Flecainide: Current status and perspectives in arrhythmia management

doi:10.4330/wjc.v7.i2.76

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Feb 26, 2015 (publication date) through Nov 19, 2024

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World Journal of Cardiology

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1949-8462

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Cardiol. Feb 26, 2015; 7(2): 76-85
Published online Feb 26, 2015. doi: 10.4330/wjc.v7.i2.76

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Figure 2 Mechanism of flecainide action during atrial fibrillation by inhibition of Na⁺ channels which reduces intracellular Ca²⁺ accumulation and reduces oxidative stress and mitochondrial dysfunction. AF; Atrial fibrillation; INa: Fast inward Na⁺ current; ROS: Reactive oxygen species; NFκB: Nuclear factor kappa β; ATP: Adenosine triphosphate.

Citation: Andrikopoulos GK, Pastromas S, Tzeis S. Flecainide: Current status and perspectives in arrhythmia management. World J Cardiol 2015; 7(2): 76-85
URL: https://www.wjgnet.com/1949-8462/full/v7/i2/76.htm
DOI: https://dx.doi.org/10.4330/wjc.v7.i2.76