The human cytomegalovirus US11 gene product dislocates MHC class I heavy chains from the endoplasmic reticulum to the cytosol (Q29618773)

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30 April 2017

main subject

endoplasmic reticulum

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6

Ploegh HL

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30 April 2017

author name string

Wiertz EJ

1

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30 April 2017

Jones TR

2

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30 April 2017

Sun L

3

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30 April 2017

Bogyo M

4

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30 April 2017

Geuze HJ

5

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30 April 2017

language of work or name

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30 April 2017

publication date

8 March 1996

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30 April 2017

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volume

84

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issue

5

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30 April 2017

page(s)

769-79

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Folding of influenza hemagglutinin in the endoplasmic reticulum

30 April 2017

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An adenovirus type 2 glycoprotein blocks cell surface expression of human histocompatibility class I antigens

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Cytomegalovirus prevents antigen presentation by blocking the transport of peptide-loaded major histocompatibility complex class I molecules into the medial-Golgi compartment

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SecA promotes preprotein translocation by undergoing ATP-driven cycles of membrane insertion and deinsertion.

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Protein translocation into proteoliposomes reconstituted from purified components of the endoplasmic reticulum membrane

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A mammalian homolog of SEC61p and SECYp is associated with ribosomes and nascent polypeptides during translocation

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Role of N-linked oligosaccharide recognition, glucose trimming, and calnexin in glycoprotein folding and quality control

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Getting the inside out: the transporter associated with antigen processing (TAP) and the presentation of viral antigen

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Herpes simplex virus turns off the TAP to evade host immunity

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Multiple proteolytic systems, including the proteasome, contribute to CFTR processing

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Replacement mutagenesis of the human cytomegalovirus genome: US10 and US11 gene products are nonessential

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Proteolytic activity of human cytomegalovirus UL80 protease cleavage site mutants

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SecA protein is exposed to the periplasmic surface of the E. coli inner membrane in its active state.

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Modulation of MHC antigen expression by viruses and oncogenes

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An analysis of class I antigens of man and other species by one-dimensional IEF and immunoblotting

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Posttranslational protein transport in yeast reconstituted with a purified complex of Sec proteins and Kar2p

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Inhibitors of the proteasome block the degradation of most cell proteins and the generation of peptides presented on MHC class I molecules

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Sec61p and BiP directly facilitate polypeptide translocation into the ER.

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N-glycans as apical sorting signals in epithelial cells

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Mitochondrial Hsp70/MIM44 complex facilitates protein import

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In vivo inhibition of cyclin B degradation and induction of cell-cycle arrest in mammalian cells by the neutral cysteine protease inhibitor N-acetylleucylleucylnorleucinal

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