Disease and metabolism

A biosensor was designed where a dehydrogenase and an enlarged coenzyme are confined behind an ultrafiltration membrane. The amino acid is determined indirectly, by measuring the fluorescence of the reduced coenzyme (kex 360 nm, kfl 460 nm) produced in reaction 22, with the aid of an optical fiber. The coenzyme is regenerated with pyruvate in a subsequent step, as shown in reaction 23. This biosensor was proposed for determination of L-alanine and L-phenylalanine for monitoring of various metabolic diseases and for dietary management363. [Pg.1103]

Bender PE, Lee JC., eds. Pharmacological modulation of interleukin-1 (1989). Annual reports in medicinal Chemistry-25. Johns. Section IV-Metabolic diseases and endocrine function. Chapter 20. [Pg.433]

Abstract This chapter will reviews the Raman spectroscopy of the subject tissues. After a brief introduction to the structure, biology, and function of these tissues, we will describe the spectra and band assignments of the tissues and then summarize applications to studies of tissue development, mechanical function and competence, and pathology. Both metabolic diseases and genetic disorders will be covered. [Pg.347]

Mundy G. R. (1999) Bone remodeling. In Primer on the Metabolic Diseases and Disorders of Mineral Metabolism, 4th edn. (ed. M. J. Favus). Lippincott Wilhams and Wilkins, Philadelphia, pp. 30-38. [Pg.4047]

Several compounds, such as creatinine and purine derivatives (allantoin, uric acid, hypoxanthine, and xanthine) present in biological samples, are important analytes for diagnoses of certain types of metabolic diseases and can serve as markers for these processes. Analyses for such substances are crucial for the diagnosis and monitoring of renal diseases, metabolic disorders, and various types of tumorigenic activity. [Pg.465]

Sukumaran S, Jusko WJ, Dubois DC, Almon RR (2011) Light-dark oscillations in the lung transcriptome implications for lung homeostasis, repair, metabolism, disease, and drug action. J Appl Physiol 110 1732-1747... [Pg.34]

Section IV - Metabolic Diseases and Endocrine Function Editor Hans-JUrgen Hess, Pfizer, Inc., Croton, Conn. [Pg.152]

After having clarified many basic aspects of lipolysis (i.e. structure, function and regulation of lipases and their catalytic mechanism) the focus will shift to the pathophysiological role of lipases in metabolic diseases and will result in strategies for pharmacological intervention. [Pg.352]

In 1954 Krebs was appointed the Whitley Chair of Biochemistry at Oxford University. That same year he received the Royal Medal of the Royal Society of London. In 1958, for his scientific work and his contributions to the lives of British people, Krebs was knighted. Even after his retirement in 1967, he continued to do research on fiver disease, the genetic bases of metabolic diseases, and the link between poor nutrition and juvenile delinquency. In addition to his Nobel Prize and Royal Medal, he received honorary degrees ftom nine universities, see also Glycolysis Krebs Cycle. [Pg.709]

Endocrine, nutritional and metabolic diseases, and immunity disorders... [Pg.38]

This investigation has been supported in part by Public Health Service Research Grants CA-04222 from the National Cancer Institute and AM-10333 from the National Institute of Arthritis and Metabolic Diseases, and by grants from the Jane Coffin Childs Memorial Fund for Medical Research, the Squibb Institute of Medical Research and the Scientific Research Fund of the Ministry of Education of Japan. [Pg.185]

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