Compartment syndrome occurs when pressure increases within a limited anatomical space, reducing blood flow. It can be acute or chronic. Acute causes include fractures and soft tissue trauma. Chronic causes repetitive activities. Compartment syndrome is diagnosed based on risk factors, symptoms of pain, pallor, paresthesia, paralysis and pulselessness, and direct measurement of compartment pressure over 30mmHg. Treatment involves surgical fasciotomy to release pressure through incisions in the fascia. For the leg, double incision fasciotomy through separate anterior and lateral incisions is most effective approach.
Compartment syndrome occurs when pressure increases within a limited anatomical space, reducing blood flow. It can be acute or chronic. Acute causes include fractures and soft tissue trauma. Chronic causes repetitive activities. Compartment syndrome is diagnosed based on risk factors, symptoms of pain, pallor, paresthesia, paralysis and pulselessness, and direct measurement of compartment pressure over 30mmHg. Treatment involves surgical fasciotomy to release pressure through incisions in the fascia. For the leg, double incision fasciotomy through separate anterior and lateral incisions is most effective approach.
Compartment syndrome occurs when pressure increases within a limited anatomical space, reducing blood flow. It can be acute or chronic. Acute causes include fractures and soft tissue trauma. Chronic causes repetitive activities. Compartment syndrome is diagnosed based on risk factors, symptoms of pain, pallor, paresthesia, paralysis and pulselessness, and direct measurement of compartment pressure over 30mmHg. Treatment involves surgical fasciotomy to release pressure through incisions in the fascia. For the leg, double incision fasciotomy through separate anterior and lateral incisions is most effective approach.
Compartment syndrome occurs when pressure increases within a limited anatomical space, reducing blood flow. It can be acute or chronic. Acute causes include fractures and soft tissue trauma. Chronic causes repetitive activities. Compartment syndrome is diagnosed based on risk factors, symptoms of pain, pallor, paresthesia, paralysis and pulselessness, and direct measurement of compartment pressure over 30mmHg. Treatment involves surgical fasciotomy to release pressure through incisions in the fascia. For the leg, double incision fasciotomy through separate anterior and lateral incisions is most effective approach.
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compartment syndrome
PENDAHULUANSindroma compartment is a condition where an increase in interstitial pressure
in a limited space, that is, in a closed compartment osteofasial. The room contains the muscles, nerves and blood vessels. When intrakompartemen pressure increases, blood perfusion to the tissue will decrease and the muscles in the compartment will be ischemic. Common clinical signs are pain, paresthesias, paresis, accompanied pulse missing. (1,2,3) Compartment syndromes can be classified into acute and chronic, depending on the cause of increased compartment pressures and duration of symptoms. A common cause of acute compartment syndrome is a fracture, soft tissue trauma, arterial damage, and burns. While chronic compartment syndrome can be caused by repetitive activities eg run. (1) INSIDENDi American, anterior distal lower extremities is the most widely studied for compartment syndrome. Considered as the second most frequent to the trauma of about 2-12%. From research McQueen (2000), compartment syndrome is diagnosed more often in men than women, but it does have a bias, where men are more often injured trauma. McQueen examine 164 patients diagnosed with compartment syndrome, 69% are related to fracture and most are fractures of the tibia. According Qvarfordt, a group of patients with leg pain, 14% of patients with anterior compartment syndrome. Compartment syndrome is found 1- 9% of fractures in the legs. (4.5) ANATOMIFascia separates muscle fibers in one group. Compartment is an enclosed area bounded by bones, interosseous membrane and fascia involving muscle tissue, nerves and blood vessels. (6) In the region of the brachium, the compartment is divided into two parts: (9.10) 1. Volar compartment: the flexor muscles of the wrist and fingers, ulnar nerve and nerve median.2. Dorsal compartment: the extensor muscles of the wrist and fingers, interosseous nerve posterior.Pada antebrachium region, the