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    Rufat Rzayev
    This document discusses various voice and speech disorders including: 1. Dysphonia plica ventricularis which causes a rough, low pitched voice from sound being produced by the vestibular folds. 2. Functional aphonia which involves the vocal cords remaining in an abducted position during attempted phonation and is treated with reassurance and psychotherapy. 3. Vocal cord nodules which present as pinkish-white nodules at the junction of the anterior 2/3 and posterior 1/3 of the vocal cords.

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    Ent MCQ

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    Rufat Rzayev
    104 views22 pages
    This document discusses various voice and speech disorders including: 1. Dysphonia plica ventricularis which causes a rough, low pitched voice from sound being produced by the vestibular folds. 2. Functional aphonia which involves the vocal cords remaining in an abducted position during attempted phonation and is treated with reassurance and psychotherapy. 3. Vocal cord nodules which present as pinkish-white nodules at the junction of the anterior 2/3 and posterior 1/3 of the vocal cords.

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    an mcq about ent and radiology

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    ent-mcq

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    This document discusses various voice and speech disorders including: 1. Dysphonia plica ventricularis which causes a rough, low pitched voice from sound being produced by the vestibular folds. 2. Functional aphonia which involves the vocal cords remaining in an abducted position during attempted phonation and is treated with reassurance and psychotherapy. 3. Vocal cord nodules which present as pinkish-white nodules at the junction of the anterior 2/3 and posterior 1/3 of the vocal cords.

    Copyright:

    © All Rights Reserved
    Download as PDF, TXT or read online from Scribd
    Download as pdf or txt
    104 views22 pages

    Ent MCQ

    Uploaded by

    Rufat Rzayev
    This document discusses various voice and speech disorders including: 1. Dysphonia plica ventricularis which causes a rough, low pitched voice from sound being produced by the vestibular folds. 2. Functional aphonia which involves the vocal cords remaining in an abducted position during attempted phonation and is treated with reassurance and psychotherapy. 3. Vocal cord nodules which present as pinkish-white nodules at the junction of the anterior 2/3 and posterior 1/3 of the vocal cords.

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    of 22

    ENT MCQ

    by pg uplods

    VOICE AND SPEECH


    DISORDER
    Topics covered
    1.dysphonia plica ventricularis
    2.functional aphonia
    3.phonosthenia
    4.hyponasality(rhinolalia clausa)
    5.hypernasality(rhimolalia aperta)
    6.puberphonia
    7.vocal cord nodule
    8.others
    Q1.in dysphonia plica ventricularis,
    sound is produced by.................
    a. vestibular folds
    b.vocal folds
    c.tongue
    d.ventricle of larynsk
    Ans 1.....................a
    •
    false vocal cords= ventricular folds= vestibular
    folds(present avobe true vocal volds)
    •
    they hv minimal role in normal phonation
    •
    in this condition voice is= rough, low
    pitched,unpleasant
    •
    diagonesed by aproximation of fase vocal folds
    on INDIRECT LARYNGOSCOPY
    •
    this cond is also known as ventricular
    dysphonia
    Q2.pitch of persons voice depends
    upon fundamental frequency that
    depends upon larynsk, in normal
    adult male its avg value is
    •
    a.125 hz
    •
    b.210 hz
    •
    c. 300 hz
    •
    d. 400 hz
    ans ..a
    •
    FUNDAMENTAL FREQUENCY IS PRODUCED BY
    LARYNSK in normal adults its value is
    •
    MALE-125 hz
    •
    FEMALE-210 hz
    •
    CHILDREN-300 hz
    Q3.what is depth kymography...?
    •
    it is an imaging method to visualise complex
    movement of vocal cord
    •
    phonation is produced by occilation of vocal
    folds which is due to pressure diff across
    vocal folds, normal minimum value is- 3-5 cm
    H2O
    Q4. true about functional aphonia
    all except.........
    •
    1.seen in emotionally labile female of age 15
    -30 yrs
    •
    2.speech therapy is imp fr its treatment
    •
    3.vocal cord remain in abducted position on
    attempt of phonation
    •
    4. on coughing vocal cord remain abducted
    •
    5. aphonia is sudden in onset
    ans is 2. and 4.
    •
    treatment is done by reassurance and
    psychotherapy .speech therapy has no role
    •
    vocal cord canbe adducted during coughing ie
    adducter function is normal
    Q.5.rhinolalia clausa is seen in all
    except........
    •
    1.nasal growth
    •
    2.adenoids
    •
    3.nasal polyp
    •
    4. palatal paralysis
    ans 4
    •
    rhinolalia clausa is seen when nose or
    nasopharynsk is blocked
    •
    eg nasal growths(polyp)
    •
    nasopharyngioma
    •
    adenoids
    •
    allergic rhiitis,common cold
    note: clausa vs aperta
    •
    rhinolalia clausa= hyponasality= nose ka kam
    role hai= nose me obstruction hai
    •
    rhinolalia aperta=hypernasality= nose ka role
    jada= abnormal connection bw oral &nasal
    cavity eg palatal paralysis
    Q.6. a patient of phonasthenia
    shows elliptual space between
    vocal cords on indirect
    laryngoscopy, muscle involved is..
    •
    a.interaretenoid
    •
    b.thyroaretenoid
    •
    c.cricothyroid
    •
    d. all of avove
    ans ...b
    •
    phonosthenia is weakness or easy fatigability
    of voice due to weakness of thyroaretenoid
    ,interaretenoid or both
    •
    on indirect laryngoscopy signs space bw vocal
    cords is seen if its,
    •
    elliptual= thyroaretenoid
    •
    triangular gap bw post commisure=
    interaretenoid
    •
    keyhole= both(q)
    Q7.true about vocal cord nodule
    all except..............
    •
    a.localised epithelial hyperplasia
    •
    b.symmetric on both cord
    •
    c.male> female
    •
    d. most common age group 20 to 30
    •
    e. pinkish white nodule at jonction of anterior
    2/3 & posterior 1/3
    ans... e
    •
    most common site of...
    •
    a..vocal nodule...junction of ant 1/3 and posterior
    2/3
    •
    b..tb...posterir part of larynx
    •
    c..lupus..anterior larynx( epiglottis>aryepiglottic fold>
    ventricular band)
    •
    d..syphills..anterior larynx ie epiglottis and
    ariepiglottic fold
    •
    e..leprosy.. anterior larynx ie epiglottis & ariepiglottic
    fold
    •
    •
    h..vocal polyp..no difinitive site
    •
    i..intubation granuloma..post 1/3 & ant 2/3
    junction of vocal cord
    Q 8. treatment of spasmodic
    dysphonia is .........
    •
    a. type 1 thyroplasty
    •
    b. type 2 thyroplasty
    •
    c. type 3 thyroplasty
    •
    d. type 4 thyroplasty
    ans b
    •
    type 1 = medialisation of vocal cord = in
    unilateral vocal cord paralysis, atropy,sulcus
    vocalis
    •
    type 2= lateralisation = spasmodic dysphonia
    •
    type 3 = shortening or relaxation of vocal cord
    = for lowering of vocal pitch in puberphonia
    •
    type 4 = lenghtening of vocal cord for
    elevating pitch of voice as in androphonia
    Q9. pachydermia laryngis is
    treated by.....
    •
    1) cordectomy
    •
    2) microsurgical' excision
    •
    3) both '
    •
    4) none
    ans 2
    •
    treatment of ptachydermia laryngis is
    microsurgical excision of hyperplastic
    epithelium and cordectomy has no role in its
    treatment

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