Scribd Logo
Upload

Welcome to Scribd!

  • 582 views

    Cva (Npte)

    Uploaded by

    papermanner
    1) A CVA, or cerebrovascular accident, is a non-traumatic vascular event that causes a sudden neurological deficit. 2) Risk factors for CVA include modifiable lifestyle factors like smoking, hypertension, and obesity as well as non-modifiable factors like age, sex, and family history. 3) CVAs can be classified temporally, based on pathophysiology (ischemic vs. hemorrhagic), and by affected neuroanatomy such as the middle cerebral artery.

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as PPT, PDF, TXT or read online from Scribd

    Cva (Npte)

    Uploaded by

    papermanner
    582 views16 pages
    1) A CVA, or cerebrovascular accident, is a non-traumatic vascular event that causes a sudden neurological deficit. 2) Risk factors for CVA include modifiable lifestyle factors like smoking, hypertension, and obesity as well as non-modifiable factors like age, sex, and family history. 3) CVAs can be classified temporally, based on pathophysiology (ischemic vs. hemorrhagic), and by affected neuroanatomy such as the middle cerebral artery.

    Original Description:

    Original Title

    CVA (NPTE)

    Copyright

    © Attribution Non-Commercial (BY-NC)

    Available Formats

    PPT, PDF, TXT or read online from Scribd

    Did you find this document useful?

    Is this content inappropriate?

    1) A CVA, or cerebrovascular accident, is a non-traumatic vascular event that causes a sudden neurological deficit. 2) Risk factors for CVA include modifiable lifestyle factors like smoking, hypertension, and obesity as well as non-modifiable factors like age, sex, and family history. 3) CVAs can be classified temporally, based on pathophysiology (ischemic vs. hemorrhagic), and by affected neuroanatomy such as the middle cerebral artery.

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as PPT, PDF, TXT or read online from Scribd
    Download as ppt, pdf, or txt
    582 views16 pages

    Cva (Npte)

    Uploaded by

    papermanner
    1) A CVA, or cerebrovascular accident, is a non-traumatic vascular event that causes a sudden neurological deficit. 2) Risk factors for CVA include modifiable lifestyle factors like smoking, hypertension, and obesity as well as non-modifiable factors like age, sex, and family history. 3) CVAs can be classified temporally, based on pathophysiology (ischemic vs. hemorrhagic), and by affected neuroanatomy such as the middle cerebral artery.

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as PPT, PDF, TXT or read online from Scribd
    Download as ppt, pdf, or txt
    of 16

    CVA

    Verse for the Day

    “ If any of you lacks wisdom, he should ask God,


    who gives generously to all without finding fault, and
    it will be given to him. ”
    James 1:5
    TOPIC OUTLINE
     Definition & Epidemiology
     Risk Factors
     Classification
     Temporal
     Pathophysiological
     Neuroanatomical
    Definition
     Cerebrovascular accident
     NONTRAUMATIC
     VASCULAR CAUSE
     SUDDEN NEUROLOGIC DEFICIT
     Epidemiology
     Old > Young
     Male > Female
     Black > White
     Asian > US
    Risk Factors
     Modifiable  Non-modifiable
     Lifestyle  Race
     Cigarette smoking  Age
     Hypercholesterolemia  Sex
     Obesity  Previous stroke
     Heart disease
     Medical
     TIA
     LEADING RISK
     DM FACTOR
     Asymptomatic carotid  Hypertension
    bruit
     ↑ hematocrit/serum
    fibrinogen
    Classification: Temporal
     TIA
     Reversible ischemic neurologic deficit
     Stroke in evolution
     Completed stroke
    Classification: Pathophysiological
     Ischemic  Hemmorhagic
     Thrombotic  Intracerebral
     Embolic
     Subarachnoid
     Hypertensive
     Lacunar
     Other
    Classification: Neuroanatomical
     ICA
     MCA
     ACA
     PCA
     Lacunar
     Brainstem
    MCA
     Contralateral hemiplegia
     Contralateral hemianesthesia
     Frontal gaze palsy
     Aphasia/Aprosodia & Affective agnosia
     Apraxia
     Contralateral hemianopsia
     Dysphagia
    ACA
     Contralateral hemiplegia
     Contralateral hemianesthesia
     Uninhibited neurogenic bladder
     Primitive reflexes
     Disconnection apraxia
     Akinetic mutism/abulia
    PCA
     Visual impairment
     Visual agnosia
     Memory deficit
     Hemisensory deficit
    Lacunar
     Pure Motor
     Posterior limb of internal capsule
     Pure Sensory
     VPL nucleus of thalamus
     Dysarthria with facial weakness
     Anterior limb of internal capsule
     Dysarthria with clumsy hand
     Dorsal pons
     Ataxic hemiparesis
     Ventral pons
    Lacunar Bridge
    (London Bridge)

    Lacunar bridge is falling down


    VPL Thalamus, sensory fall down
    Posterior limb, motor fall down
    It’s in internal capsule

    Anterior limb, voice & face fall down


    Dorsal pons, voice & hand fall down
    Ataxic plus body half fall down
    It’s in ventral pons
    Brainstem
    Syndrome Area Ipsi Contra
    Weber medial basal midbrain CN III hemiplegia
    Benedikt tegmetum of midbrain CN III pain & T°,
    proprioception, tremor,
    chorea, ataxia
    Locked-In bilateral basal pons (+) upward gaze only
    Millard-Gubler lateral pons CN VI, VII hemiplegia
    Wallenberg lateral medulla CN X & V, ataxia, pain & T° of body
    (PICA/Lateral nystagmus,
    Medullary Horner’s syndrome,
    Syndrome) pain & T° of face
    AICA cerebellum, brainstem CN V, VI, VII, pain & T° of body
    ataxia, Horner’s
    syndrome, pain &
    T° of face
    SCA cerebellum, brainstem ataxia, Horner’s pain & T° of body & face
    Prognosis
     Poor Predictors
     coma at onset  visual-spatial
     persistent incontinence perceptual deficit
     poor cognitive  unilateral hemineglect
    function  significant
     severe hemiplegia cardiovascular disease
     lack of return of motor  large cerebral lesion
    function after 1 month  presence of multiple
     prior stroke neurologic deficits
    Medical Management
     Thrombosis & TIA
     Spasticity
     Seizures
     RSD

    You might also like