compartment is divided into 3 parts: (9.10) 1. Volar compartment: the flexor muscles of the wrist and fingers, ulnar nerve and nerve median.2. Dorsal compartment: the extensor muscles of the wrist and fingers, posterior.3 interosseous nerve. Mobile wad: muscle extensor carpi radialis longus, extensor carpi radialis brevis muscle, muscle brachioradialis.Pada wrist joint region, the compartment is divided into six sections: (9.10) 1. Compartment I: muscle abductor pollicis longus and extensor pollicis muscle brevis.2. Compartment II: extensor carpi radialis brevis muscle, the extensor carpi radialis muscle longus.3. Compartment III: extensor pollicis muscle longus.4. Compartment IV: extensor digitorum communis muscles, the extensor muscles indicis.5. Compartment V: minimi.6 digit extensor muscle. Compartment VI: extensor carpi muscles ulnaris.Pada regio cruris, the compartment is divided into four parts: (9.10) 1. Anterior Compartment: tibialis anterior and extensor muscles toes, profunda.2 peroneal nerve. The lateral compartment: peroneus longus and brevis muscle, peroneal nerve superfisial.3. Superficial posterior compartment: the gastrocnemius and soleus muscles, nerves sural.4. The deep posterior compartment: tibialis posterior and flexor muscles toes, tibial nerve. ETIOLOGIPenyebab compartment syndrome is pressure within the compartment is too high, more than 30 mmHg. The cause of the increased pressure intrakompartemen is an increase volume of fluid in the compartment or decrease the volume of the compartment. (9) The increase in the volume of fluid in the compartment can be caused by: (9) Increased permeability of capillaries, as a result of shock, burns, trauma directly. Increased capillary pressure, as a result of exercise or the presence of venous obstruction. Muscle hypertrophy. Bleeding. Infusion infiltration. Decreased volume compartment can be caused by: (9) bandage too tight.
PATOGENESISPerkembangan compartment syndrome depends not only on the pressure
intrakompartemen but also systemic blood pressure. The pathophysiology of compartment syndrome involving local hemostasis normal tissue which leads to increased tissue pressure, decreased capillary blood flow and local tissue necrosis due to hypoxia. (1) When the pressure within the compartment exceeds the blood pressure in the capillaries and cause capillary collapse, nutrients can not flow out to the cells and the metabolism can not be excluded. In just a few hours, the cells are not getting the food will be damaged. First of all the cells will experience swelling, then the cell will stop releasing chemicals that cause further swelling. Swelling growing causes increased pressure. (12, 13) which passes through the capillary blood flow will stop. Under these circumstances oxygen delivery will also be stopped. Hypoxia causes the cells to release of vasoactive substances (eg, histamine, serotonin), which increases endothelial permeability. Capillaries in the event of loss of fluid resulting in increased tissue pressure and aggravate damage to surrounding tissue and muscle tissue necrosis. (5) DIAGNOSISSindroma compartment can be diagnosed based on knowledge of risk factors, subjective complaints and the presence of physical signs and clinical symptoms. The risk factors in compartment syndrome include severe fractures and trauma to the soft tissue, the use of bandages. (15,16) Clinical symptoms are commonly seen in the compartment syndrome includes 5 P, namely: (17) 1. Pain (pain): pain in the fingers or toes during passive stretching of the muscles are affected, when there is trauma langsung.2. Pallor (pale): skin feels cold when on palpation, skin color is usually pale, gray or keputihan.3. Paresthesias: usually asymptomatic heartburn and itching in the area lesi.4. Paralysis: usually begins with the inability to move the joints, is a sign of a slow diketahui.5. Pulselesness (decrease or loss of pulse): due to a disturbance arterial.Pengukuran perfusion pressure is one of the additional compartments in helping to establish the diagnosis. Compartment pressure measurements are usually performed in patients with decreased awareness of physical examination did not give satisfactory results. Compartment pressure measurements can be done using injection techniques or wick catheter. (15,16) compartment pressure measurement procedures, among others: (19) a. Mechanical injeksi.Jarum size 18 is connected with 20 cc syringe through the copy and the air duct. These