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1
Chapter 10
10.2 Thiamin
10.2.1 Identify the coenzyme form of thiamin.
10.2.2 Describe dietary sources of thiamin.
10.2.3 Explain the consequences of a thiamin deficiency or toxicity.
10.3 Riboflavin
10.3.1 Name the coenzyme forms of riboflavin and discuss the vitamin’s primary functions in
the body.
10.3.2 Identify dietary sources of riboflavin.
10.3.3 List the signs and symptoms of a riboflavin deficiency.
10.4 Niacin
10.4.1 Name the coenzyme forms of niacin and discuss the vitamin’s primary functions in the
body.
10.4.2 Identify dietary sources of niacin.
10.4.3 Explain the signs and symptoms of a niacin deficiency or toxicity.
10.4.4 Discuss the clinical use of megadoses of niacin.
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Education.
2
10.6 Biotin
10.6.1 Summarize the importance of biotin in the body.
10.6.2 Identify dietary sources of biotin.
10.6.3 Describe the signs of a biotin deficiency and identify the primary cause of the deficiency.
10.8 Folate
10.8.1 Name the coenzyme form of folate and discuss the vitamin’s role in the body.
10.8.2 Identify sources of folate.
10.8.3 Compare the absorption of folate from natural and synthetic sources.
10.8.4 Explain the consequences of a folate deficiency in both pregnant and nonpregnant
adults.
10.10 Vitamin C
10.10.1 Describe the role of vitamin C in collagen synthesis, antioxidant activities, iron
absorption, and immune function.
10.10.2 Identify dietary sources of vitamin C.
10.10.3 Explain the consequences of a vitamin C deficiency or toxicity.
Overview
Chapter 10 discusses water-soluble vitamins and vitamin-like substances, including choline and
taurine. Basic information about each water-soluble vitamin’s functions, dietary sources, and
dietary adequacy is provided. The last section of the chapter focuses on the role of diet in cancer.
1. Have students read the Case Study in the opening of the chapter and answer the questions
that follow. After they have read the chapter, students should answer the questions again and
compare their answers to the response provided at the end of the chapter.
2. Have students answer the Quiz Yourself questions. Students should save their responses and
answer the questions again, after they have read the chapter.
3. Assign Connect® and LearnSmart® activities for Chapter 10.
4. Assign the Personal Dietary Analysis feature in Connect or at the end of the chapter.
5. Have each student choose a water-soluble vitamin and search medical literature for
information about the vitamin’s deficiency disease. The student should prepare a brief report
concerning the signs and symptoms of the disease and its prevalence in the world.
6. Have students choose a water-soluble vitamin and search medical literature for information
concerning therapeutic uses of the particular micronutrient or risk of toxicity disorders from
ingesting excesses of the vitamin. Students should report their findings for the class.
7. Have students choose a water-soluble vitamin and summarize the DRI report that describes
how its RDA or AI was determined. The reports can be accessed online at
http://www.iom.edu/Activities/Nutrition/SummaryDRIs/DRI-Tables.aspx .
8. Assign the Critical Thinking questions and Practice Test at the end of the chapter.
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Education.
4
II. Thiamin
A. Thiamin functions as part of the coenzyme thiamin pyrophosphate (TPP). TPP
participates in chemical reactions that removes a carbon dioxide molecule from a larger
compound.
1. Such reactions are needed for catabolism of carbohydrate to release energy,
metabolism of branched-chain amino acids, and synthesis of neurotransmitters.
B. Whole-grain products, pork, legumes, and orange juice are good sources of thiamin.
Overheating food destroys the vitamin.
1. Thiamin-deficient diet results in beriberi.
2. Wernicke-Korsakoff syndrome is a degenerative brain disease associated with
thiamin deficiency. Occurs primarily among alcoholics in the United States.
Alcohol reduces thiamin absorption and increases the vitamin’s excretion.
3. Toxicity is rare.
III. Riboflavin
A. Riboflavin is a component of flavin mononucleotide (FMN) and flavin adenine
dinucleotide (FAD). These coenzymes play key roles in energy, fatty acid, and folate
metabolism.
B. Milk, yogurt and other milk products, enriched cereals, and liver are among the best
sources of riboflavin. Riboflavin’s chemical structure is destroyed by exposure to light.
