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Advance Cardiovascular Life
Support
Chanak Trikhatri
History
ACLS (Advacnce Cardiovascular Life support
was started formally since 1999 the concept
and practice was established by the American
Heart Association.
Definition
Advance Cardiovascular Life Support is systematic
design course for health care worker and provider
who either directly or participate in the
management of cardiopulmonary and
cardiovascular emergency.
Goal of ACLS
The main goal of ACLS is to support and
restore effective oxygenation, ventilation,
circulation with return of intact neurological
function
Systemic Approach
• If patient is unconscious use the BLS protocol
for initial assessment and treatment then use
the ACLS protocols
• If patient is conscious use ACLS protocols
directly
Anatomy
The heart structures that you must know in order
to understand ECG:
• Sino-atrial (SA) node (impulse generate 70/m
and velocity of conduction 0.05 m/sec)
• Atrium (singular is atrium)
• Atrio-ventricular (AV) node (impulse generate
60/m and velocity of conduction 0.05 m/sec)
Anatomy Cont…
• Ventricular conducting pathways (i.e. bundle
of His, right and left bundle branches (impulse
generate 36/m and velocity of conduction 1
m/sec))
• Ventricles
• Purkinje fiber (impulse generate 40/m and
velocity of conduction 4 m/sec)
ECG
Physiology
A normal heart beat follows this sequence:
1. the SA node generates an electrical current
2. this current travels through the atrium
3. the current then enters the AV node, where is
slows down for a little delay
4. the current enters the ventricular conduction
pathways where it travels rapidly and the
5. ventricles contract
6. and then the ventricles relax
ECG
ECG
Reading of ECG
1. Rate
2. Rhythm
3. P wave
4. PR interval
5. QRS complex
Normal Sinus Rhythm
Bradycardia
Any rhythm disorder with a heart rate < 60/min.
When bradycardia is the cause of the symptoms
the rate is generally <50/min. Sinus bradycardia is
caused by the SA node firing at a rate of less than
60 bpm. The remaining electrical activity
downstream is normal. Rates less than 60 bpm are
commonly found in healthy adults.
ECG
Ventricular Tachycardia ( VT)
Any abnormal disorder with an abnormal fast
heart beat at a rate of 100-250 beat per
minute that are initiated in the ventricles and
it does not show any response to pressure on
carotid artery
Ventricular Tachycardia (VT)
Ventricular Fibrillation (VF)
When part of the heart fibrillates, there is no
coordinated contraction. However, there is a very
high amount of uncoordinated electrical activity
which results in quivering. It is this quivering that
gives rise to the noisy ECG signal in ventricular
fibrillation. There are no ventricular contractions
and therefore no cardiac output, no pulse, and no
blood pressure.
Ventricular Fibrillation (VF)
Asystole
Asystole is the absence of any systolic function.
Therefore, there are no atrial or ventricular
contractions. It is a flat line. Asystole is the last
stage of deterioration of the heart. In an
“electrically failing” heart, not all steps will
necessarily occur (i.e. could go from bradycardia
straight to asystole)
Asystole
Pulseless Electrical Activity
PEA encompasses a heterogeneous group of
rhythms that are organized or semiorganized but
lack a palpable pulse includes: Idioventricular
rhythms, Ventricular escape rhythms,
Postdefibrillation idioventricular rhythms, Sinus
rhythm. Even sinus rhythm without a detectable
pulse is called PEA.
Cardiac Arrest Algorithm
ECG
Drug Therapy
• Epinephrine / Adrenalin IV/IO dose: 1 mg
every 3-5 minute
• Vasopressin IV/IO dose: 40 units can replace
the first or second dose of epinephrine
• Amiodarone IV/IO dose: First dose 300mg
bolus, second dose 150 mg
Shock Energy
• Monophasic : 360 J
• Biphasic: 120-200 J
Reversible Causes
• Hypovolemia
• Hypoxia
• Hydrogen Ion acidosis
• Hypo/hyperkalemia
• Hypothermia
• Tension pneumothorax
• Tamponade cardiac
• Toxins
• Thrombosis pulmonary
• Thrombosis coronary
Do Not Attempt to resuscitate
• Rigor Mortis
• Written Documents
• Threat to safety of providers
Chest Pain
Chest pain or Acute coronary syndrome due
to poor supply of the oxygenated blood to the
myocardal tissue which lead to poor perfusion
then ischemia develop and cause severe chest
pain.
Management of Chest Pain
• Assess the symptoms suggestion ischemia or
myocardial infraction.
• Emergency assessment, care and hospital
prepartion.
• Monitor, support ABC be prepared to provide
CPR and defibrillation
• Administer (MONA) Morphin, Oxygen,
Nitroglycerin, Aspirin if needed
Management of Chest Pain
• Obtain 12 lead ECG
• Oxygen saturation 94% start at 4 L/min titrate
• Aspirin 160 to 325 mg if not given by
emergency
• Nitroglycerin sublingual or spray
• Morphin IV if discomfort is not relieved by
nitroglycerin
ECG
Summary
Advance Cardiovascular Life Support is design
protocol which help patient to support and
recover from the cardiovascular and
cardiopulmonary emergency and help to
return to the intact neurological function with
the help of health care provider who are
skilled and expertise on ACLS practice.
