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Hypertoniker - Compliance-Gespräch
VIGGO, Magensonde Halsschmerz
t page until you have thought this through.
nd you probably figured DM2 Diagnose erklaeren
out that they radikulaerer Ruckenschmerz
roxysmal atrial tachycardia,EKG: Hinterwandinfarkt, Vorhofflimmern,
and atrial Anorexia nervosa - Notwendigkeit der
hen you get your EKG onVorderwandinfarkt, Hemiblock
this patient you Aufnahme
see one normal P wave forBenzo Gespraech
every QRS Hörtest: Aortenstenose
cardia. With atrial flutter Leber Ikterus
and regular block, obere GI Blutung Management
with AVNRT or the less LuFu + BGA
common PAT, the KU: Lunge, Pulse, Leber
erage sinus tachycardia,
and you will see
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es. Nachrichten maligne, infektiös
Hypothyreose somatoforme Störung
Hyperthyreose Herz: Prävention, Risikofaktoren, Therapie
Niereninsuffizienz: 10 Laborwerte
Supraventricular Arrhythmias
- look for P waves
- prominent in leads II and V1
cs EKG
AV nodal reentrant tachykardia regular
P waves are retrograde if visible
ble RR = 150 - 250 bpm
carotid massage: slows or terminates
rminates
Atrial Flutter regular, saw-toothes
2:1, 3:1, 4:1, block
one-
e- RR = 250 - 350 bpm
ne- ventricular rate: 1/2, 1/3, 1/4 of atrial rate
carotid massage: increases risk of block
Atrial Fibrilation irregular (absolute arrhythmia)
undulating baseline
RR = 350 - 500 bpm
ventricular rate: variable
carotid massage: may slow ventricular rate
ar
raterate
ar rate Multifocal atrial tachykardia irregular
at least 3 different P-wave morphologies
RR = 100-200 bpm, sometimes < 100 bpm
carotid massage: no effect
bpm
pm
bpm
Paroxismal atrial tachykardia regular
RR = 100 - 200 bpm
charachteristic warm-up period in the automatic
form
carotid massage: no effect, or only mild slowing
mild
dild
supraventricular
praventricular
upraventricular
s..They
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aventricular
Ventricular Arrhythmias
Premature ventricular contractions QRS is wide and bizzare (ventricular depolarisation
does not follow the normal ventricular conduction
pathways)
bigeminy, trigeminy if the beats alternates with
normal sinus beats
can trigger a ventricular tachykardia, if a PCV falls on
the T wave oft he second sinus beat
Ventricular tachykardia RR = 120 - 200 bpm
often after an AMI
Ventricular fibrilation jerks of about spasmodically (coarse ventricular
fibrillation) or undulates gently (fine vent. fibrillation)
heart generates no cardiac output
Rules of Malignancy for PVCs
Accelerated idioventricular rhythm
benign rhythm
RR = 50 - 100 bpm
ventricular escape focus that has accelerated
Frequent PVCs sufficiently to drive the heart
rarely sustained, does not progress to Vfib
Consecutive PVCs
ignancyMultiform
for PVCs PVCs
Torsades de pointes ventricular tachykardia that is usually seen in patients
with prolonged QT intervals
tQT normaly 40 % oft he cardiac cycle
VCs R-on-T phenomenon QRS complexes spiral around the baseline, changing
les of Malignancy
PVCs for PVCs
Any PVC occurring
theisr axis and amplitude
during an acute myocardial infarction (or in any patient
B blockers, TCA, phenothiazines