channels are then connected to a standard mercury manometer. After the needle is injected into the compartment, the air pressure in the syringe to be increased so that the meniscus copy-air seemed to be moving. Then the pressure in the compartment can be read on the manometer raksa.b. Kateter.Wick Wick engineering plastic catheter and sheath are connected to the transducer and recorder. Catheter and the jar is filled by a three-way connected with the transducer. It is necessary to ensure that no air bubbles in the system because it gives a low yield or obscure measurements. Tip of the catheter should be able to stop a water meniscus that can be ascertained and it is known that in such networks bypassed a large trocar, then the needle is withdrawn and the catheter is wrapped into kulit.TERAPIPenanganan compartment syndrome include: 1. Medical Therapy / non surgical. (11) • Placing foot level of the heart, to maintain the height of the compartment are minimal, elevation avoided because it can reduce blood flow and will more aggravate ischemia. • In case of a decrease in the volume of the compartment, the cast must be opened and the pads kontriksi removed. • Correcting hypoperfusion by way of crystalloid and blood products. • Provision of mannitol, vasodilator drugs known as inhibitors or sympathetic. 2. Surgical treatment / operatif.Fasciotomi is the operative treatment of compartment syndrome with fracture stabilization and repair of blood vessels. The success of the decompression for improvement of perfusion is 6 hours. (11) Therapy for acute and chronic compartment syndrome is usually surgery. Long incision is made on the fascia to relieve increased pressure inside. The wound is left open (covered with a sterile dressing) and closed at a second operation, usually 5 days later. if there is a muscle necrosis, do debridement, otherwise healthy tissue, the wound can be in sewing (without strain), or a skin graft may be needed to close this wound. (8,20) The indications for Fasciotomy are: (21) 1. There are clinical signs of the syndrome kompartemen.2. Intrakompartemen pressure exceeded 30 mmHg. Fasciotomy THE REGIO CRURISAda 3 Fasciotomy approach to compartment regio cruris: fibulektomy, single incision Fasciotomy perifibular, and Fasciotomy double incision. Fibulektomi is a radical procedure and is rarely done, and if there is, including the indication of acute compartment syndrome. Single incision may be used for soft tissue of the extremities. Double incision technique is more safely and effectively. (1.19) Fasciotomy single incision (davey, Rorabeck, and Fowler): Made incision lateral, longitudinal line of the fibula, all from the distal head of the fibula to 3-4 cm proximal to the lateral malleolus. The skin was opened in the anterior and not to injure the superficial peroneal nerve. Created fasciotomy of the anterior longitudinal and lateral compartments. Next the skin is opened to the posterior and do Fasciotomy superficial posterior compartment. The boundary between the superficial and lateral compartments and the interval is extended upwards by cutting the soleus of the fibula. Peroneal muscle and blood vessels pulled back. Later identified the posterior tibialis muscle fascia to the fibula and do inisisi longitudinally. (1.19) Fasciotomy double incision (Mubarak and Hargens): 20-25 cm long incision is made in the anterior compartment, halfway between the fibula and tibia caput. Subcutaneous dissection is used to expose the fascia compartment. Transverse incision is made in the lateral septum and identification intermuskular superficial peroneal nerve in the posterior septum. Go towards the anterior compartment proximally and distally on the anterior tibial line. Then do Fasciotomy the lateral compartment toward proximal and distal tubules line created longiotudinal second fibula.Insisi 1 cm behind the line of the posterior tibia. Extensive subcutaneous dissection is used to identify the fascia. Vein and nerve saphenus pulled anteriorly. Transverse incision is made to identify the septum between the deep and superficial posterior compartment. Gastrocsoleus fascia is then opened along the compartment. Another incision is made in the flexor digitorum longus and freed entire deep posterior compartment. After the posterior compartment is opened, the identification of the posterior tibialis muscle compartment. If an increase in pressure in this compartment, immediately opened. (1, 19) Fasciotomy THE