NATURAL HISTORY OF: MOLAR PREGNANCY

CONCEPT : It is a rare mass or tumor that forms inside the uterus at the beginning of a pregnancy. It is a type of gestational trophoblastic disease (GD)

PREPATHOGENIC PERIOD PATHOGENIC PERIOD

HOST FACTORS : Age of the mother. Death: Death of the mother due to metastasis, hemorrhage, hemodynamic aspects an
Previous molar pregnancy.
anemia.
Chronic condition: Cancer can develop after a normal pregnancy, however, it is most often
associated with a complete hydatidiform mole.
AGENT FACTORS: endogenous estrogens, diet Disability: Physical Effects Sterility. Spontaneous abortions. Ectopic pregnancies. Stillbirths of
high in beta carotene, diet high in animal fat, children. Menstrual disorders. Hemorrhage. Infections
ethnicity, ABO blood group Disease: Choriocarcinoma is a generally aggressive type of placental cancer, which is why it is part of the
spectrum of gestational trophoblastic diseases. It is characterized by frequent metastasis to the lungs.
ENVIRONMENTAL FACTORS: environmental
toxins, smoking, alcohol consumption, Signs and Symptoms: Abnormal uterine bleeding in the first trimester of pregnancy
socioeconomic status and exposure to CLINICAL HOTZONTE
herbicides
Trophoblast disease: it is one that derives from the outermost part of the egg and it is not known whether it is
pathological from its differentiation or is a product of the degeneration of a healthy trophoblast.
PREVENTION LEVELS
PRIMARY LEVEL SECUNDARY LEVEL TERTIARY LEVEL
HEALTH PROMOTION EARLY DIAGNOSIS TIMELY TREATMENT LIMITATION OF DAMAGE REHABILITATION

health education, timely laboratory tests After an initial diagnosis, a If sensitization to the RhD Depends on the effects of
medical tests, using your β HCG > 100,000 mIU/mL, careful history and examination antigen is detected, the titers chemotherapy
sexuality responsibly transvaginal hemorrhage in are done to ensure that the should be repeated every
the first trimester of cancer has not spread to other month until 24 weeks of
pregnancy, Size organs. Chemotherapy is the gestation, and every two
excessive uterus, fetal parts main type of treatment. weeks thereafter.
and heartbeat are not Hysterectomy and radiation
perceived therapy are rarely required.
,ultrasound of the pelvis, chest
x-ray
.
 NATURAL HISTORY OF: HYPEREMESIS GRAVIDIC |
CONCEPT: Nausea is present in 70-80% of pregnant women and vomiting in 50% of cases. Classic pregnancy vomiting occurs once or twice a
day.
PREPATHOGENIC PERIOD PATHOGENIC PERIOD
GUEST FACTORS: CELLULAR AND SIGNS AND DISEASE: INABILITY: DAMAG CHRONIC DEATH OR
pregnant women from the TISSUE SYMPTOMS: -Liver E: STATE: RECOVERY:
first half of pregnancy CHANGES: Vomiting appears disorders Sometimes to in the severe -Isolation from
In multiple in the morning, -Very severe be able to embryo dehydration its natural
AGENT FACTORS: pregnancies and Lose weight, Pain weight loss ingest environment
Etiological-It is the in gestational palpitations and -Depression -Use of
presence of intense and trophoblastic confusion. -Gallbladder antihemetics.
persistent nausea and disease.http://e disease -Recovery
vomiting during pregnancy s.wikipedia.org/ involves instant
that can lead to wiki/Hiper hydration.
dehydration. %C3%A9mesis_ Intravenously
grav -medication
ENVIRONMENTAL %C3%ADdica - administration
FACTORS: It occurs at any cite_note-
socioeconomic level. medlineplus-6
Any geographic area. has been
related to high
levels
PREVENTION LEVELS
PRIMARY LEVEL SECUNDARY LEVEL TERTIARY LEVEL
HEALTH SPECIFIC EARLY TIMELY TREATMENT LIMITATION OF DAMAGE REHABILITATION
PROMOTION PROTECTION DIAGNOSIS
Promote social To avoid the use of Detection of the Intravenous hydration often includes A complete diet that Through therapies
participation tobacco disease when you electrolyte supplementation because includes proteins,
activities, provide Avoid drinking alcohol go to a doctor. persistent vomiting often leads to carbohydrates, vitamins
information Eat fruits and electrolyte deficiency. For the same and minerals in adequate
through teaching vegetables reason, the administration of quantities.
materials. Drink plenty of fluids thiamine or vitamin B supplements is Go to the doctor if you
indicated. suffer from frequent
vomiting
 NATURAL HISTORY OF PREMATURE
PREPATHOGENIC PERIOD PATHOGENIC PERIOD
CONCEPT: Live newborn before completing 37 weeks of gestation

DEATH
 TISSUE CHANGES RECOVERY DEATH
AGENT: - Pregestational risk factors: -
History of premature birth. -Maternal age of SIGNS AND SYMPTOMS COMPLICATIO AFTERMAT Immediate and
16 or over 35 years. -Maternal malnutrition. NS H quality care for
-Low socio-economic level. -Drug premature
addictions. Prenatal risk factors -Little or no newborns,
prenatal care. -Genitourinary infections. - Patent ductus Sensorineural ICU.
Wrong estimation of gestational age. - arteriosus, apnea, hearing loss,
Cyanosis, jaundice, pallor or RDS or hyaline
Polyhydramnios. -Bleeding in the third language
Within the uterus, many of plethora. Skin: red, membrane,
trimester of pregnancy. defects, vision
the organic functions of the translucent, obvious blood intraventricular loss, mental
fetus, such as respiration, vessels, lack of hemorrhage,
retardation
HOST: Neonate born before 37 SEC. nutrient provision, subcutaneous fat; presence retinopathy of
metabolism, excretion, and of lanugo; large head size in prematurity,
ENVIRONMENT: Extrauterine adaptation defense against infection, relation to the body; skull: hydroelectrolyte
folding bones smooth and imbalance,
are largely performed by the nutritional
placenta and the mother. flat fontanel; Ears: minimal
imbalance, sepsis,
The transition forces a cartilage, folded folded; nails: thermoregulation
series of changes in the short and soft. disorders.
functioning of organs and
systems
PRIMARY PREVENTION SECONDARY PREVENTION TERTIARY PREVENTION
Health promotion Specific protection Early diagnostic Timely treatment Damage Limitation Rehabilitation

* Preparation of Prenatal control should be * Prevention of low weight in the *Transfer to neonatal
*Promotion and medical history. * *Prenatal Control. aimed at the detection and RN. *Management of intensive care unit. TX
guidance on family Promotion so that *Identification of signs control of obstetric risk factors, Prematurity involves the timely
planning. the woman goes the prevention, detection and diagnosis and treatment of the
PALIATVO Maintenance
and symptoms, of the vital functions of
to consult with her treatment of anemia, threat of premature birth, as well
presumptive, probable
*Health self-care partner or a family preeclampsia, cervicovaginal as the acceleration of the the preterm newborn.
measures. member, to and certain of infections and urinary biosynthesis of surfactant factors
*Establishment of integrate the pregnancy. infections, hemorrhagic in the fetal lung and the
comprehensive family into the *Confirmation of complications of pregnancy, appropriate management of the
diagnosis. control of the pregnancy. intrauterine growth retardation preterm (preterm) newborn.
pregnant woman. and other intercurrent
pathologies with the
pregnancy.