Neuronal excitatory properties of human immunodeficiency virus type 1 Tat protein. (Q45757726)

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9 November 2019

title

Neuronal excitatory properties of human immunodeficiency virus type 1 Tat protein (English)

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9 November 2019

7

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9 November 2019

author name string

J Cheng

1

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9 November 2019

A Nath

2

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9 November 2019

B Knudsen

3

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9 November 2019

S Hochman

4

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9 November 2019

J D Geiger

5

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9 November 2019

M Ma

6

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9 November 2019

language of work or name

English

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publication date

1 January 1998

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9 November 2019

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9 November 2019

volume

82

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9 November 2019

issue

1

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9 November 2019

page(s)

97-106

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Quisqualate- and kainate-activated channels in mouse central neurones in culture

9 November 2019

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AMPA receptor desensitization predicts the selective vulnerability of cerebellar Purkinje cells to excitotoxicity

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Calcium-mediated neurotoxicity: relationship to specific channel types and role in ischemic damage

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The role of Tat in the human immunodeficiency virus life cycle indicates a primary effect on transcriptional elongation

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AMPA Neurotoxicity in Rat Cerebellar and Hippocampal Slices: Histological Evidence for Three Mechanisms

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Prominent cortical atrophy with neuronal loss as correlate of human immunodeficiency virus encephalopathy

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Glutamate receptor-induced 45Ca2+ accumulation in cortical cell culture correlates with subsequent neuronal degeneration

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Neurotoxicity of peptide analogues of the transactivating protein tat from Maedi-Visna virus and human immunodeficiency virus

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Ca2+ permeability of KA-AMPA--gated glutamate receptor channels depends on subunit composition

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Permeation of calcium through excitatory amino acid receptor channels in cultured rat hippocampal neurones

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Glutamate receptor channels in isolated patches from CA1 and CA3 pyramidal cells of rat hippocampal slices

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Human immunodeficiency virus-infected macrophages, gp120, and N-methyl-D-aspartate receptor-mediated neurotoxicity

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Neuronal injury associated with HIV-1 and potential treatment with calcium-channel and NMDA antagonists

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Human immunodeficiency virus type 1 tat activates non-N-methyl-D-aspartate excitatory amino acid receptors and causes neurotoxicity

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Selective neuronal vulnerability in HIV encephalitis

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Mixed-agonist action of excitatory amino acids on mouse spinal cord neurones under voltage clamp

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Identification of a human immunodeficiency virus type 1 Tat epitope that is neuroexcitatory and neurotoxic

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The AIDS dementia complex: II. Neuropathology

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The basic domain of the lentiviral Tat protein is responsible for damages in mouse brain: involvement of cytokines

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The brain in AIDS: central nervous system HIV-1 infection and AIDS dementia complex

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7 January 2021

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7 January 2021

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Identifiers

DOI

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9 November 2019

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