REGIO ANTEBRACHIUMPendekatan volar (Henry) Decompression volar compartment deep and superficial flexors can be done with a single incision. Skin incision starting from the proximal to the fossa antecubiti to the palmar carpal tunnel area. Compartment pressure can be measured during the operation to confirm decompression. No use of tourniquets. Skin incision from the medial to the biceps tendon, adjacent to the elbow and then to the radial side of the hand and extended towards the distal sepenjang brachioradialis, continued to palmar. Then the superficial flexor compartment is incised, starting at point 1 or 2 cm above the elbow downwards until the wrist. (1.19) Then the radial nerve is identified under brachioradialis, they then pulled to the radial direction, then the flexor carpi radialis and the radial artery is pulled to the side that will expose the ulnar flexor digitorum profundus flexor pollicis longus, pronatus Quadratus, and pronatus teres. Because of compartment syndrome usually involves the deep flexor compartment, must decompression fascia around the muscle to ensure that adequate decompression has done. (1.19) UlnarPendekatan Volar volar ulnar approach done in the same way to approach Henry. Disupinasikan arm and medial incision starting from the top of the biceps tendon, past the elbow fold, continuing down through the line of the ulnar forearm, and up to carpal tunnel along the thenar fold. Superficial fascia of the flexor carpi ulnar is incised up to the elbow aponeurosis and to carpal tunnel distally. Then look for the boundary between the flexor carpi ulnaris and flexor digitorum sublimis. On the basis of the flexor digitorum contained sublimis ulnar artery and nerve, to look for and protected. The fascia of the deep flexor compartment then incised. (1.19) compartment DorsalSetelah approach superficial and deep flexor forearm decompressed, should decide whether to Fasciotomy dorsal (extensors). It is better determined by measurement of compartment pressure Fasciotomy intraoperatively after flexor compartment. If there peningktan pressure on the dorsal compartment continues to increase, Fasciotomy should be done with the forearm pronated position. Straight incision from the lateral epicondyle to the midline of the wrist. The boundary between the extensor carpi radialis brevis and extensor digitorum communist identified then do Fasciotomy. (1.19) BANDINGDiferensial DIAGNOSIS diagnosis of compartment syndrome include tendinitis, fatigue fractures and shin splints. This situation is connected by pain in the lower limbs due to exercise. But give the same symptoms as compartment syndrome. (22,23) on tendinitis Symptoms usually appear after exercise, pain is often caused by strain on the tendon. In the fatigue fracture, the bone area which extends from one side attacked bone to another bone. At shin splints, pain is usually only at the height of the medial tibia behind, often at a meeting in half and the distal third of the tibia. (22,23) Complications (21.24) • Failure to reduce intrakompartemen pressure can cause tissue necrosis, during perfusion capillary is still lacking and causing hypoxia on the network. • contracture Volkmann is a deformity of the limbs which is a continuation of compartment syndrome of acute who did not receive treatment for more than a few weeks or months. • Infections. • hypesthesia and pain. • Complications which may arise from the systemic compartment syndrome include acute renal failure, sepsis, and acute respiratory distress syndrome (ARDS) fatal case of sepsis multisistem.PROGNOSISSindroma organ failure in acute compartment tend to have an end result that is ugly. Muscle tolerance to ischemia is 4 hours. Irreversible damage occurs when more than 8 hours. If the diagnosis is delayed, can cause nerve trauma and loss of muscle function. Although Fasciotomy done quickly and early, nearly 20% of patients experience sensory and motor deficits were persistent. (11) LIST PUSTAKA1. Frederick Azar. Compartment syndrome in Campbell`s operative orthopedics. 10th Ed. Vol 3. Mosby. USA. 2003. p: 2449-572. DeLee C Jesse, David Drez. Compartment syndrome in DeLee & Drez`s orthopedic sports medicine. 2nd Ed. Vol 1. Saunders. USA. 2003. p: 13-43. Louis C. Argenta. Compartment syndromes in Basic sciense for surgeons. Saunders. Philadelphia. 