misconceptualised liking as an anticipatory cognition, assessing how much participants think
they would like the reward, prior to consumption. This study explores whether levels of nicotine
dependency differentially effect wanting and liking responses to smoking-related cues, and
whether anticipated and consummatory liking are equivalent, and dissociable from wanting.
Heavy (HS, mean=16 cigarettes/day) and light non-daily (LS, mean=2 cigarettes/day) smokers
completed wanting and anticipated liking questionnaires pre- and immediately post-exposure
to smoking-related and neutral cues, and 45 minutes later (end of session). Consummatory liking
was measured post-session, whilst smoking.
Wanting and anticipated liking responses revealed identical patterns of results in HS and LS.
At baseline, no differences were seen between HS and LS on wanting or anticipated liking,
however immediately after exposure to both cues, and at the end of sessions, HS reported greater
drug wanting and anticipated liking than LS. Irrespective of smoking group, smoking-related
cues increased wanting and anticipated liking compared to neutral cues and this effect was
maintained until end of session. Conversely, HS and LS did not differ on consummatory liking.
Analyses confirmed the relationship between wanting and anticipated liking was significantly
stronger than wanting and consummatory liking or anticipated and consummatory liking.
Wanting and anticipated liking appear to be equivalent concepts measuring anticipation of
reward, both of which are dissociable from consummatory liking. Furthermore, heavier smoking
increases anticipatory desire for rewards, but not smoking pleasure. Future research attempting
to dissociate these concepts should ensure liking is appropriately measured after consumption.
(1) Psychology Department - Leeds Beckett University UK | (2) Institute of Psychological Sciences University of Leeds UK
Email: [email protected] | [email protected] | [email protected] |
[email protected]
Presenter and Poster Info
TOWARDS PHARMACOLOGICAL VALIDATION OF THE NOVELTYSUPPRESSED FEEDING TEST IN THE RAT AS A MODEL TO PREDICT THE
TIME-COURSE OF ANXIOLYTIC DRUG ACTION.
Auber, Alessia (1) | Guidi, Alessia (1) | Poffe, Alessandro (1) | Corsi, Mauro (2) | Gerrard,
Philip, A (1)
Currently available animal models of acute anxiolytic/antidepressant drug action provide useful
tools for drug discovery, they do not, however, allow investigation of temporal aspects needed
for clinical efficacy which may require several weeks (e.g. Serotonin Reuptake Inhibitors).
The aim of our study was to validate pharmacologically the Novelty-Suppressed Feeding Test
(NSFT), which has been suggested to exhibit predictive validity for time-course of anxiolyticeffects.
Rats were food-deprived for 23 hours, placed into a novel environment containing food and the
latency to begin eating was recorded.
Acute injection of Diazepam (2mg/Kg, i.p., 1 hour before test) reduced latency to eat compared
to controls (n=5; one-tailed t-test p<=0.05). Both acute and chronic treatment with Fluoxetine
(10mg/Kg, p.o., 1 hour before test,1 or 28 days respectively) tended to increase latency to eat
compared to controls (n=5-6 or n=13-14, one-tailed t-test p=0.05 or 0.06 respectively). In
summary, the well-known anxiolytic effect of Diazepam has been observed in the NSFT. Acute
Fluoxetine induces an anxiogenic-like effect and this is in line with the exacerbation of anxiety
symptoms reported by humans after a single administration of Fluoxetine. Chronic Fluoxetine
treatment also induced an anxiogenic-like effect which is in contrast with the anxiolytic-effect
observed in humans following chronic administration. Several studies have investigated the
effect of chronic Fluoxetine in different animal models of anxiety and contrasting results were
observed. It has been demonstrated that chronic Fluoxetine elicits different changes in stressed
mice compared to normal animals. This suggests that the anxiolytic effect of Fluoxetine, and
therefore the predictive validity of NSFT, may need to be assessed in animals subjected to
manipulations inducing a pathological state. Further experiment evaluating the effect of acute
vs. chronic Fluoxetine in stressed rats will be needed in order to set-up an animal-model with
predictive validity for the time-course of anxiolytic effect.
