Autophagic vacuolation induced by excess ROS generation in HABP1/p32/gC1qR overexpressing fibroblasts and its reversal by polymeric hyaluronan (Q35036082)

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English	Autophagic vacuolation induced by excess ROS generation in HABP1/p32/gC1qR overexpressing fibroblasts and its reversal by polymeric hyaluronan	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

5 August 2017

Autophagic vacuolation induced by excess ROS generation in HABP1/p32/gC1qR overexpressing fibroblasts and its reversal by polymeric hyaluronan (English)

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Europe PubMed Central

PubMed publication ID

5 August 2017

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Shubhra Dutta

3

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Paramita Saha

1

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Europe PubMed Central

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5 August 2017

Anindya Roy Chowdhury

2

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5 August 2017

Soumya Chatterjee

4

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5 August 2017

Ilora Ghosh

5

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5 August 2017

Kasturi Datta

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5 August 2017

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publication date

18 October 2013

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Europe PubMed Central

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5 August 2017

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Europe PubMed Central

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5 August 2017

8

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Europe PubMed Central

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5 August 2017

10

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Europe PubMed Central

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5 August 2017

e78131

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Europe PubMed Central

PubMed publication ID

5 August 2017

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Creative Commons Attribution 4.0 International

18 October 2013

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Europe PubMed Central

11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC3799741/fullTextXML

based on heuristic

inferred from PubMed Central ID database lookup

p32/gC1qR is indispensable for fetal development and mitochondrial translation: importance of its RNA-binding ability

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Overexpression of hyaluronan-binding protein 1 (HABP1/p32/gC1qR) in HepG2 cells leads to increased hyaluronan synthesis and cell proliferation by up-regulation of cyclin D1 in AKT-dependent pathway

1 reference

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Evidence for Serpentine as a novel antioxidant by a redox sensitive HABP1 overexpressing cell line by inhibiting its nuclear translocation of NF-κB.

1 reference

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Agarose and polyacrylamide gel electrophoresis methods for molecular mass analysis of 5- to 500-kDa hyaluronan

1 reference

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Hypoxia triggers AMPK activation through reactive oxygen species-mediated activation of calcium release-activated calcium channels

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Hyaluronan-binding protein 1 (HABP1/p32/gC1qR) induces melanoma cell migration and tumor growth by NF-kappa B dependent MMP-2 activation through integrin α(v)β(3) interaction

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ROS and p53: a versatile partnership

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Autophagy in the Pathogenesis of Disease

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Excessive reactive oxygen species induces apoptosis in fibroblasts: role of mitochondrially accumulated hyaluronic acid binding protein 1 (HABP1/p32/gC1qR)

1 reference

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Mitochondrial electron-transport-chain inhibitors of complexes I and II induce autophagic cell death mediated by reactive oxygen species

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ROS, mitochondria and the regulation of autophagy

1 reference

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The autophagic inducer smARF interacts with and is stabilized by the mitochondrial p32 protein

1 reference

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Reactive oxygen species are essential for autophagy and specifically regulate the activity of Atg4

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PTEN regulates fibroblast elimination during collagen matrix contraction

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DRAM, a p53-induced modulator of autophagy, is critical for apoptosis

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Degradative action of reactive oxygen species on hyaluronan.

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Hyaluronan constitutively regulates ErbB2 phosphorylation and signaling complex formation in carcinoma cells.

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Intracellular hyaluronan: a new frontier for inflammation?

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Mammalian antioxidant defenses are not inducible by H2O2.

1 reference

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Constitutive expression of hyaluronan binding protein 1 (HABP1/p32/gC1qR) in normal fibroblast cells perturbs its growth characteristics and induces apoptosis

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Hyaluronan binding protein 1 (HABP1)/C1QBP/p32 is an endogenous substrate for MAP kinase and is translocated to the nucleus upon mitogenic stimulation

1 reference

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Identifiers

10.1371/JOURNAL.PONE.0078131

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Europe PubMed Central

PubMed publication ID

5 August 2017

2013PLoSO...878131S

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PMC publication ID

1 reference

Europe PubMed Central

PubMed publication ID

5 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

5 August 2017

ResearchGate publication ID

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