Physio RR Notes

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TRIMMED Raked Raisin

Physiology
Cardona system patent
Action potential of S A node

Note
RM P SA 60mi
Pacemaker of Heart
H

alt Highest fog of Ap


Generation
q
time toy Yest
5 711 Ii
Restless membrane Étpecahand
Potential

puke Depolarization Cat Influx


Time 3
Repolarization jÉÉfflise

Ade If Hca Locke Ineboiadin


te
Action fetid of V.my

It
jot

QE
o
phase 4 RMP 90mV

phase
o
Dp Nat Inf
phase 2
plate cats lief
please Z RP
Kt
Eff
Depolarization Endo Epi
Repolarization
Epi Ende
A V Node lat speed of

of
Etten

GapJunction's

Gate Keeper of
Heart
1 I f
Mk site of
Conduction Block
Purkinje fastest speed of
Is conduction

Highest no of Gap
function's

I
n n Tt
t
n nm m

o o o o o

t R

LEI ER

Eres
Pr Interval A p Travels from S A Node
To through A
Node
120 200
F
milk
san Ava
3 5 Small q
Squares

PR Interval Always Inversly

Proportional
to Heart Rate

1st Degree the Rheumatic Heartfeet


Hemochromatosis
Sarcoidosis

PR 70.2 Sec prolonged

Rx Atropine

2ndDegree Ha

Mobitz Cakebachphenomenon
Type I
24 I

Progressive Serial prolongation of PR


69th Sudden drop
Infra nodal Problems A V problem

Moltz
types Emmi IIjÉ

PI Lot Prolonget
Block
Bundeft
So III bloat
Most D theantblock
Severe bowdycandia HR 20hpm
Cardiac
Can cause Sudden Death