Thank
You
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ECG

  • 2. History ACLS (Advacnce Cardiovascular Life support was started formally since 1999 the concept and practice was established by the American Heart Association.
  • 3. Definition Advance Cardiovascular Life Support is systematic design course for health care worker and provider who either directly or participate in the management of cardiopulmonary and cardiovascular emergency.
  • 4. Goal of ACLS The main goal of ACLS is to support and restore effective oxygenation, ventilation, circulation with return of intact neurological function
  • 5. Systemic Approach • If patient is unconscious use the BLS protocol for initial assessment and treatment then use the ACLS protocols • If patient is conscious use ACLS protocols directly
  • 6. Anatomy The heart structures that you must know in order to understand ECG: • Sino-atrial (SA) node (impulse generate 70/m and velocity of conduction 0.05 m/sec) • Atrium (singular is atrium) • Atrio-ventricular (AV) node (impulse generate 60/m and velocity of conduction 0.05 m/sec)
  • 7. Anatomy Cont… • Ventricular conducting pathways (i.e. bundle of His, right and left bundle branches (impulse generate 36/m and velocity of conduction 1 m/sec)) • Ventricles • Purkinje fiber (impulse generate 40/m and velocity of conduction 4 m/sec)
  • 9. Physiology A normal heart beat follows this sequence: 1. the SA node generates an electrical current 2. this current travels through the atrium 3. the current then enters the AV node, where is slows down for a little delay 4. the current enters the ventricular conduction pathways where it travels rapidly and the 5. ventricles contract 6. and then the ventricles relax
  • 12. Reading of ECG 1. Rate 2. Rhythm 3. P wave 4. PR interval 5. QRS complex
  • 14. Bradycardia Any rhythm disorder with a heart rate < 60/min. When bradycardia is the cause of the symptoms the rate is generally <50/min. Sinus bradycardia is caused by the SA node firing at a rate of less than 60 bpm. The remaining electrical activity downstream is normal. Rates less than 60 bpm are commonly found in healthy adults.
  • 16. Ventricular Tachycardia ( VT) Any abnormal disorder with an abnormal fast heart beat at a rate of 100-250 beat per minute that are initiated in the ventricles and it does not show any response to pressure on carotid artery
  • 18. Ventricular Fibrillation (VF) When part of the heart fibrillates, there is no coordinated contraction. However, there is a very high amount of uncoordinated electrical activity which results in quivering. It is this quivering that gives rise to the noisy ECG signal in ventricular fibrillation. There are no ventricular contractions and therefore no cardiac output, no pulse, and no blood pressure.
  • 20. Asystole Asystole is the absence of any systolic function. Therefore, there are no atrial or ventricular contractions. It is a flat line. Asystole is the last stage of deterioration of the heart. In an “electrically failing” heart, not all steps will necessarily occur (i.e. could go from bradycardia straight to asystole)
  • 22. Pulseless Electrical Activity PEA encompasses a heterogeneous group of rhythms that are organized or semiorganized but lack a palpable pulse includes: Idioventricular rhythms, Ventricular escape rhythms, Postdefibrillation idioventricular rhythms, Sinus rhythm. Even sinus rhythm without a detectable pulse is called PEA.
  • 25. Drug Therapy • Epinephrine / Adrenalin IV/IO dose: 1 mg every 3-5 minute • Vasopressin IV/IO dose: 40 units can replace the first or second dose of epinephrine • Amiodarone IV/IO dose: First dose 300mg bolus, second dose 150 mg
  • 26. Shock Energy • Monophasic : 360 J • Biphasic: 120-200 J
  • 27. Reversible Causes • Hypovolemia • Hypoxia • Hydrogen Ion acidosis • Hypo/hyperkalemia • Hypothermia • Tension pneumothorax • Tamponade cardiac • Toxins • Thrombosis pulmonary • Thrombosis coronary
  • 28. Do Not Attempt to resuscitate • Rigor Mortis • Written Documents • Threat to safety of providers
  • 29. Chest Pain Chest pain or Acute coronary syndrome due to poor supply of the oxygenated blood to the myocardal tissue which lead to poor perfusion then ischemia develop and cause severe chest pain.
  • 30. Management of Chest Pain • Assess the symptoms suggestion ischemia or myocardial infraction. • Emergency assessment, care and hospital prepartion. • Monitor, support ABC be prepared to provide CPR and defibrillation • Administer (MONA) Morphin, Oxygen, Nitroglycerin, Aspirin if needed
  • 31. Management of Chest Pain • Obtain 12 lead ECG • Oxygen saturation 94% start at 4 L/min titrate • Aspirin 160 to 325 mg if not given by emergency • Nitroglycerin sublingual or spray • Morphin IV if discomfort is not relieved by nitroglycerin
  • 33. Summary Advance Cardiovascular Life Support is design protocol which help patient to support and recover from the cardiovascular and cardiopulmonary emergency and help to return to the intact neurological function with the help of health care provider who are skilled and expertise on ACLS practice.