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Pathway Gagal Ginjal Kronik: Intoleransi Aktivitas

This document summarizes the chronic kidney disease (CKD) pathway. It shows that CKD can be caused by infections, vascular issues like arteriosclerosis, or toxic substances accumulating in the kidneys. This leads to a decrease in the glomerular filtration rate and kidney function over time. As kidney function declines, it causes retention of sodium and water, decreased secretion of potassium, hyperkalemia, increased creatinine and BUN levels, metabolic acidosis, edema, fluid overload, and cardiovascular complications like arrhythmias and decreased cardiac output. Ultimately this pathway can result in anemia, decreased exercise tolerance, nutritional issues, and respiratory changes.

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Reza Apriandi
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Download as DOCX, PDF, TXT or read online on Scribd
292 views

Pathway Gagal Ginjal Kronik: Intoleransi Aktivitas

This document summarizes the chronic kidney disease (CKD) pathway. It shows that CKD can be caused by infections, vascular issues like arteriosclerosis, or toxic substances accumulating in the kidneys. This leads to a decrease in the glomerular filtration rate and kidney function over time. As kidney function declines, it causes retention of sodium and water, decreased secretion of potassium, hyperkalemia, increased creatinine and BUN levels, metabolic acidosis, edema, fluid overload, and cardiovascular complications like arrhythmias and decreased cardiac output. Ultimately this pathway can result in anemia, decreased exercise tolerance, nutritional issues, and respiratory changes.

Uploaded by

Reza Apriandi
Copyright
© © All Rights Reserved
Available Formats
Download as DOCX, PDF, TXT or read online on Scribd
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PATHWAY GAGAL GINJAL KRONIK Obs.

TU

Retensi urine

Infeksi Vaskuler Zat toksik


Refluks
Reaksi Ag - Ab Arterosklerosis Akumulasi di ginjal
Hidronefrosis

Suplai Vaskuler Ginjal


Peningkatan
tekanan
Iskemia
GFR turun
Kompresi
Gg. Fungsi renal Nefron Nekrosis
CKD

Penurunan fungsi Retensi Na Sekresi K ↓ Sekresi eritropoietin ↓


eksresi ginjal & H2O

Hiperkalemia Produksi Hb ↓
Sindrom uremia CES ↑

Tekanan kapiler naik Gg. Penghantaran Oksihemoglobin ↓


HCO3- ↓ kelistrikan jantung
S
Volume Intoleransi aktivitas
Asidosis intertisial naik
Disritmia

Mual, muntah
Edema Edema paru
↑ preload
Hiper-
ventilasi Risiko gangguan
nutrisi Kelebihan volume Gangguan SUMBER : Nanda. 2015. Nursing
↑ beban ↓ COP
cairan pertukaran gas
jantung Diagnoses Definition dan
Perubahan pola nafas
GGg. Cardiac Output Classification.

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