Gastritis
Gastritis
1. Relief of pain:
Encourage clients to learn relaxation techniques
Encourage clients to avoid foods and beverages that irritate the stomach, such as alcohol
Encourage clients to use diet pd regular intervals.
2. Maintaining adequate nutrition remains
Provide eat small but frequent meals and do not irritate.
Give solid foods as soon as possible
Provide a drink that contains no caffeine
3. Hyperthermia
Monitor vital signs every 2 hours
Apply a cold compress
Management of giving antipyretics as indicated
4. Maintain body fluid volume
Observation of fluid intake and output
Observe for signs of dehydration
5. Reduce anxiety
Encourage clients to express their problems and fears
Help clients identify situations that cause anxiety
Teach stress management strategies
6. Increase the client's knowledge about the disease
Assess client's level of knowledge
Provide the required information by using the right words and the corresponding time
Reassure the client that the disease can be overcome.
Nursing Interventions:
• Review the level of pain.
• Provide information about the different strategies chosen to reduce pain.
• Encourage clients to use the chosen strategy to reduce pain.
• Encourage clients to avoid eating foods that stimulate an increase in stomach acid.
• Collaboration with the medical team for the administration of anti-analgesic.
Rational:
• In order to determine the level of pain experienced by the client.
• Able to learn methods of pain reduction and can do it.
• Assist in menurunhkan experienced pain threshold.
• In order for clients to find foods that stimulate stomach acid and does not consume
them.
• Reduce the level of pain experienced by the client.
Nursing Interventions:
• Describe the client and family about the importance of food for the body.
• Monitor the amount of food intake.
• Monitor and record the number of vomiting, frequency and color
• Provide a varied diet according to his diet to stimulate appetite.
• Provide food in small portions but frequently.
• Collaboration with the medical team for the administration of anti-emetic drugs.
Rational
• Clients and families can learn the importance of
• To know the food is consumed.
• As the data to perform nursing actions and subsequent treatment.
• To klirn be motivated and stimulates appetite.
• To reduce the feelings and needs food for patients.
• As a therapy for inhibiting / stimulating nausea and vomiting.
Nursing Interventions:
· Assess the possibility of signs of dehydration and record intake and output.
· Assess the balance of fluids and electrolytes every 24 hours.
· Encourage clients to keep the peroral intake is to eat and drink a little but often.
· Encourage clients to avoid consuming foods and beverages that contain caffeine.
Rational:
· Detecting the early signs of dehydration.
· Detecting early indicator of fluid and electrolyte imbalance.
· In order for the client's body fluid balance can be maintained.
· Caffeine is a central nervous system stimulant that can increase the activity of gastric
and pepsin secretion leading to increased secretion of gastric acid that can cause
reactions of nausea and vomiting.
Purpose: No Anxiety
Nursing Interventions:
• Assess the client's anxiety.
• Give the client an opportunity to express his anxiety.
• Explain to clients that can challenge dijalankankan diet after recovery.
• Explain to the client about medical procedures / treatments will be done and
encouraged cooperative therein.
• Provide motivation to the client about his recovery.
Rational:
• As the initial data to determine the client's anxiety level.
• In order to determine the cause of anxiety is experienced as well as reduce the
psychological burden of the client.
• The client can adhere to diet and avoid disease relapse again.
• Able to understand and accept all the measures taken to cure the disease process.
• Clients and families are optimistic for the healing of disease and comply with all
recommended clients are given.
Gastritis is an inflammation of the lining of the stomach, and has many possible
causes. The main acute causes are excessive alcohol consumption or prolonged use
of nonsteroidal anti-inflammatory drugs (also known as NSAIDs) such as aspirin
or ibuprofen. Sometimes gastritis develops after major surgery, traumatic injury,
burns, or severe infections. Gastritis may also occur in those who have had weight
loss surgery resulting in the banding or reconstruction of the digestive tract.
Chronic causes are infection with bacteria, primarily Helicobacter pylori, chronic
bile reflux, stress and certain autoimmune disorders can cause gastritis as well. The
most common symptom is abdominal upset or pain. Other symptoms are
indigestion, abdominal bloating, nausea, and vomiting and pernicious anemia.
Some may have a feeling of fullness or burning in the upper abdomen. A
gastroscopy, blood test, complete blood count test, or a stool test may be used
to diagnose gastritis. Treatment includes taking antacids or other medicines, such
as proton pump inhibitors or antibiotics, and avoiding hot or spicy foods. For those
with pernicious anemia, B12 injections are given. wikipedia
1. Risk for Imbalanced Fluid Volume and Electrolytes : less than body
requirements related to inadequate intake, vomiting
1. Risk for Imbalanced Fluid Volume and Electrolytes : less than body
requirements related to inadequate intake, vomiting
Goal:
Disorders of fluid balance did not occur.
Expected results:
Moist mucous membranes, good skin turgor, electrolytes returned to normal,
capillary filling pink, vital signs stable, the balance of input and output.
Nursing Intervention :
Goal:
Nutritional deficiencies resolved.
