PBL Icterus 3
PBL Icterus 3
PBL Icterus 3
T U T O R : D R . Z U L F A H M I D A H M . K E S
5. Describe the pathophysiology of the symptoms iv. Pathogenesis decrease hepatic uptake of
of jaundice! bilirubin conjugation
The convex anterior surface is divided into two lobes of the left lobe and
right lobe that is approximately 2 times the left lobe. In the area between
falsiform with gall bladder ligament in the right lobe can sometimes be found
quadrate lobe and caudate lobe which is normally covered by the inferior
vena cava and the ligament venosum the posterior surface. The liver is
divided into 8 segments with different functions. Basically, the line Cantile
contained from the vena cava to the heart of the gallbladder has been split
into two functional lobes, and in the presence of an area with relatively little
vascularization, sometimes used as a boundary resection. Further division
into eight segments based on the flow branch blood vessels and bile ducts
owned by each segment.
Intrahepatic bile ducts are slowly together to form larger ducts that can
channel the liver bile into eight segments. In the right liver segment,
combined these branches form a channel in the anterior and posterior are
then joined to form the right hepatic duct. In some people, the right hepatic
duct is approximately 1 cm outside the liver. The ducts are then joined by
three segments of the segment of the left heart (left hepatic duct) into hepatic
duct communists.
2.HISTOLOGY
1. Over production
Increasing the amount of hemoglobin released from red blood cells that are
old or who have hemolysis will increase the production of bilirubin.
Destruction of erythrocytes cause hyperbilirubinemia most frequently due
to intravascular hemolysis (autoimmune disorders, microangiopathy or
hemoglobinopathies) or as a result of a large hematoma resorption.
Jaundice arising often called hemolytic jaundice.