Mk boooodycoothemipost IT
MI b
dthm.pe MI
M1 Iot 3
of
a

V.fi
Rx Temporarypocemaker

Éth completetleartblog
Complete Ivigsociation
on ordination
Lack of co

Blue cotta ventricles

p p s
1
Enfeoff
No of P No of qrs

moved Complexes

Rx
jPoetpacemaker
dote A V Dissociation

I
37113g ventiated
Taha
A dead
Cardiac
wrath

Duration 0.81
phase

Rhythm

Ied doing
Atrialsystole

Katyn
A 70 0
t t

Due To Atrial passive

EÉÉ fitting
mete contraction
É
No outflow of flood

e
alt closure of
EDU 120140mL
Preload

al lol
V
reforested

17

fÉ9
g

_HIIE8mmtlg
12omatly
REP simmtly
231 of LV

SEP 12ommtly 8ommtly

Id of Sr

Events Aortic valve Efection


gymmtly
open's click

Rp'D Efate phase


Rapid Ejection phase
Slow Election phase

somntly AÉ 22
close
Hetty
É

ftp.t.x.t

I
LA Stuart's to Receive blood from

PIE's DIgIvr
Starts with closure of
E
Aortic of pulmonic Valves
Arpa

gfig1
_isÉ

Rapid vent Fgf


metallic
Tricuspidate
I

county
e

cqisÉÉÉÉ

Tt És
VENTRICULAR

GALLOP RHYTHM
É fATIE

j

Cardiac motility

Heart on Echo

9 IT that soonof ED
side of
D 2 Bell
2
Sy stets
2 Employee
Seen in
Eal myxoma
At
Left atria

Heard after opening


Snap
s
0

s
I
Passystolic murmur
three mama
THEHITI
A Mitral Regurgitation

2 Tricuspid Regurgitation

Ventianlarfle
2

S
S

1
thing
chook min
many
murmu s.FI

tEgptetaudy Aorteriosu

AORTIC
TETRIS
4

Et
l
o
I 10
IP Eg Inspiration
It

Decreases

If are
Kumuggd

Seen in Constrictive

Pettis
Be Regulation

Bo receptor
Refine
Drano ReceptorEgging
I

2
noticed
Afferent's ath 10th

3 Center
medullacts

Herring 4th
Ig CYANS
y
Mr 10th

sensitive Io
Breceptor a

Pressuredman
Mean
that
DI typulsepressure
80 t
Bp S BP DBP
MAP 93
I 120 80

NTS
Y
3
SMS psn

III
Eat
MARES WAI

II Be RIDE in
HeartRate

Damping of carotid Sinus


I
DEE.EE
Ii

9th ath

TBI
lot

HEARTRATE DECREASEL

2
DEN
I
If

KEEP
IBP
7
4ft

HEART RATE INCREASED


11 11 I R IL

A CARID

EL MASSAGE

HEART RATE

DECREAS
AWS
D
Eincple's
mum

DRANK TAILING LAI


1401
Come
for L SI
Ready
2 EICK PRINCILEI
Estimation of cardiac output by
the Rate of Oz caption
AI
cog
VOI V Blood
200ft
A Vo Diff Isomgg

É
Estoy

Er Refleney

Reflex
Duhig bag
g dy
Reftest
seen in Ied trauma
I Vt
If
2 FARISH REFLEX
GOLD
cardiac Reflex
Brady
Serafin Irn

Wage e fiber's
3 GAIN EDGE REFL
_t

Tachycardia Reflex

A Blood TO
HRT
IRI
R Atia
L pressure Vol
sensing
Buarooreceptor's

tail Reflex

VASCULAR PHYSIOLOGY
hmmm
ummm

AIA WIND KISSEL EFFEI


Elastic Recoil
Feet I

AORTA MAX VELOCITY T

CAPILLARIES MIN LELOCII

ARTERIOLE LAY It 14 1
RESISTANCE DI

LAX IIe

H
POISELILLE AGEN FORMULA

Lgtiffscoaty
El CutgcopI

E dos hes whathappens


To
flow

BE 12214 2 2 2 2
Imes Increased
i
RESPIRATORY
mmpysooGIumm

WEIBLE AIRWAY GENERATIONI 22

Trachea Ge
ZEOLI 623

N0Guaseo_
I
Exchange
Dead Space
Loml
Edith t.Bmgnch.de
MAX Smooth
muscle
gas
hang
e
Ppsudostratified
c gZone
cilliated columnar

Ik go.it ant
CLARA Cells

Respiratory Zone Simple Squamous

Stem cells
2pneumoytes
Type

Disorder's
Emmy
D KARTAGENER SYNDROME

Defect Dyneinproteen

Toad Bronchiectasis

IS
Situs Inverters
Sinusitis
Desetooocardia

2 CYSTIC FIBROSIS

AI
CFTR GENE

DEFECTIVE CT channels 7
Thick sputum

Cilia Cannot clean sputum

We's Saltybabies flop Ba


b
Y
Rx Iacafto oh

SURFACTANT
HI CAI
AM S

aced from type 2 pneumocytes

stored in Lamellar end


made up of Lipoproteins
a
Lecithin Sphingomyelin imit
y my
Coppa
Function Dc tensions
Gage
collapsing force

Ed hung Lecithin Sphinomyelin


g teeth Spann
sent
721
Indicates fetal
Lung maturity
Best test test for
fetal Lung maturity phosphatidy ee

Swift production
stimulated by Inhibited by
I

D Cath 1
Insulin
2
theyroxin
3
prolactin
Reform Is ISurfactant
Respiratory Distress
Syndrome
Preterm Labor't

Degpethasone
Indi
Ey Lyx 4dg

Expiating
vs
Expiration m

hive Roy Eve process

Diaphragm Ireful Exam

Cz 4 s Internal IC

phrenic Transverse ABD


Fe
Rectus ABD
Tmp
top

MELANIE QE BREAtHINGm

it

Note
Ipp Is Always Negative

in law
Ah III
Pressure
O 2 5

2
5mmtly

During Inspection
g

ht

E omntly

Ipp 2 5 G mmHg

IAP O 1

Dungy Expiration

It

Ipp Gm 2
mtg Smutty
g g
IAP O 1

compliance vs Elastance
T t

Elastic Recoil
Stretchability

value Kml tomy

Emphysema
Linares
pfibrosy
Dad
formula
compliance
ftp
Pressure Volume Loop
a Iolume hoof
t
iidst
STI
11
7

