Strategies for neuroprotection against L-trans-2,4-pyrrolidine dicarboxylate-induced neuronal damage during energy impairment in vitro (Q43599704)

25 November 2017

title

Strategies for neuroprotection against L-trans-2,4-pyrrolidine dicarboxylate-induced neuronal damage during energy impairment in vitro (English)

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25 November 2017

O García

1

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25 November 2017

L Massieu

series ordinal

2

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25 November 2017

publication date

1 May 2001

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Journal of Neuroscience Research

25 November 2017

published in

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volume

64

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issue

4

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page(s)

418-428

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Glutamate-induced neuronal death: A succession of necrosis or apoptosis depending on mitochondrial function

25 November 2017

cites work

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Elevation of the Extracellular Concentrations of Glutamate and Aspartate in Rat Hippocampus During Transient Cerebral Ischemia Monitored by Intracerebral Microdialysis

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Characterization of the cellular reduction of 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT): subcellular localization, substrate dependence, and involvement of mitochondrial electron transport in MTT reduction

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The glutamate transport inhibitor L-trans-pyrrolidine-2,4-dicarboxylate indirectly evokes NMDA receptor mediated neurotoxicity in rat cortical cultures

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Conformationally defined neurotransmitter analogs. Selective inhibition of glutamate uptake by one pyrrolidine-2,4-dicarboxylate diastereomer

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Metabolic inhibition potentiates AMPA-induced Ca2+ fluxes and neurotoxicity in rat cerebellar granule cells

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Ions and energy in mammalian brain

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Interference with cellular energy metabolism reduces kynurenic acid formation in rat brain slices: reversal by lactate and pyruvate

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Metabolic control of kynurenic acid formation in the rat brain

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D-beta-hydroxybutyrate protects neurons in models of Alzheimer's and Parkinson's disease

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Excitotoxicity is required for induction of oxidative stress and apoptosis in mouse striatum by the mitochondrial toxin, 3-nitropropionic acid

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NMDA-dependent superoxide production and neurotoxicity

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K(+)-induced reversal of astrocyte glutamate uptake is limited by compensatory changes in intracellular Na+.

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Glutamate Neurotoxicity Is Independent of Calpain I Inhibition in Primary Cultures of Cerebellar Granule Cells

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Pyruvate and lactate protect striatal neurons against N-methyl-D-aspartate-induced neurotoxicity

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Glutamate becomes neurotoxic via the N-methyl-D-aspartate receptor when intracellular energy levels are reduced

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21 January 2018

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21 January 2018

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The competitive transport inhibitor L-trans-pyrrolidine-2, 4-dicarboxylate triggers excitotoxicity in rat cortical neuron-astrocyte co-cultures via glutamate release rather than uptake inhibition.

21 January 2018

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21 January 2018

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Protective Effect of the Energy Precursor Creatine Against Toxicity of Glutamate and β-Amyloid in Rat Hippocampal Neurons

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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In vivo potentiation of glutamate-mediated neuronal damage after chronic administration of the glycolysis inhibitor iodoacetate

21 January 2018

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3-Nitropropionic acid: an astrocyte-sparing neurotoxin in vitro

21 January 2018

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Distinct influx pathways, not calcium load, determine neuronal vulnerability to calcium neurotoxicity.

21 January 2018

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Neuroprotective strategies for treatment of lesions produced by mitochondrial toxins: implications for neurodegenerative diseases

21 January 2018

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Lactate-Supported Synaptic Function in the Rat Hippocampal Slice Preparation

21 January 2018

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Selective neurotoxicity of ruthenium red in primary cultures

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Inhibition of glutamate uptake induces progressive accumulation of extracellular glutamate and neuronal damage in rat cortical cultures

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Oxidative stress during energy impairment in mesencephalic cultures is not a downstream consequence of a secondary excitotoxicity

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21 January 2018

Identifiers

DOI

10.1002/JNR.1093

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25 November 2017

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