C. Most Americans consume adequate amounts of riboflavin. The vitamin is rapidly
excreted in urine.
1. Ariboflavinosis is the riboflavin deficiency disorder but the condition is rare in
the United States.
a. Glossitis and cheilosis are signs of the deficiency.
IV. Niacin
A. Niacin has two forms: nicotinic acid and nicotinamide. The body uses these
substances to form the coenzymes nicotinamide adenine dinucleotide (NAD) and
nicotinamide adenine dinucleotide phosphate (NADP). Niacin-containing coenzymes
participate in at least 200 chemical reaction, including pathways involved in energy
metabolism.
B. Major food sources of niacin include enriched cereals, beef liver, and tuna. Refer
students to Table 10.4.
1. The amino acid tryptophan can be converted to niacin. Sixty milligrams of
tryptophan yield about 1 mg niacin.
C. Niacin recommendations are provided as niacin equivalents (NEs).
1. In the United States, people with alcoholism, anorexia nervosa, and those with
rare disorders that disrupt tryptophan metabolism are at risk of niacin deficiency.
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Education.
5
a. Early signs and symptoms of mild niacin deficiency are poor appetite,
weight loss, and weakness.
b. If the deficiency state continues, the condition becomes pellagra. The
classic signs and symptoms of pellagra are dermatitis, diarrhea, dementia,
and death—the “4 Ds of pellagra.” Refer students to Figure 10.6.
2. The UL for niacin is 35 mg/d. Megadoses of niacin-containing supplements can
cause skin flushing, GI tract upset, and liver damage.
V. Pantothenic Acid
A. Pantothenic acid is a component of coenzyme A, the coenzyme that helps release
energy from macronutrients and is needed for fatty acid synthesis.
B. Pantothenic acid is available from a wide variety of foods. Rich food sources include
fortified cereals, beef and chicken liver, and sunflower seeds.
C. Most Americans consume the AI amount or more daily, so dietary deficiencies are
rare.
1. People who abuse alcohol may develop pantothenic acid deficiency,
particularly if their overall diet is nutritionally inadequate.
2. No UL has been established for pantothenic acid, because there have been no
reports of toxicity.
VI. Biotin
A. In its coenzyme form, biotin participates in chemical reactions that add carbon dioxide
to other compounds. The coenzyme is essential for regenerating oxaloacetate in the citric
acid cycle (refer students to Figure 8.16).
B. Good food sources of biotin include liver, eggs, and peanuts.
C. Severe biotin deficiencies are rare because intestinal bacteria synthesize the vitamin
and it is widespread in foods.
1. Avidin is a protein in raw egg white that binds biotin, inhibiting its absorption.
Refer students to the Did You Know? feature in section 10.6.
2. No UL for biotin because the micronutrient appears to be nontoxic.
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Education.
6
VIII. Folate
A. Folate is the name for a group of related compounds that includes folic acid.
1. Folic acid is the synthetic form vitamin are converted to a group of coenzymes
called tetrahydrofolic acid (THFA).
2. THFA accepts a single-carbon group (e.g., CH3) from one compound and
transfers it to another compound.
a. As cells prepare to divide, they need THFA to make DNA.
b. Certain roles of folate and vitamin B-12 are interrelated. Refer students
to Figure 10.9.
B. Leafy vegetables, liver, legumes, and orange juice are good sources of natural folate.
Enriched grain products are among the richest sources of folic acid in the American diet.
Folate is easily destroyed by heat, oxidation, and UV light. Refer students to section 10.8
for information about dietary folate equivalents.
C. Folate naturally occurs in foods with a string of glutamates attached to its basic
structure. Refer students to Appendix D. Folic acid has only one glutamate. No enzymatic
activity is needed to digest folic acid. Folate, however, must be broken down to form
folic acid, which reduces its bioavailability.
1. Vitamin B-12 is need to activate folate after it is absorbed in the small intestine.
D. The risk of folate deficiency increases during periods of rapid growth (e.g.,
pregnancy). Alcohol consumption and use of certain medications increase the risk of
deficiency.
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Education.
7
1. Red blood cell (RBC) formation requires rapid cell division of RBC precursor
cells. If folate is deficient, these cells cannot divide normally, and some abnormal
RBCs (megaloblasts) enter the bloodstream, resulting in megaloblastic anemia.