2004. p: 143-44. Paula Richard. Compartment syndrome, extremity. Available at http://www.emedicine.com. Accessed on May 28th 2007.5. Apostle Abraham. Compartment syndrome. Available at http://www.emedicine.com. Accessed on May 29th 2007.6. Peter Cameron, George Jelinek. Textbook of compartment syndrome in adult emergency medicine. 2nd Ed. Churchill Livingstone. New York. 2004. p: 84-57. Anonymous. Compartment syndrome. Available at http://www.AAOS.com. Accessed on May 28th 2007.8. Andrew L, Chen. Compartment syndrome. Available at http://www.medlineplus.com. Accessed on May 28th 2007.9. Marc F Swiontkowski. Compartmental syndromes in the Manual of orthopedics. 5th Ed. Lippincott Williams & Wilkins. USA. 2001. p: 20-810. Preston R Miller, John M Kane. Compartment syndrome and rhabdomyolysis in The trauma manual. 2nd Ed. Lippincott Williams & Wilkins. USA. 2002. p: 335-711. Stephen Wallace. Compartment syndrome, lower extremity. Available at http://www.emedicine.com. Accessed on June 4th 2007.12. Anglen J, Banovetz. Pathophysiology of compartment syndrome in the leg well the resulting from the fracture table positioning. Clinical Orthopedics and Related Research. 1994. p: 239-4213. Kearns, Daly, Sheehan, Murray. Oral Vitamin C Reduces the injury to skeletal muscle the caused by compartment syndrome. Journal of Bone and Joint Surgery. Aug 2004.14. Louis Solomon, David Warwick. Compartment syndrome in Appley`s system of orthopedics and fractures. 8th Ed. Oxford University Press. New York. 2001. p: 563-415. Townsend M Courtney, Beau Champ. Acute compartment syndrome in Textbook of Surgery. 17th Ed. Elsevier Saunders. USA. 2004. p: 554-716. Pink P Mitchell, Edward Abraham. Textbook of compartment syndrome in critical care. 5th Ed. Elsevier Saunders. USA. 2005. p: 209917. Ronald McRae, Max Esser. Compartment syndromes in Practical fracture treatment. Churchill Livingstone. New York. 2002. p: 9918. Fred Flandry. Compartment syndrome: swelling out of control. Available at http://www.hughston.com. Accessed on May 28th 2007.19. Amendola, Bruce Twaddle. Compartment syndromes Skeletal trauma in basic science, management, and reconstruction. Vol 1. 3rd Ed. Saunders. 2003. p: 268-9220. Brian J Awbrey, Shingo Tanabe. Chronic exercise- induced compartment syndrome of the leg. Harvard Orthopedic Journal.21. Kalb L Robert. Compartment syndrome Procedures for evaluation in primary care. Mosby. USA. 2003. p: 1419- 2922. Frederick A. compartmental syndromes. Available at http://www.wikipedia.org. Accessed on June 4th 2007.23. Richard Braver. Surgical pearls: How to test and treat exertional compartment syndrome. American College of Foot and Ankle Surgeons. May 2002. p: 22-424. Anonymous. Compartment syndrome. Available at http://www.wikipedia.org. Accessed on May 29th, 2007. Brian J Awbrey, Shingo Tanabe. Chronic exercise-induced compartment syndrome of the leg. Harvard Orthopedic Journal.21. Kalb L Robert. Compartment syndrome Procedures for evaluation in primary care. Mosby. USA. 2003. p: 1419-2922. Frederick A. compartmental syndromes. Available at http://www.wikipedia.org. Accessed on June 4th 2007.23. Richard Braver. Surgical pearls: How to test and treat exertional compartment syndrome. American College of Foot and Ankle Surgeons. May 2002. p: 22-424. Anonymous. Compartment syndrome. Available at http://www.wikipedia.org. Accessed on May 29th, 2007. Brian J Awbrey, Shingo Tanabe. Chronic exercise-induced compartment syndrome of the leg. Harvard Orthopedic Journal.21. Kalb L Robert. Compartment syndrome Procedures for evaluation in primary care. Mosby. USA. 2003. p: 1419-2922. Frederick A. compartmental syndromes. Available at http://www.wikipedia.org. Accessed on June 4th 2007.23. Richard Braver. Surgical pearls: How to test and treat exertional compartment syndrome. American College of Foot and Ankle Surgeons. May 2002. p: 22-424. Anonymous. Compartment syndrome. Available at http://www.wikipedia.org. Accessed on May 29th, 2007. Available at http://www.wikipedia.org. Accessed on June 4th 2007.23. Richard Braver. Surgical pearls: How to test and treat exertional compartment syndrome. American College of Foot and Ankle Surgeons. May 2002. p: 22-424. Anonymous. Compartment syndrome. Available at http://www.wikipedia.org. Accessed on May 29th, 2007. Available at http://www.wikipedia.org. Accessed on June 4th 2007.23. Richard Braver. Surgical pearls: How to test and treat exertional compartment syndrome. American College of Foot and Ankle Surgeons. May 2002. p: 22-424. Anonymous. Compartment syndrome. Available at http://www.wikipedia.org. Accessed on May 29th, 2007.