(1) Translational Pharmacology-Aptuit-Italy | (2) In Vitro Pharmacology-Aptuit-Italy
Email: [email protected] | [email protected] | [email protected] |
[email protected] | [email protected]
Presenter and Poster Info
(1) Department of Psychology and Behavioural and Clinical Neuroscience Institute - University of
Cambridge - United Kingdom | (2) Department of Neuroscience - University of Uppsala - Sweden (3)
New York University Medical Center - USA
Email: [email protected] | [email protected] | [email protected] | [email protected] |
[email protected] | [email protected] | [email protected]
Presenter and Poster Info
Roman-Lopez, P | [email protected]
Sunday, 13th September
(1) Sylics (Synaptologics BV) - The Netherlands | (2) VU University Amsterdam - The Netherlands
Email: [email protected] | [email protected] | [email protected] |
[email protected] | [email protected] | [email protected]
Presenter and Poster Info
Roman-Lopez, P | [email protected]
Sunday, 13th September
RELATIONSHIP BETWEEN BODY MASS INDEX, FAT MASS AND LEAN MASS
WITH FIBROMYALGIA IMPACT QUESTIONNAIRE IN A GROUP OF
FIBROMYALGIA PATIENTS
Roman-Lopez, P (1) | Sanchez-Labraca, N (1) | Estevez, AF (2) | Cardona, Diana (1)
Patients suffering from fibromyalgia (FM) had widespread musculoskeletal pain and stiffness,
fatigue, sleep disorders, cognitive impairment and other symptoms, which seriously affect their
quality of life evaluated by the Fibromyalgia Impact Questionnaire, making it difficult to
perform normal activities. Moreover, FM has been associated with a higher prevalence of
overweight and obesity than in the general population. The objective of this study was to
evaluate the relationship between body mass index (BMI), fat mass (fM) and lean mass (lM) with
quality of life in a group of FM patients. 60 FM patients, members of different FM associations
from Almeria (Spain) participated in our study. Some anthropometric measures were taken like
weight, height, BMI, body fat mass and lean mass. Our results showed that BMI, fM and lM
correlated differently with several items of the Fibromyalgia Impact Questionnaire. The present
findings reveal some interesting relationships, which need to be further investigated to improve
the management of FM patients.
(1) Department of Nursing Physiotherapy and Medicine - University of Almeria - Spain | (2)
Department of Psychology - University of Almeria - Spain
Email: [email protected] | [email protected] | [email protected] | [email protected]
Presenter and Poster Info
Roman-Lopez P | [email protected]
Sunday, 13th September
Roman-Lopez P | [email protected]
Sunday, 13th September
(1) Translational Neuropsychiatry Unit - Department of Clinical Medicine - Aarhus University Denmark
Email: [email protected] | [email protected] | [email protected] |
[email protected]
Presenter and Poster Info
A DOUBLE-COIL TMS STUDY OF IPSILATERAL DORSAL PREMOTORPREMOTOR CORTEX INTERACTION DURING ACTION PREPARATION
Luigi, Cattaneo (1) | Sara, Parmigiani (1) | Guido, Barchiesi (1)
Introduction: The ability to withhold a forthcoming action and to release it when appropriate
is a crucial feature in the life of a primate. In the present series of experiments we investigate
the possibility that in humans the dorsal premotor cortex (PMCd) could play a role in such
behavior. To do so we first devised a protocol to assess in vivo the ipsilateral PMCd-motor cortex
(M1) connectivity. We then tested the modulation of such connectivity in a delayed simplechoice conditional associative task, and in a delayed dual-choice conditional associative task.
Methods: we tested short-latency interactions between PMCd and the ipsilateral M1 with the
dual coil technique. The conditioning TMS (cTMS) was delivered over PMCd and the test TMS
(tTMS) was delivered over the mouth-related M1. Motor Evoked potentials were recorded via
surface EMG from the lips. cTMS+tTMS trials were intermingled with tTMS only trials. This
procedure was carried out in the first experiment at rest and in the second experiment during
the delay time in a single and dual choice delayed tasks.
Results: the first experiment provided evidence that PMCd exerts a powerful inhibitory effect
during rest on the ipsilateral mouth-related M1. This interaction occurred at an interval
between cTMS and tTMS of 6 ms. The results of the following experiments indicated that the
PMCd exerts an inhibitory effect on M1 during the delay period, which is reversed into excitation
by the go-signal.