Refer students to Figure 10.10.
2. During pregnancy, folate deficiency can result in neural tube defects in the
embryo. Refer students to Figure 10.11.
a. Women of childbearing age should consume 400 µg of folic acid daily
to help prevent neural defects.
b. The prevalence of neural tube defects decreased by about 30% since
folic acid was included as an enrichment vitamin.
3. Folate deficiencies are associated with increased blood homocysteine. Folic
acid supplementation may reduce risk of stroke.
a. Supplementation doesn’t appear to slow the progression of Alzheimer’s
disease.
4. Folate naturally in food isn’t toxic, but the UL for folic acid is 1000 µg/day.
a. Folic acid can cure anemia that occurs with B-12 deficiency, but it
doesn’t prevent the serious nervous system damage that occurs in a B-12
deficiency.
2. Most cases of vitamin B-12 deficiency result from problems that interfere with
the vitamin’s absorption and not from inadequate intake.
a. Food-cobalamin malabsorption is caused by the inability to release
cobalamin from food protein. Reduced stomach acid contributes to the
malabsorption.
i. Causes of reduced stomach acid include aging, gastritis, and
medications.
ii. To overcome the lack of acid, people can take synthetic forms of
the vitamin that don’t need to be separated from animal protein.
b. People with pernicious anemia don’t synthesize intrinsic factor in their
stomach. Injections of vitamin B-12 are needed to treat this disorder.
3. Vitamin B-12 is also needed for neural tube formation in embryos, and the
vitamin may be helpful for improving psychological health.
X. Vitamin C
A. Vitamin C doesn’t function as part of a coenzyme, but serves as a nutrient cofactor
that facilitates certain chemical reactions. Vitamin C has a variety of roles in the body,
including collagen synthesis, antioxidant activity, and immune function.
1. Vitamin C participates in reactions that form and maintain collagen. Also, the
vitamin donates electrons to radicals or to vitamin E (antioxidant activity).
2. Taking high doses of vitamin C may be harmful because in high doses, the
vitamin acts as a prooxidant.
3. White blood cells have relatively high amounts of vitamin C, which may limit
free radical damage within the cells.
4. Vitamin C is necessary for the synthesis of important substances including bile,
certain neurotransmitters, and thyroxin.
5. Vitamin C enhances the absorption of iron from plant foods.
B. Plant foods are the best dietary sources of vitamin C. The vitamin is unstable in the
presence of heat, oxygen, light, alkaline conditions, and the minerals copper and iron.
1. Vitamin C is absorbed in the small intestine. As intake of the vitamin increases,
the rate of absorption decreases.
2. According to MyPlate, adults should eat 3.5 to 5 cups of fruits and vegetables
daily. People should choose vitamin C-rich produce to meet this
recommendation. Refer students to Table 10.10 for some foods that are rich
sources of the vitamin.
D. Vitamin C deficiency is called scurvy. Signs and symptoms of scurvy include
petechiae, bruising, spongy gums that bleed easily, swollen and sore joints, and tooth
loss. Without treatment, people with scurvy die, most likely from infection.
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Education.
9
Copyright © 2016 McGraw-Hill Education. All rights reserved. No reproduction or distribution without the prior written consent of McGraw-Hill
Education.
10
3. Some rapidly dividing cells form benign tumors, which are not considered
cancerous and do not spread.
4. A carcinogen is an environmental factor that triggers cancer. Refer students to
Figure 10.15, which illustrates the steps in cancer development and progression.
B. Cancer has numerous risk factors, including advancing age, family history, tobacco
use, and radiation exposure.
1. Nutrition-related risk factors include alcohol consumption and having excess
body fat.
2. Cancer causation is a complex process and may not be due to a single factor.
C. Certain foods and beverages promote cancer development.
1. Alcohol is a carcinogen and increases the risk of mouth, throat, esophagus,
larynx, liver, breast, colon, and rectal cancers.
2. Consuming foods contaminated with aflatoxin increases risk of liver cancer.
3. Consuming large amounts of processed/and or red meat is associated with
increased risk of colon cancers.
4. Fried, grilled, and broiled meats may increase the risk of colorectal and
pancreatic cancer due to the production of heterocyclic amines when the foods are
cooked at high temperatures. Charred portions of food should be avoided.
