Review On Cardiovascular Diseases: BY: Fidel G. Yongque III, RN

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Review on

Cardiovascular
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Diseases
BY: Fidel G. Yongque III, RN
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MANAGEMENT OF CLIENTS WITH
MYOCARDIAL INFARCTION

 Coronary Artery Disease (CAD) – OBSTRUCTION

 Causes:

1. Atherosclerosis
2. Clot formation
3. Vasospasm
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RISK FACTORS

PRECIPITATING FACTORS: 4E’S

EXERCISE
EMOTIONAL EXTREMES
EATING A HEAVY MEAL
ENVIRONMENTAL – cold (promotes vasoconstriction)
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Signs and Symptoms of CAD
1. ANGINA PECTORIS – chest pain

PATTERNS:
a. STABLE ANGINA – predictable chest pain; relieved by rest or
nitroglycerine or both.
b. UNSTABLE ANGINA – paroxysmal chest pain triggered by an
unpredictable degree of exertion or emotion, which
may occur at night. It must be treated as a medical
emergency with the client receiving immediate medical
attention
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Signs and Symptoms of CAD

c. VARIANT/PRINZMETAL’S ANGINA – longer duration, may occur while


the client is at rest. It results from coronary artery spasm
and may be associated with elevation of ST segment on
ECG.

d. NOCTURNAL ANGINA – REM sleep during dreaming

e. ANGINA DECUBITUS – paroxysmal chest pain that occurs when the


client reclines and lessens when the client sits
or stands up.
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Signs and Symptoms of CAD

f. INTRACTABLE ANGINA – chronic, incapacitating angina;


unresponsive to intervention.

g. POST INFARCTION – pain occurs after MI, when residual ischemia


may cause episodes of angina.
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Diagnostic Tools

 ECG – ST elevation or ST depression

 Fasting Lipid Profile – elevated cholesterol level, elevated LDL

Normal Total cholesterol level: less than 200mg/dL


 Coronary Angiography/Angiogram/Cardiac Catheterization –
most accurate test
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General Management for CAD

 AHA recommends that people with angina control their modifiable


risk factors and seek prompt treatment for episodes of chest pain:
ABCDE
 A for aspirin & antianginal therapy

 B for beta-blocker therapy & BP control

 C for cigarette & cholesterol

 D for diet & diabetes

 E for education & exercise


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Lifestyle Modification

 LOW FAT, LOW CHOLESTEROL DIET

(+) HDL (polyunsaturated fat) – ex. Vegetable oil (olive oil,


corn oil, sunflower & safflower oil)

(-)LDL (saturated fat) – animal oil


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ANTICHOLESTEROL OR
ANTILIPEMICS

 Fibrates – Fenofibrate, Gemfibrozil (Lopid)

 Statins: Atorvastatin (Lipitor), Simvastatin (Zocor), Rosuvastatin


(Crestor)

*AVOID fibrates & statins taken together – prevent rhabdomyolysis


(rupture of muscle fibers)
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Medications – Relieve Acute Pain and
Restore Coronary Artery Flow

 Opiate analgesics
 Vasodilators
 Beta-adrenergic Blockers
 Calcium-channel Blockers
 Antiplatelets
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Nitrates
 Nitroglycerine – photosensitive = use dark-colored containers

a. Sublingual – normal: tingling, fizzling = potent. After 3-6mos


(potency)
b. Transdermal patch – anterior chest wall

Protection: 24 hours
Placement: 12 hours = nitrate-free periods
Swim/shower? = yes – water resistant
c. Dosing: Maximum of 3 doses with 5 mins interval for each dose
d. HEADACHE is expected signs and symptoms
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2. MYOCARDIAL INFARCTION – prolonged ischemia, endpoint of


CHD

Signs & Symptoms:


a. Atypical chest, abdominal, back or stomach pain

b. Nausea or dizziness

c. Shortness of breath and DOB

d. Unexplained anxiety, weakness or fatigue

e. Palpitations, cold sweat or paleness


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Diagnostic Tests for MI

 Increase WBCs (leukocytosis) -10,000mm3 to 20,000mm3

 Increased ESR – inflammation

 (+) cardiac enzymes: CPK-MB

 (+) cardiac proteins: Troponin I & T

 ECG Changes: Pathologic Q wave

ST elevation
T inversion
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Management for MYOCARDIAL
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INFARCTION
PLEASE REMEMBER MONAF!!!
 MORPHINE – decreased cardiac workload = O2 demand =
relieved of pain
 OXYGEN
 NITRATES – dilate coronary artery
 ASA – platelet aggregator inhibitor = prevent clot formation
 FIBRINOLYTIC/THROMBOLYTIC AGENT – dissolve clot
formation. Ex: streptokinase, urokinase or tPA (tissue
plasminogen activator)
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Surgery

 PTCA (Percutaneous Transluminal Coronary Angioplasty)


 CABG (Coronary Artery Bypass Graft)
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Common Complications of
MYOCARDIAL INFARCTION

 CARDIOGENIC SHOCK

 DYSRHYTHMIAS

 HEART FAILURE

 PULMONARY EDEMA

 PULMONARY EMBOLISM

 RECURRENT MI
Change in
z condition of Activation of Formation of a
plaque in the platelets thrombus
coronary artery

Ischemia of
Coronary blood
Myocardial Cell tissue in the
supply less than
Death region supplied
demand
by the artery

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