Comments: our data are compatible with the possibility that, in a delayed visual conditional
associative task, PMCd is the premotor structure that actually inhibits the motor cortex during
the set period and possibly releases the programmed movement when the go-signal occurs.
(1) University of Trento - Italy
Email: [email protected] | [email protected] | [email protected]
Presenter and Poster Info
Rejlova M. | [email protected]
Sunday, 13th September
Gandolfi M. | [email protected]
Sunday, 13th September
Chauveau F | [email protected]
Tuesday 15th September
(1) Department of Zoology and Developmental Neurobiology - Institute of Biology - Otto von Guericke
University Magdeburg - Magdeburg, Germany
Email: [email protected]
Presenter and Poster Info
Banasikowski T. J. | [email protected]
Sunday, 13th September
(1) Institute of Physiology Academy of Science - Czech Republic | (2) National Institute of Mental
Health - Czech Republic
Email: [email protected]
Presenter and Poster Info
(1) Department of Neurological and Movement Sciences - University of Verona - Verona - Italy. | (2)
Morton and Gloria Shulman Movement Disorders Clinic and the Edmond J. Safra Program in Parkinsons
Disease - Toronto Western Hospital and Division of Neurology - University of Toronto - Toronto,
Ontario - Canada. | (3) Neuromotor and Cognitive Rehabilitation Research Centre (CRRNC) - University
of Verona - Italy. | (4) Department of Clinical and Experimental Medicine - University of Messina Italy. | (5) Neurology Unit - Department of Clinical and Experimental Medicine - University of Pisa. |
(6) Department of Neurological and Neurosensorial Sciences - Neurology and Neurohysiology Unit University of Siena - Italy. | (7) Department of Neuroscience and Imaging - University of ChietiPescara. (8) Aging Research Center Ce.S.I. University Foundation - Chieti Behavioural Neurology and
Movement Disorders Unit. | (9) Department of Neurology and Psychiatry - University of Rome Sapienza
- Rome - Italy (10) IRCSS Neuromed Institute - Pozzilli-Isernia - Italy. |
(11) Department of Geriatrics - Neuroscience and Orthopedics - Universit Cattolica del Sacro Cuore University Hospital Agostino Gemelli - Rome - Italy | (12) Department of Neurology - "F. Miulli"
General Hospital - Acquaviva delle Fonti (BA) - Italy | (13) Department of Neurology - AOB Brotzu Cagliari - Italy. | (10) | (14) Department G.F. Ingrassia, Area of Neurosciences; University of Catania,
Italy. | (15) Neurological Department and INSPE-Institute of Experimental Neurology, University
Hospital San Raffaele, Milan, Italy | (16) Division of Neurology, Civil Hospital Voghera (PV), Italy. |
(17) Department of Neurosciences, Rehabilitation, Ophthalmology, Genetics, Maternal and Child
Health, University of Genova, Italy | (18) U.O.C. Parkinson e Disordini del Movimento, IRCCS
Fondazione Istituto Neurologico Nazionale "C. Mondino" Pavia, Italy | (19) Neurology Unit, Ospedale
Misericordia, Grosseto, Italy | (20) Department of Basic Medical Sciences, Neuroscience and Sense
Organs, University of Bari "Aldo Moro", Italy. | (21) Parkinson Institute, Istituti Clinici di
Perfezionamento, Milan, Italy. | (22) Movement Disorders Center, Neurological Clinic, Department of
Public Health, Clinical and Molecular Medicine, University of Cagliari, Italy | (23) Department of
Systems Medicine, University of Rome Tor Vergata and IRCCS Santa Lucia Foundation, Rome, Italy. |
(24) Research Support and Biostatistical Unit, University Hospital Verona, Italy | (25) Center for
Neurodegenerative Diseasese (CEMAND), University of Salerno, Italy | (26) Department of Motor
Sciences and Health, University of Naples Parthenope, Italy | (27) IDC "Hermitage-Capodimonte",
Naples, Italy
Email: [email protected]
Presenter and Poster Info
The study was supported by the project National Institute of Mental Health (NIMH - CZ), grant number
ED2.1.00/03.0078 and the European Regional Development Fund, grant IGA MH CR no. NT/13897 and
grant MI CR no. VG/20122015080.