Expected results:
Normal albumin value, no nausea and vomiting, weight within normal limits,
normal bowel sounds.
Nursing Intervention :
Assess food intake, body weight measured regularly, give oral care on a regular
basis, encourage clients to eat little but often, give food in warm, auscultation
bowel sounds, assess food preferences, check the laboratory, for example:
Hemoglobin, hematocrit, albumin.
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Peptic Ulcer
is a lesion in the mucosa of the lower esophagus, stomach, pylorus, or duodenum.
also known as ulcus pepticum, PUD or peptic ulcer disease, is an ulcer (defined as
mucosal erosions equal to or greater than 0.5 cm) of an area of the gastrointestinal tract
that is usually acidic and thus extremely painful
Causative factors include mucosal infection by the bacterium Helicobacter pylori
(mechanism unclear).
Use of non-steroidal anti-inflammatory drugs (NSAIDs), especially aspirin.
Genetic factors such as cigarette smoking, stress, and lower socio-economic status may
play a role.
Complications include GI hemorrhage, perforation, and gastric outlet obstruction.
Classification
Assessment
1. Abdominal pain
o Occurs in the epigastric area radiating to the back; described as dull, aching, and
gnawing.
o Pain may increase when the stomach is empty, at night, or approximately 1 to 3
hours after eating. Pain is relieved by taking antacids (common with duodenal
ulcers).
2. Nausea, anorexia, early satiety (common with gastric ulcers), belching.
3. Dizziness, syncope, hematemesis, melena with GI hemorrhage:
o Positive fecal occult blood
o Decreased hemoglobin and hematocrit, indicating anemia.
o Orthostatic blood pressure and pulse changes.
4. Peptic ulcer disease may be asymptomatic in up to 50% of persons affected
5. Differentiating Gastric and Duodenal Ulcers:
Gastric Ulcer Duodenal Ulcer
Gnawing epigastric pain Gnawing epigastric pain
occurring 30 minutes to 1 occurring 2-3 hours after
hour after meals meals
Aggravated by eating Relieved by food (because the
(because acid secretion pyloric sphincter, at the
increase at meal time) leads junction of stomach and
to weight loss duodenum, closes upon eating
to concentrate food in the
stomach) causes weight gain
Relieved by vomiting Not relived
(because acid is expelled out)
No pain at hours of sleep Pain at hours of sleep
(HCl production decreases at (because gastric emptying
hours of sleep) continuous at hours of sleep)
More common in persons More common between ages
older than age 50 25 and 50
Diagnostic Evaluation
Pharmacologic Interventions
1. Histamine2 (H2) receptor antagonists such as ranitidine to reduce gastric acid secretions.
2. Antisecretory or proton-pump inhibitor, such as omeprazole, to help ulcer heal quickly in
4 to 8 hours.
3. Cytoprotective drug sucralfate, which protects ulcer surface against acid, bile, and pepsin.
4. Antacids to reduce acid concentration and help reduce symptoms.
5. Anti-biotic as part of a multi-drug regimen to eliminate H. pylori to prevent reoccurrence.
Surgical Interventions
1. Gastroduodenostomy (Billroth I)
o Partial gastrectomy with removal of antrum and pylorus; gastric stump is
anastomosed to duodenum.
2. Gastrojejunostomy (Billroth II)
o Partial gastrectomy with removal of antrum and pylorus; gastric stump is
anastomosed to jejunum.
3. Antrectomy
o Antrum (lower half of stomach), pylorus and small cuff of duodenum are
resected; stomach is anastomosed to jejunum and duodenal stump is closed.
4. Total gastrectomy
o Removal of stomach with anastomosis of esophagus to jejunum or duodenum.
5. Pyloroplasty
o Longitudinal incision is made in the pylorus, and closed transversely to permit the
muscle to relax and established an enlarged outlet; often performed with
vagotomy.
Nursing Interventions
1. Monitor the patient for signs of bleeding through fecal occult blood, vomiting, persistent
diarrhea, and change in vital signs.
2. Monitor intake and output.
3. Monitor the patient’s hemoglobin, hematocrit, and electrolyte levels.
4. Administered prescribed I.V. fluids and blood replacements if acute bleeding is present.
5. Maintain nasogastric tube for acute bleeding, perforation, and postoperatively, monitor
tube drainage for amount and color.
6. Perform saline lavage if ordered for acute bleeding.
7. Encourage bed rest to reduce stimulation that may enhance gastric secretion.
8. Provide small, frequent meals to prevent gastric distention if not actively bleeding.
9. Watch for diarrhea caused by antacids and other medications.
10. Restrict foods and fluids that promote diarrhea and encourage good perineal care.
11. Advise patient to avoid extremely hot or cold food and fluids, to chew thoroughly, and to
eat in a leisurely fashion to reduce pain.