D. Dietary factors that may reduce risk of cancer include eating high amounts of fruits
and vegetables that are rich sources of vitamin C and phytochemicals.
1. Results of studies do not provide conclusive evidence that folate intake or
taking vitamin C or E supplements reduces cancer risk.
E. According to the American Cancer Society, people can reduce their risk of cancer by
limit alcohol consumption; achieving and maintaining a healthy weight; adopting a
physically active lifestyle; and eating a healthy diet that limits intake of red and processed
meats and emphasizes plant foods.
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attention to regiments or brigades, so sudden was the affair, formed
two masses, one of which under general Dilke marched hastily
against Ruffin, and the other under colonel Wheately against Laval.
Duncan’s guns ravaged the French ranks; Laval’s artillery replied
vigorously; Ruffin’s batteries took Wheately’s column in flank; and
the infantry on both sides pressed forward eagerly, and with a
pealing musketry; but, when near together, a fierce, rapid, prolonged
charge of the British overthrew the first line of the French, and,
notwithstanding its extreme valour, drove it in confusion, over a
narrow dip of ground upon the second, which was almost
immediately broken in the same manner, and only the chosen
battalion, hitherto posted on the right, remained to cover the retreat.
Meanwhile Brown, on receiving his orders, had marched headlong
against Ruffin. Nearly half of his detachment went down under the
enemy’s first fire; yet he maintained the fight, until Dilke’s column,
which had crossed a deep hollow and never stopt even to re-form
the regiments, came up, with little order indeed, but in a fierce mood,
when the whole run up towards the summit; there was no slackness
on any side, and at the very edge of the ascent their gallant
opponents met them. A dreadful, and for some time a doubtful, fight
ensued, but Ruffin and Chaudron Rousseau, commanding the
chosen grenadiers, both fell mortally wounded; the English bore
strongly onward, and their incessant slaughtering fire forced the
French from the hill with the loss of three guns and many brave
soldiers.
The discomfitted divisions, retiring concentrically, soon met, and
with infinite spirit endeavoured to re-form and renew the action; but
the play of Duncan’s guns, close, rapid, and murderous, rendered
the attempt vain. Victor was soon in full retreat, and the British
having been twenty-four hours under arms, without food, were too
exhausted to pursue.
While these terrible combats of infantry were fighting, La Peña
looked idly on, neither sending his cavalry, nor his horse-artillery, nor
any part of his army, to the assistance of his ally, nor yet menacing
the right of the enemy, which was close to him and weak. The
Spanish Walloon guards, the regiment of Ciudad Real, and some
Guerilla cavalry, indeed turned without orders, coming up just as the
action ceased; and it was expected that colonel Whittingham, an
Englishman commanding a powerful body of horse, would have
done as much; but no stroke in aid of the British was struck by a
Spanish sabre that day, although the French cavalry did not exceed
two hundred and fifty men, and it is evident that the eight hundred
under Whittingham might, by sweeping round the left of Ruffin’s
division, have rendered the defeat ruinous. So certain, indeed, was
this, that colonel Frederick Ponsonby, drawing off the hundred and
eighty German hussars belonging to the English army, reached the
field of battle, charged the French squadrons just as their retreating
divisions met, overthrew them, took two guns, and even attempted,
though vainly, to sabre Rousseau’s chosen battalions.
Such was the fight of Barosa. Short, for it lasted only one hour and
a half, but most violent and bloody; for fifty officers, sixty serjeants,
and above eleven hundred British soldiers, and more than two
thousand Frenchmen were killed and wounded; and from the latter,
six guns, an eagle, and two generals (both mortally wounded) were
taken, together with four hundred other prisoners.
After the action, Graham remained some hours on the height, still
hoping that La Peña would awake to the prospect of success and
glory, which the extreme valour of the British had opened. Four
thousand men and a powerful artillery had come over the Santi Petri;
hence the Spanish general was at the head of twelve thousand
infantry and eight hundred cavalry, all fresh troops; while before him
were only the remains of the French line of battle retreating in the
greatest disorder upon Chiclana. But all military feeling Appendix, No. IX.
being extinct in La Peña, Graham would no longer Section 1.
endure such command. The morning of the 6th saw the British filing
over Zaya’s bridge into the Isla.
Vol. 3, Plate 9.
BATTLE of BAROSA
5th March, 1811.