I's

A on

Surface Tension

Surfactant concentration

Ite Lungs are more Compliant

thane
During Expiation
Inspiration
I

compliance of he filled
being

Increases

Dears
I 1

ALVEOLI
i

I
got

Amity
LUNG VOLUMES

CAPACITIEL
I

Volumes capacities that

cannot be measured spironty


Cannot I T

D Residual Volume

functional Residual
capacity
Total
3
Lung capacity
Methods used to measure

FRC

D Body plethysmography

4 Nitrogen cooshoot
2 Helium dilution method
ventilation
Ako
1 Eml

Conducting zone
Respiratory
150 ml Lone
X 350 ml
RR
250 12 hoooml
alk's min

Alveolar
ventilation
or

Perfusion To Alveoli

5lb's min

Yg
So
Upenatftig Rat 45 0.8

ÉÉE
ÉÉ

EU Boose
TM
III
Yg Apex
3ft
M TB Affects upper lobes

dlt more 02 availability


Ya Boose
I
Note t As we move
from Apex

To Lose Both ventilation of

Perfusion ProgressivelyIncreast

V MISMATCH
g
t
good ie 4
D
g e

Is affected
T
p
Lenin P.Embolism
Affected o
I
p
e

9 good T
Seen in foreign body

obstruction

Atelectasis

Airway
compression

DEAD SPACES
1 ANATOMICAL DE 150 ml
I 19 tome
2 Alveolar D S Always pathological

Absent in Healthy Ind

Ios tale
2
physiological D s

Ind t
d

So In Healthy And Anatomical as


nip

physiological D

Anatomical Dead Space measured by

1
Single breath Na method
g
Aka Fowlertimethod

physiological D s
measured by
Equation

Vo
viliagjjd

Diffusion
ummmm
of ur
Gooses
mum

measured DLCO
by
c ppl t
Co to
210 Times more
affinity
Hb

Normal 25mL min


mmHg
DIO

Eels btwn

2K
conc Gradient Surface
Area

th
Fj omnibus

SF decreased Emphysema

Thickness
Lol fibrosis
Increased p I
atgm

Lemond Decreasedin

polycythemia Ahmad
Em Emphysema

Pol fibrosis

T
Emmet of Gases in mm

fr

C
Co2

Hb 02 HI
mayor myo
Ie 70

Hb coz
2k
III
mimino

I
Responsible for
partialpressure
of 02

Haldane's of
Loading
Iff hooding p
02 onto Hb
To
Leading

Releasing of Co2

Into Lungs

Bhor's
Release of o

Effect
from Hb to

Tissues
É

Ethan

Pqo2 4ommty

Association dissociation
Hb
02 curve
Normal
1050 27 mmHg

Off

FEE

state
how Affinity
Right shift
Seen is Muscles
Exercising
D Hypercarbia
2
Hypoxia
I
Hypof
3
Abs kept II

5 2,3 D PG

Note p
50 value 7Increased

left shift thigh Affinity


State
D Temperature t

Ht
2 Alkalosis I

3 I CO2
4 Fetal Hb

He 150 deoused

piste

É
Pneumotaxic
PONG s
center
12

Apneustic
center
center

medulla 2 Center's

DRG

URG

Pore botzinger Complex


I

Pacemaker
of Respiration
Initiation
Ap
DRE Produce Ramp
DRI Produce RAMP
Signals
I

Slow Rise in TV
maintainance

Pneumotaxic

10 Termination
v
of
Apneustic Inspiration
center
t
DRG RAMP
I

CHEMORECEPTORS

a
cermet peripheral
Borin Carotid Body
sensitive too Aortic Body
CO2
too
Sensitive
HTIPH too
Hypoxia
It 1PM
CENTRAL NERVOUS
mm
SYSTEM
mm
ummm

