Phosphate
Phosphate
Phosphate
Angeles City
COLLEGE OF ALLIED MEDICAL PROFESSIONS
PHOSPHATE
Submitted by:
Group 1
BSMT 3-D
Group Leader: Beltran, Cherish Inna S.
Members: Balatbat, Allyson M.
De Jesus, Roselle Ivy L.
Miranda, Alda Janelle M.
Submitted to:
Maam Anna Kamile Suyat
Maam Engracia Arceo
CC02 Laboratory Professors
I.
Physiologic
II.
Clinical Significance
Hypophosphatemia
1. Alcohol abuse most common cause
2. Primary hyperparathyroidism
3. Avitaminosis D
4. Myxedema
Hyperphosphatemia
1. Hypoparathyroidism
2. Renal failure (tubular failure)
3. Lymphoblastic leukemia
4. Hypervitaminosis D
Patients who are at greater risk for hyperphosphatemia are those with acute or chronic
renal failure. An increase intake of phosphate or increase release of cellular phosphate
may also cause hyperphosphatemia. Neonates are especially susceptible to
hyperphosphatemia caused by increased intake, such as from cow's milk of laxatives.
Increase breakdown of cells can sometimes lead to hyperphosphotemia, as with severe
infections, intensive exercise, neoplastic disorders or intravascular hemolysis.
Hypoparathyroidism may also cause hyperphosphatemia.
III.
SERUM
Neonate
Children 15y
Adult
Urine (24h)
IV.
Important Notes
1.45-2.91 mmol/L
1.07-1.74 mmol/L
0.78-1.42 mmol/L
13-42 mmol/L
4.5-9.0 mg/dL
3.3-5.4 mg/dL
2.4-4.4 mg/dL
1.4.1.3
g/d
V.
References