12. Administer medications properly and teach patient dose and duration of each medication.
13. Advise patient to modify lifestyle to include health practices that will prevent recurrences
of ulcer pain and bleeding.
nursing interventions:
assess/monitor/evaluate/observe (to evaluate the patient's condition)
o assess frequency, character and amount of any nausea
o assess the duration of nausea
o assess what conditions cause or make the nausea worse
care/perform/provide/assist (performing actual patient care)
o place an emesis basin within patient's reach
o assist with or offer mouth care after each episode of emesis or
q4h
o offer ice chips, ginger ale or warm both if allowed per diet
o if allowed, offer dry (toast, crackers) and bland foods (broth,
rice, bananas, jell-o)
o do not give fried or greasy foods
o give antiemetics as ordered by the doctor
o give antacids as ordered by the doctor
teach/educate/instruct/supervise (educating patient or caregiver)
o teach the patient that his symptoms of distension, belching and
flatulence are a result of the disease process and as medical
treatment is effective they will disappear.
(http://www.webmd.com/a-to-z-guides/flatulence-gas)
o teach the patient to change positions slowly
o teach the patient about the appropriate foods to eat when
nauseated and those to avoid
o teach the patient the importance of maintaining fluid intake
o teach the patient that they need to contact the doctor if vomiting
persists for more than 24 hours
manage/refer/contact/notify (managing the care on behalf of the
patient or caregiver)
o notify the doctor if the patient vomits black or bloody emesis or
develops a fever
this is done for each nursing diagnosis (problem) with attention to the
symptoms of the problem since that is what you are aiming your
nursing treatments at. so, you will be looking for nursing interventions
for the following:
o hematemesis
o melena
o symptoms of dehydration
o symptoms of hemorrhage
o weight loss
o anorexia
o intolerance of fatty foods (you don't want the patient eating fried
or greasy food anyway!)
o mid-epigastric pain (burning sensation) sometimes presence at
night
and this is how you use the nursing process to think critically and
work out the answer to your question.
TREATMENT.
Peptic ulcer disease is characterized by ulcer formation in the esophagus, stomach, or
duodenum, areas of the gastro intestinal mucosa that are exposed to gastric acid and
pepsin. Gastric and duodenal ulcers are more common then esophageal ulcers. Peptic
ulcers are attributed to an imbalance between cell-destructive and cell-protective
effects . Cell-destructive effects include those of gastric acid , pepsin, Helicobacter
pylori infection, and ingestion of nonsteroidal anti-inflammatory drugs . Gastric acid,
a strong acid that can digest the stomach wall, is secreted by parietal cells in the
mucosa of the stomach antrum, near the pylorus. The parietal cells contain receptors
for acetylcholine, gastrin, and histamine, substances that stimulate gastric acid
production. Acetylcholine is released by vagus nerve endings in response to stimuli,
such as thinking SECTION 10 DRUGS AFFECTING THE DIGESTIVE SYSTEM
about or ingesting food. Gastrin is a hormone released by cells in the stomach and
duodenum in response to food ingestion and stretching of the stomach wall. It is
secreted into the bloodstream and eventually circulated to the parietal cells. Histamine
is released from cells in the gastric mucosa and diffuses into nearby parietal cells. An
enzyme system catalyzes the production of gastric acid and acts as a gastric acid pump
to move gastric acid from parietal cells in the mucosal lining of the stomach into the
stomach lumen. Pepsin is a proteolytic enzyme that helps digest protein foods and also
can digest the stomach wall. Pepsin is derived from a precursor called pepsinogen,
which is secreted by chief cells in the gastric mucosa. Pepsinogen is converted to
pepsin only in a highly acidic environment . H. pylori is a gram-negative bacterium
found in the gastric mucosa of most clients with chronic gastritis, about 75% of clients
with gastric ulcers, and more than 90% of clients with duodenal ulcers. It is spread
mainly by the fecal-oral route. However, iatrogenic spread by contaminated
endoscopes, biopsy forceps, and nasogastric tubes has also occurred. Once in the
body, the organism colonizes the mucus-secreting epithelial cells of the stomach
mucosa and is thought to produce gastritis and ulceration by impairing mucosal
function. Eradication of the organism accelerates ulcer healing and significantly
decreases the rate of ulcer recurrence. Cell-protective effects normally prevent
autodigestion of stomach and duodenal tissues and ulcer formation. A gastric or
duodenal ulcer may penetrate only the mucosal surface or it may extend into the
smooth muscle layers. When superficial lesions heal, no defects remain. When smooth
muscle heals, however, scar tissue remains and the mucosa that regenerates to cover
the scarred muscle tissue may be defective. These defects contribute to repeated
episodes of ulceration. Although there is considerable overlap in etiology, clinical
manifestations, and treatment of gastric and duodenal ulcers, there are differences as
well. Gastric ulcers are often associated with stress , NSAID ingestion, or H. pylori
infection of the stomach. They are often manifested by painless bleeding and take
longer to heal than duodenal ulcers. Gastric ulcers associated with stress may occur in
any age group and are usually acute in nature; those associated with H. pylori
infection or NSAID ingestion are more likely to occur in older adults, especially in the
sixth and seventh decades, and to be chronic in nature. Duodenal ulcers are strongly
associ BOX 60–1