31

t
t.IE

Ap fiber's are ineuron's

Pain

Acute sharp chronic Dull


shap chronic Dull

fast stalvisceral
t T
d
Affibes fibgt
at Etalon

Bradykinin

Ia ang pain
sensation Ends
2n.ggge
tinos Zc.O
ftp.t

Fifth

V R
F

Dorsal
t
Piggly

Purely
vent

Horn's motor

Be magenta
Root's are Pure butnerves
are mixed

O s s e

t 311
I Fw
3
311,2

É
y
Y It a
31

l s
Boon Ed Syndrome

try

Ipsilateral Dorsalcolumn
Loss
Tensations
contralateral
Antedated
Column Sensation's

Ipsilatal motor Loss


Syringomyelia

iETE

II
i

Loss of pain Temp


Dorsalcolumnfensation

Associated with

And Eri malformation

Typed
Q
O
O

Sensory Homunculus

mapping own areas dedicated

E Sensory Processing

My Rantation FACE II
111 ID
NDS thumb

LEAST Representation Trunk a


HIPI
Law
ar of Projection
u

mugs

ETH

Ej

O
E ol s phantom Limb
Eples Limb
phenomenon

Y s
1 I
A

Lower Motor
Neuron's

Iota f motor
motor
In's Neuron's
t t

Skeletal Skeletal
Muscles Muscled
t t

Intrated
fiber's
Iff
fiber's

Muscle
Indies

ye ye

I
UPPER LOWER MOTOR NEURON
LESIONS

r E
E

STRETCH

REFLEX
Aka Myotatic Reflex
1
x
211
Moto synaptic Reflex
MUSCLE MUSCLE
METERS
SIDE
LENGI
CHANGES A

E I
IFF

Ia Tty
y

I
Inverse Stretch Reflex

7 s
y

I x

me
I

EEGme

If I E Eager waves

frequency 2 Brain
activity
MT TN
7612
I I 3013
1 IIHz
I 71
I LIK

d waves
Eye closed
EH Relaxed state
tittttaine

f Gave Awake Attention

Egg Desynoronization
Cave

for tal con


I
frontal cortex
V leaves
Efeme focus
7GHz

O Caves Gave

4 F
Memory
Hy
Hippocampus

I 7
Lone caves

1 3 Hz
NREM
stiff
0
EYE
IEEE Ttm

NREMwn

Esta Ito
M
Sta g 1211
O
3

IS
stagey
REM
mm SLEEP
mm

13 Hz f waves

Paradoxical Sleep
PG yikes are Seen
Penile Erection
Muscles Relaxed

PARASOMNIASI
MREMI Rim
DAIEI RRÉ D NIGHI
MARI
SOMNAMBULISM at
3 SLEEP TALKING
4 BRUXISM CNI
5

NOCTURNAL
ENURISIS

thermal Chantress

BARELY REMI
GIT 3 4 GI
I

Rodina

Suprachiasmatic circadian
Nucleus Rhythm
B Clock

Lal Hunger
center
Loral preoptic theist center

Holy

ventromedial satiety
Holly Tate

Epona optic
Ilocky
ADI
SIP
O
Ventricular
Para oxytocin
Ilkley
I that adapt
A 89 H te C Is
Anton ftp.th
E.ly
Poster Hypothalamus wore

It Accumberf tpleasure
center
Ideal fort

bundle
Addition
enter
cds
Gastro Intestinal
System

WE

Get yall Ilayi


I I
Strongest Submosa
Layer

Serosa Absent
Esophagus
Jon

MEISSNERS Emad
PLEXUS
GI Seaton

Auerbach
MYENTERIC plenty 7
PLEXIS
oemotility

MAX E seat
GIFT
AID
LEH Highly
Aad
Most Boosie

Secretion K'É
681

Bonner Metroducing
Glands in
Gland Judean
Secretion's
Saka
Major Salivary Submandibular
Stab
Gland

minor
Salivary Ebner's Gland

Gland Kal Lipases

Enzymes
31

ptyalin e
cant

I Lid
Lipase
No Enzymes for Protein

Eg
Contain Ab
If
Herve Innervation
parotid gland then
9
th
X b d bul
S bmandibular