London Published by T. & W. Boone Novr 1830.
On the French side, Cassagne’s reserve came in from Medina, a
council of war was held in the night of the 5th, and Victor, although of
a disponding nature, proposed another attack; but the suggestion
being ill received, nothing was done; and the 6th, Admiral Keats,
landing his seamen and marines, dismantled, with exception of
Catalina, every fort from Rota to Santa Maria, and even obtained
momentary possession of the latter place. Confusion and alarm then
prevailed in the French camp; the duke of Belluno, leaving garrisons
at the great points of his lines, and a rear guard at Official Abstracts of
Chiclana, retreated behind the San Pedro, where he Military
MSS.
Reports,
S K I R M I S H AT P O M B A L .
Pack’s brigade and the cavalry, the first, third, fourth, fifth, sixth,
and light divisions, and the Portuguese troops, which were attached,
like the ancient Latin auxiliaries of the Roman legion, to each British
division, were assembling in front of the enemy on the 10th; when
Massena, who had sent his baggage over the Soure river in the night
by the bridge of Pombal, suddenly retired through that town. He was
so closely followed by the light division, that the streets being still
encumbered, Ney drew up a rear-guard on a height behind the town,
and threw a detachment into the old castle of Pombal. He had,
however, waited too long. The French army was moving in some
confusion and in a very extended column of march, by a narrow
defile, between the mountains and the Soure river, which was
fordable; and the British divisions were in rapid motion along the left
bank, with the design of crossing lower down, and cutting Massena’s
line of retreat. But darkness came on too fast, and the operation
terminated with a sharp skirmish at Pombal, whence the ninety-fifth
and the third caçadores of the light division, after some changes of
fortune, drove the French from the castle and town with such vigour,
that the latter could not destroy the bridge, although it was mined.
About forty of the allies were hurt, and the loss of the enemy was
somewhat greater.
In the night Massena continued his retreat, which now assumed a
regular and concentrated form. The baggage and sick, protected by
the reserve cavalry, marched first; these were followed by the eighth
corps; and the sixth, with some light cavalry, and the best horsed of
the artillery, were destined to stem the pursuit. Ney had been
ordered to detach Marcognet’s brigade on the 10th, from the Lys, to
seize Coimbra; but some delay having taken place, Montbrun was
now appointed for that service.
Lord Wellington’s immediate object was to save Coimbra, and he
designed, by skilful, rather than daring, operations, to oblige
Massena to quit the Portuguese territory: the moral effect of such an
event, he judged, would be sufficient; but as his reinforcements were
still distant, he was obliged to retain the fourth division and the heavy
cavalry from the relief of Badajos, and was therefore willing to strike
a sudden stroke, if a fair occasion offered. Howbeit the country was
full of strong positions, the roads hollow and confined by mountains
on either hand, every village formed a defile; the weather also, being
moderate, was favourable to the enemy, and Ney, with a wonderfully
happy mixture of courage, readiness, and skill, illustrated every
league of ground by some signal combination of war.
Day-break, on the 12th, saw both armies in movement, and eight
miles of march, and some slight skirmishing, brought the head of the
British into a hollow way, leading to a high table-land on which Ney
had disposed five thousand infantry, a few squadrons of cavalry, and
some light guns. His centre was opposite the hollow road, his wings
were covered by wooded heights, which he occupied with light
troops; his right rested on the ravine of the Soure, his left on the
Redinha, which circling round his rear fell into the Soure. Behind him
the village of Redinha, situated in a hollow, covered a narrow bridge
and a long and dangerous defile; and, beyond the stream, some very
rugged heights, commanding a view of the position in front of the
village, were occupied by a division of infantry, a regiment of cavalry,
and a battery of heavy guns; all so skilfully disposed as to give the
appearance of a very considerable force.
C O M B AT O F R E D I N H A .
C O M B AT O F C A S A L N O VA .
C O M B AT O F F O Z D ’ A R O N C E .
The 15th, the weather was so obscure that the allies could not
reach the Ceira, before four o’clock in the evening, and the troops,
as they came up, proceeded to kindle fires for the night; thinking that
Ney’s position being strong, nothing would be done. The French right
rested on some thickly wooded and rugged ground, and their left
upon the village of Foz d’Aronce, but lord Wellington, having cast a