7the Salglan oWWt.eo

8 02
8
00
o

a psmccf.cz
mom
steamy

Biz
cerebiriform
App era
of G Re

favela cell
Hypeplasia

DE menitrier Disease

Aare 7
Eff

mach
Dylan Inst

Gastric Lipids

Pepsinogen Roten
Note

Pepsinogen pep

Substances their

Absorption Sites

felt Duodena

Bq
Lotty EA't
B12 Toluene
Pancreatic Secretion's
Enzymes

D P canty

Chotrypsinogen iii
Protein

p Lipase Ltd
pp
Torypsinogen IIrypsin
Duodenume

maggot
I
Eat secretion

Aichi tedchyme
Diet

Produce 5Defence
lysozyme

I
Gaitan CcK
pz_
I Seaton
imply
t

take
Subsas with fast
Eric Emptying I

carat paten Lipid

Hone Broad During


fasting Ghrelin
bl g Glenelg
Anger

GE motility

pacemaker caff
t Int of
cells
I
SPIKE POTENTIALS

it M m
de

f
BER SLOWWAVEL

NotAfx

d
u r

n n
I L

M M
nd
M

BER Frequently
Duodenum I
ILEUM 8

CECUM
I
Peristalsis
Fei

t
II

Sub p
ÉGMENTATON
MAJI

to

aim
MAINLY HE'S I Digestion

MIGRATORY MOTI
COMPLEX

enfatingstate
St ch Col
a colors
Eastick of IIT sweeper

meted motive
I 90min
MIKEY ELGIN

ELECTRICAL
BER S Waves
MOTILITY

MECHANICAL Mmc

MOTILITY EASINI
FTD
FEI
Peristalsis Segment
mof
ENDOCRINE

SystEMnm

I
ant
pituitary
Acidophil c
Somatotropes
cells
Lactotropes

Inhibitory control By
Hypothalamus is shown on

y
2
Itysteg
pituitary hmm ACTH
stalk
Damaged gg
FSH

LH

Prolactin

Sheehan's
t

is

TSH
Boggs

FSH
LH
ACTH
Prolactin

GROWTH HORMONE

Anabolic catabolic

fund's
Adipose tissue

Muscles

Bones viscera

Li
Liver

Diabitogenic Hormone

I Gath Hormone

children

Adults

Looker Dwarfism 9
p o

Defect In
Gtl

ILG F

Thyroid gland
Step's In Tz Ty Production

D OXIDATION

2 ORGANIFICATION

3 COUPLING

MEDIATED BY
MID MILD 31

D WOLF CHAIKOFF
crim ummm

Effect
Lwgols T T Production
T
The

Iodine I
vascularity
I Size

2
Job Base Done

Effects
Is Induced Hyperthyroidism

I
Seen after Is Soon
usually

2 Thyrotoxicosis
thyrotoxicosis all
i
factit Excessive Intake

of thyroid
Hormones

function's catabolic

T Glucose

T Lipolysis
T
proteolysis
oly
T BMR

Thermogenic
Brain Development
Heart Rate Inc
Bone Growth Inc

congenital Gitinism
Hypothyroidism It

Thyroid Hormones Receptor's


t
Hyperthyroidism

Most Reliable Sign

Hypothyroidism

Most Reliable Sig

Insulinmum

Produced
from
Hormone

Produced
In Equeimolay
Concentration

marker
of

Insulin
Stored
along
withe
Structure L 21

p 30

f cell Sensitises

blood Glo Jewels I

69th the Help of

Release
Insulin
from f cells
t
occur's with the
Help of closure

of

Insulin Independent Glu

is Seen in
uptake

acts
Insulin on cells

U n which R to's 7
VIA which Receptor's

Action of Insulin on

Joon's
Cortisol
Protein's

Carbohydrates

Lipids
Bones

Blood vessels

GIT

R dud e
Reproductive
System

Immune
System

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